Amyloidosis, primary localized cutaneous, 2

disease
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Also known as amyloidosis, primary localised cutaneous, type 2amyloidosis, primary localized cutaneous, type 2PLCA2

Summary

Amyloidosis, primary localized cutaneous, 2 (MONDO:0013502) is a disease with 1 cohort gene.

At a glance

  • Cohort genes: 1
  • ClinVar variants: 8

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameamyloidosis, primary localized cutaneous, 2
Mondo IDMONDO:0013502
OMIM613955
DOIDDOID:0080931
UMLSC3151404
MedGen462754
GARD0018638
Is cancer (heuristic)no

Also known as: amyloidosis, primary localised cutaneous, type 2 · amyloidosis, primary localized cutaneous, 2 · amyloidosis, primary localized cutaneous, type 2 · PLCA2

Data availability: 8 ClinVar variants · 3 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by developmental or physiological process › metabolic diseaseproteostasis deficienciesamyloidosisprimary cutaneous amyloidosisfamilial primary localized cutaneous amyloidosisamyloidosis, primary localized cutaneous, 2

Related subtypes (2): amyloidosis, primary localized cutaneous, 1, amyloidosis, primary localized cutaneous, 3

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

8 retrieved; paginated sample, class counts are floors:

4 benign, 3 uncertain significance, 1 pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
30790NM_139017.7(IL31RA):c.1562C>T (p.Ser521Phe)IL31RAPathogenicno assertion criteria provided
2378759NM_139017.7(IL31RA):c.1327T>A (p.Ser443Thr)IL31RAUncertain significancecriteria provided, multiple submitters, no conflicts
2509555NM_139017.7(IL31RA):c.2143G>A (p.Ala715Thr)IL31RAUncertain significancecriteria provided, multiple submitters, no conflicts
3779766NM_139017.7(IL31RA):c.1070-3C>GIL31RAUncertain significancecriteria provided, single submitter
1098854NM_139017.7(IL31RA):c.1449T>G (p.Gly483=)IL31RABenigncriteria provided, multiple submitters, no conflicts
1300053NM_139017.7(IL31RA):c.129C>T (p.Pro43=)IL31RABenigncriteria provided, multiple submitters, no conflicts
1300054NM_139017.7(IL31RA):c.606+29A>TIL31RABenigncriteria provided, multiple submitters, no conflicts
1300055NM_139017.7(IL31RA):c.1819-26T>GIL31RABenigncriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 4 · Orphanet: 1 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
IL31RASupportiveAutosomal dominantfamilial primary localized cutaneous amyloidosis4

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
IL31RAOrphanet:353220Familial primary localized cutaneous amyloidosis

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
IL31RAHGNC:18969ENSG00000164509Q8NI17Interleukin-31 receptor subunit alphagencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
IL31RAInterleukin-31 receptor subunit alphaAssociates with OSMR to form the interleukin-31 receptor which activates STAT3 and to a lower extent STAT1 and STAT5.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 0 · Druggable fraction: 1.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Antibody/Immunoglobulin129.2×0.034

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
IL31RAAntibody/ImmunoglobulinyesFN3_dom, Ig-like_fold, TypeI_recpt_CBD

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
male germ line stem cell (sensu Vertebrata) in testis1
monocyte1
primordial germ cell in gonad1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
IL31RA129broadmarkermale germ line stem cell (sensu Vertebrata) in testis, primordial germ cell in gonad, monocyte

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
IL31RA2,789

Structural data

PDB: 1 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
IL31RAQ8NI171

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 1 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
IL-6-type cytokine receptor ligand interactions1634.4×0.002IL31RA

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
defense response to other organism116852.0×9e-04IL31RA
acute inflammatory response to antigenic stimulus12808.7×0.002IL31RA
glandular epithelial cell differentiation12106.5×0.002IL31RA
homeostatic process11685.2×0.002IL31RA
negative regulation of macrophage activation11685.2×0.002IL31RA
monocyte differentiation1802.5×0.003IL31RA
positive regulation of tyrosine phosphorylation of STAT protein1732.7×0.003IL31RA
macrophage differentiation1468.1×0.004IL31RA
cell surface receptor signaling pathway via JAK-STAT1290.6×0.006IL31RA
defense response1216.1×0.007IL31RA
cell surface receptor protein tyrosine kinase signaling pathway1173.7×0.008IL31RA
MAPK cascade1153.2×0.009IL31RA
cytokine-mediated signaling pathway1130.6×0.009IL31RA
negative regulation of apoptotic process134.8×0.032IL31RA
positive regulation of cell population proliferation133.6×0.032IL31RA
positive regulation of DNA-templated transcription127.9×0.036IL31RA

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 1 of 1 evidence-associated genes (100%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
IL31RA00

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1IL31RA
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug0

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
IL31RA0

Clinical trials & evidence

Clinical trials

Clinical trials: 0.