Alexia
diseaseOn this page
Also known as acquired alexiaacquired dyslexiaacquired global dyslexiaacquired reading disabilitiesacquired reading disabilityacquired spelling dyslexiaacquired Word blindnessacquired Word Blindnessesalexia, acquiredblindness, acquired WordBlindnesses, acquired Worddisabilities, acquired readingdisability, acquired readingdyslexia, acquired globaldyslexia, acquired spellingglobal dyslexia, acquiredreading disabilities, acquiredreading disability, acquiredspelling dyslexia, acquiredWord blindness, acquired
Summary
Alexia (MONDO:0001712) is a disease and 4 clinical trials. A subtype of visual agnosia — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
At a glance
- Clinical trials: 4
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | alexia |
| Mondo ID | MONDO:0001712 |
| MeSH | D004411 |
| DOID | DOID:13417 |
| ICD-11 | 262295178 |
| UMLS | C0002018 |
| MedGen | 1809 |
| GARD | 0027588 |
| Is cancer (heuristic) | no |
Also known as: acquired alexia · acquired dyslexia · acquired global dyslexia · acquired reading disabilities · acquired reading disability · acquired spelling dyslexia · acquired Word blindness · acquired Word Blindnesses · alexia, acquired · blindness, acquired Word · Blindnesses, acquired Word · disabilities, acquired reading · disability, acquired reading · dyslexia, acquired global · dyslexia, acquired spelling · global dyslexia, acquired · reading disabilities, acquired · reading disability, acquired · spelling dyslexia, acquired · Word blindness, acquired (+1 more)
Disease family
This is a subtype of visual agnosia. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by developmental or physiological process › psychiatric disorder › mental disorder › visual agnosia › alexia
Related subtypes (6): apperceptive agnosia, associative visual agnosia, color agnosia, form agnosia, topographical agnosia, alexia without agraphia
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 4.
Phase distribution (across all retrieved trials)
| Phase | Trials |
|---|---|
| Not specified | 4 |
Top trials by phase / activity
| NCT | Phase | Status | Title |
|---|---|---|---|
| NCT04991519 | Not specified | ACTIVE_NOT_RECRUITING | Brain-based Understanding of Individual Language Differences After Stroke |
| NCT06700005 | Not specified | RECRUITING | ReadMap: Reading in Stroke Alexia and Typical Aging |
| NCT03662243 | Not specified | COMPLETED | Treatment for Reading and Writing Deficits Following Acquired Brain Injury |
| NCT04849091 | Not specified | UNKNOWN | Clinical Effectiveness of iReadMore for People With Alexia |
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.