AH amyloidosis

disease
On this page

Also known as heavy chain amyloidosis

Summary

AH amyloidosis (MONDO:0018613) is a disease and 1 clinical trial. A subtype of amyloidosis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 8
  • Clinical trials: 1

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families12WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Signs & symptoms

Clinical features (HPO)

8 HPO clinical features (Orphanet curated; top 8 by frequency):

HPO IDTermFrequency
HP:0000112NephropathyVery frequent (80-99%)
HP:0000853GoiterFrequent (30-79%)
HP:0001917Renal amyloidosisFrequent (30-79%)
HP:0030843Cardiac amyloidosisFrequent (30-79%)
HP:0031047ParaproteinemiaFrequent (30-79%)
HP:0031185Increased circulating NT-proBNP concentrationFrequent (30-79%)
HP:0100292Amyloidosis of peripheral nervesFrequent (30-79%)
HP:0009830Peripheral neuropathyOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical nameAH amyloidosis
Mondo IDMONDO:0018613
Orphanet442582
DOIDDOID:0080934
ICD-111511136608
NCITC158962
UMLSC5204115
MedGen1684787
GARD0021847
Is cancer (heuristic)no

Also known as: heavy chain amyloidosis

Disease family

This is a subtype of amyloidosis. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by developmental or physiological process › metabolic diseaseproteostasis deficienciesamyloidosisAH amyloidosis

Related subtypes (11): primary cutaneous amyloidosis, wild type ATTR amyloidosis, ALECT2 amyloidosis, AApoAIV amyloidosis, ABeta2M amyloidosis, hereditary amyloidosis, AL amyloidosis, AA amyloidosis, amyloidosis bronchopulmonary, soft tissue amyloid neoplasm, immunoglobulin heavy-and-light chain

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified1

Top trials by phase / activity

NCTPhaseStatusTitle
NCT06065852Not specifiedRECRUITINGNational Registry of Rare Kidney Diseases

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.