Age related macular degeneration 7

disease
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Also known as age related macular degeneration type 7age-related macular degeneration caused by mutation in HTRA1ARMD7HTRA1 age-related macular degenerationmacular degeneration, age-related, 7macular degeneration, age-related, neovascular typemacular Degeneration, age-related, type 7

Summary

Age related macular degeneration 7 (MONDO:0012419) is a disease with 2 cohort genes.

At a glance

  • Cohort genes: 2
  • ClinVar variants: 5

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameage related macular degeneration 7
Mondo IDMONDO:0012419
MeSHC565718
OMIM610149
DOIDDOID:0110019
UMLSC1857813
MedGen347554
GARD0024864
Is cancer (heuristic)no

Also known as: age related macular degeneration type 7 · age-related macular degeneration caused by mutation in HTRA1 · ARMD7 · HTRA1 age-related macular degeneration · macular degeneration, age-related, 7 · macular degeneration, age-related, neovascular type · macular Degeneration, age-related, type 7

Data availability: 5 ClinVar variants.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disorderretinal disorderretinal degenerationmacular degenerationdegeneration of macula and posterior poleage-related macular degenerationage related macular degeneration 7

Related subtypes (14): wet macular degeneration, age related macular degeneration 2, age related macular degeneration 1, macular degeneration, age-related, 3, age related macular degeneration 4, age related macular degeneration 9, age related macular degeneration 10, age related macular degeneration 11, age related macular degeneration 6, age related macular degeneration 8, age related macular degeneration 12, age related macular degeneration 14, macular dystrophy with central cone involvement, dry age related macular degeneration

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

5 retrieved; paginated sample, class counts are floors:

1 uncertain significance, 1 conflicting classifications of pathogenicity, 1 pathogenic/likely pathogenic, 1 association, 1 risk factor

ClinVarVariant (HGVS)GeneClassificationReview
1948614NM_002775.5(HTRA1):c.972+1G>AHTRA1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
7486NC_000010.11:g.122461028G>AHTRA1risk factorno assertion criteria provided
1704529NM_002775.5(HTRA1):c.958G>A (p.Asp320Asn)HTRA1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2627016NM_001922.5(DCT):c.1180-122A>GDCTassociationno assertion criteria provided
1501480NM_002775.5(HTRA1):c.820C>T (p.Arg274Trp)HTRA1Uncertain significancecriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 0 · Orphanet: 5 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
DCTOrphanet:597733Oculocutaneous albinism type 8
HTRA1Orphanet:199354Cerebral autosomal recessive arteriopathy-subcortical infarcts-leukoencephalopathy
HTRA1Orphanet:252128Malignant peripheral nerve sheath tumor with perineurial differentiation
HTRA1Orphanet:252212Malignant triton tumor
HTRA1Orphanet:482077HTRA1-related autosomal dominant cerebral small vessel disease

Cohort genes → proteins

2 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
DCTHGNC:2709ENSG00000080166P40126L-dopachrome tautomeraseclinvar
HTRA1HGNC:9476ENSG00000166033Q92743Serine protease HTRA1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
DCTL-dopachrome tautomerasePlays a role in melanin biosynthesis.
HTRA1Serine protease HTRA1Serine protease with a variety of targets, including extracellular matrix proteins such as fibronectin.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.5

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Protease118.3×0.108
Other/Unknown10.9×0.805

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
DCTOther/UnknownnoTyrosinase_Cu-bd, Di-copper_centre_dom_sf, Tyrosinase/Hemocyanin
HTRA1Proteaseyes3.4.21.107IGFBP-like, PDZ, Peptidase_S1C

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
secondary oocyte1
upper arm skin1
upper leg skin1
metanephric glomerulus1
renal glomerulus1
tendon of biceps brachii1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
DCT184broadmarkerupper leg skin, upper arm skin, secondary oocyte
HTRA1287ubiquitousmarkertendon of biceps brachii, renal glomerulus, metanephric glomerulus

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
HTRA12,843
DCT1,692

Structural data

PDB: 2 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
HTRA1Q9274318
DCTP401261

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 3. Enrichment computed across 2 evidence-associated genes (2 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Melanin biosynthesis11142.0×0.003DCT
Regulation of MITF-M-dependent genes involved in pigmentation1132.8×0.011DCT
Degradation of the extracellular matrix158.9×0.017HTRA1

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
melanin biosynthetic process from tyrosine12106.5×0.002DCT
ventricular zone neuroblast division12106.5×0.002DCT
response to blue light11685.2×0.002DCT
chorionic trophoblast cell differentiation11404.3×0.002HTRA1
developmental pigmentation11053.2×0.003DCT
programmed cell death1648.1×0.004HTRA1
cell development1443.5×0.005DCT
positive regulation of neuroblast proliferation1290.6×0.006DCT
placenta development1221.7×0.007HTRA1
negative regulation of BMP signaling pathway1145.3×0.010HTRA1
epidermis development1105.3×0.012DCT
negative regulation of transforming growth factor beta receptor signaling pathway186.9×0.013HTRA1
positive regulation of apoptotic process128.4×0.038HTRA1
proteolysis117.1×0.058HTRA1

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 2

Druggability breadth: 1 of 2 evidence-associated genes (50%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
DCT00
HTRA100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
HTRA128Binding:28

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
HTRA13.4.21.107, 3.4.21.108peptidase Do, HtrA2 peptidase

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1HTRA1
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1DCT

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
DCT0
HTRA128

Clinical trials & evidence

Clinical trials

Clinical trials: 0.