Allergic otitis media

Summary

Allergic otitis media (MONDO:0021202) is a disease. A subtype of allergic disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	allergic otitis media
Mondo ID	MONDO:0021202
SNOMED CT	26169004
UMLS	C0271447
MedGen	543352
Is cancer (heuristic)	no

Also known as: allergic form of otitis media (disease) · allergic otitis media (disease)

Disease family

This is a subtype of allergic disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › immune system disorder › hypersensitivity reaction disease › allergic disease › allergic otitis media

Related subtypes (11): allergic respiratory disease, drug allergy, gastrointestinal allergy, latex allergy, atopic eczema, atopic IgE-mediated allergic disorder, eye allergy, vulvovaginitis, allergic seminal, alpha-gal syndrome, venom allergy, food allergy

Subtypes (1): acute allergic serous otitis media

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.