ALECT2 amyloidosis

disease
On this page

Also known as LECT2 amyloidosisleukocyte chemotactic factor-2 amyloidosis

Summary

ALECT2 amyloidosis (MONDO:0018588) is a disease and 1 clinical trial. A subtype of amyloidosis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Phenotypes (HPO): 12
  • Clinical trials: 1

Clinical features

Signs & symptoms

Clinical features (HPO)

12 HPO clinical features (Orphanet curated; top 12 by frequency):

HPO IDTermFrequency
HP:0001917Renal amyloidosisVery frequent (80-99%)
HP:0012280Hepatic amyloidosisVery frequent (80-99%)
HP:0000083Renal insufficiencyFrequent (30-79%)
HP:0000093ProteinuriaFrequent (30-79%)
HP:0000822HypertensionFrequent (30-79%)
HP:0003259Elevated circulating creatinine concentrationFrequent (30-79%)
HP:0033247Pulmonary amyloidosisFrequent (30-79%)
HP:0000853GoiterOccasional (5-29%)
HP:0002910Elevated circulating hepatic transaminase concentrationOccasional (5-29%)
HP:0012145Abnormality of multiple cell lineages in the bone marrowOccasional (5-29%)
HP:6000815Amyloid goiterOccasional (5-29%)
HP:0002907Microscopic hematuriaVery rare (<1-4%)

Identifiers

Disease identifiers

FieldValue
Canonical nameALECT2 amyloidosis
Mondo IDMONDO:0018588
Orphanet439224
ICD-111363099940
UMLSC5680047
MedGen1801621
GARD0021827
Is cancer (heuristic)no

Also known as: LECT2 amyloidosis · leukocyte chemotactic factor-2 amyloidosis

Disease family

This is a subtype of amyloidosis. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by developmental or physiological process › metabolic diseaseproteostasis deficienciesamyloidosisALECT2 amyloidosis

Related subtypes (11): primary cutaneous amyloidosis, wild type ATTR amyloidosis, AApoAIV amyloidosis, ABeta2M amyloidosis, AH amyloidosis, hereditary amyloidosis, AL amyloidosis, AA amyloidosis, amyloidosis bronchopulmonary, soft tissue amyloid neoplasm, immunoglobulin heavy-and-light chain

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified1

Top trials by phase / activity

NCTPhaseStatusTitle
NCT03774784Not specifiedTERMINATEDA Study of the Natural History of Leukocyte Chemotactic Factor 2 Amyloidosis (ALECT2) Disease

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.