Adult hypophosphatasia

disease
On this page

Also known as hypophosphatasia of adults

Summary

Adult hypophosphatasia (MONDO:1010154) is a disease with 1 cohort gene and 1 clinical trial.

At a glance

  • Prevalence: Unknown (Worldwide)
  • Cohort genes: 1
  • ClinVar variants: 343
  • Clinical trials: 1

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameadult hypophosphatasia
Mondo IDMONDO:1010154
OMIM146300
Orphanet247676
DOIDDOID:0110913
SNOMED CT20756002
UMLSC0268413
MedGen120636
GARD0028131
Is cancer (heuristic)no

Also known as: adult hypophosphatasia · hypophosphatasia of adults

Data availability: 343 ClinVar variants.

Disease family

Classification path: disease › human disease › disease by developmental or physiological process › metabolic diseasedevelopmental anomaly of metabolic originhypophosphatasiaadult hypophosphatasia

Related subtypes (7): odontohypophosphatasia, ALPL-related autosomal dominant hypophosphatasia, ALPL-related autosomal recessive hypophosphatasia, moderate hypophosphatasia, prenatal benign hypophosphatasia, childhood hypophosphatasia, infantile hypophosphatasia

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

343 retrieved; paginated sample, class counts are floors:

119 pathogenic/likely pathogenic, 53 conflicting classifications of pathogenicity, 49 uncertain significance, 40 pathogenic, 39 likely pathogenic, 21 likely benign, 17 benign, 5 benign/likely benign

ClinVarVariant (HGVS)GeneClassificationReview
1003680NM_000478.6(ALPL):c.1022A>G (p.His341Arg)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1030812NM_000478.6(ALPL):c.1328C>T (p.Ala443Val)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1047062NM_000478.6(ALPL):c.655A>G (p.Met219Val)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1067343NM_000478.6(ALPL):c.508A>G (p.Asn170Asp)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1070177NM_000478.6(ALPL):c.212G>A (p.Arg71His)ALPLPathogeniccriteria provided, multiple submitters, no conflicts
1070178NM_000478.6(ALPL):c.223G>A (p.Gly75Ser)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1072423NM_000478.6(ALPL):c.1240C>A (p.Leu414Met)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1074126NM_000478.6(ALPL):c.976G>C (p.Gly326Arg)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1074643NM_000478.6(ALPL):c.997G>T (p.Gly333Ter)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1076060NM_000478.6(ALPL):c.203C>T (p.Thr68Met)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1076087NM_000478.6(ALPL):c.303C>A (p.Tyr101Ter)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1076159NM_000478.6(ALPL):c.484G>A (p.Gly162Ser)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1076160NM_000478.6(ALPL):c.532T>C (p.Tyr178His)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1076161NM_000478.6(ALPL):c.659G>C (p.Gly220Ala)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1224321NM_000478.6(ALPL):c.1166C>A (p.Thr389Asn)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1224381NM_000478.6(ALPL):c.657G>T (p.Met219Ile)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1300140NM_000478.6(ALPL):c.1427A>C (p.Glu476Ala)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1323883NM_000478.6(ALPL):c.874C>A (p.Pro292Thr)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1334796NM_000478.6(ALPL):c.214A>G (p.Ile72Val)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1339258NM_000478.6(ALPL):c.247G>T (p.Glu83Ter)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1355608NM_000478.6(ALPL):c.480del (p.Val161fs)ALPLPathogeniccriteria provided, multiple submitters, no conflicts
13662NM_000478.6(ALPL):c.535G>A (p.Ala179Thr)ALPLPathogeniccriteria provided, multiple submitters, no conflicts
13663NM_000478.6(ALPL):c.211C>T (p.Arg71Cys)ALPLPathogeniccriteria provided, multiple submitters, no conflicts
13664NM_000478.6(ALPL):c.881A>C (p.Asp294Ala)ALPLPathogeniccriteria provided, multiple submitters, no conflicts
13665NM_000478.6(ALPL):c.212G>C (p.Arg71Pro)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
13667NM_000478.6(ALPL):c.98C>T (p.Ala33Val)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
13671NM_000478.6(ALPL):c.1133A>T (p.Asp378Val)ALPLPathogeniccriteria provided, multiple submitters, no conflicts
13672NM_000478.6(ALPL):c.1001G>A (p.Gly334Asp)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
13673NM_000478.6(ALPL):c.979T>C (p.Phe327Leu)ALPLPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
13674NM_000478.6(ALPL):c.1559del (p.Leu520fs)ALPLPathogeniccriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 0 · Orphanet: 6 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
ALPLOrphanet:247623Perinatal lethal hypophosphatasia
ALPLOrphanet:247638Prenatal benign hypophosphatasia
ALPLOrphanet:247651Infantile hypophosphatasia
ALPLOrphanet:247667Childhood-onset hypophosphatasia
ALPLOrphanet:247676Adult hypophosphatasia
ALPLOrphanet:247685Odontohypophosphatasia

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
ALPLHGNC:438ENSG00000162551P05186Alkaline phosphatase, tissue-nonspecific isozymeclinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
ALPLAlkaline phosphatase, tissue-nonspecific isozymeAlkaline phosphatase that metabolizes various phosphate compounds and plays a key role in skeletal mineralization and adaptive thermogenesis.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 0 · Druggable fraction: 1.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Phosphatase183.9×0.012

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
ALPLPhosphataseyes3.1.3.1Alkaline_phosphatase, Alkaline_phosphatase_core_sf, Alkaline_phosphatase_AS

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
left adrenal gland cortex1
right adrenal gland1
right adrenal gland cortex1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
ALPL200broadmarkerright adrenal gland, right adrenal gland cortex, left adrenal gland cortex

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
ALPL2,146

Structural data

PDB: 1 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
ALPLP051865

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 1 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Post-translational modification: synthesis of GPI-anchored proteins1167.9×0.006ALPL

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
pyridoxal 5’-phosphate metabolic process116852.0×8e-04ALPL
response to vitamin B618426.0×8e-04ALPL
futile creatine cycle18426.0×8e-04ALPL
response to macrophage colony-stimulating factor14213.0×1e-03ALPL
inhibition of non-skeletal tissue mineralization14213.0×1e-03ALPL
developmental process involved in reproduction13370.4×0.001ALPL
cementum mineralization12407.4×0.001ALPL
response to sodium phosphate11685.2×0.002ALPL
phosphate ion homeostasis11053.2×0.002ALPL
cellular homeostasis1802.5×0.002ALPL
response to vitamin D1802.5×0.002ALPL
response to antibiotic1702.2×0.002ALPL
endochondral ossification1543.6×0.003ALPL
calcium ion homeostasis1443.5×0.003ALPL
response to glucocorticoid1324.1×0.004ALPL
bone mineralization1271.8×0.005ALPL
response to insulin1230.8×0.005ALPL
positive regulation of cold-induced thermogenesis1163.6×0.007ALPL
skeletal system development1125.8×0.008ALPL
response to lipopolysaccharide1124.8×0.008ALPL
osteoblast differentiation1121.2×0.008ALPL

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 1 · Undrugged: 0

Druggability breadth: 1 of 1 evidence-associated genes (100%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
ALPLSULCONAZOLE NITRATE

Top cohort targets by molecule count

SymbolMoleculesMax phase
ALPL74

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
SULCONAZOLE NITRATE4ALPL
THEOPHYLLINE4ALPL
LEVAMISOLE4ALPL
MICONAZOLE NITRATE4ALPL
LEVAMISOLE HYDROCHLORIDE4ALPL
ISOQUERCETIN2ALPL
(-)-EPICATECHIN2ALPL

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
ALPL58Binding:50, Functional:4, ADMET:3, Toxicity:1

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
ALPL3.1.3.1alkaline phosphatase

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

7 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
SULCONAZOLE NITRATE4ALPL
THEOPHYLLINE4ALPL
LEVAMISOLE4ALPL
MICONAZOLE NITRATE4ALPL
LEVAMISOLE HYDROCHLORIDE4ALPL
ISOQUERCETIN2ALPL
(-)-EPICATECHIN2ALPL

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1ALPL
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug0

Undrugged target profiles

0 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified1

Top trials by phase / activity

NCTPhaseStatusTitle
NCT01793168Not specifiedRECRUITINGRare Disease Patient Registry & Natural History Study - Coordination of Rare Diseases at Sanford