Aneurysm, intracranial berry, 5

Summary

Aneurysm, intracranial berry, 5 (MONDO:0010468) is a disease. A subtype of intracranial berry aneurysm — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	aneurysm, intracranial berry, 5
Mondo ID	MONDO:0010468
MeSH	C563670
OMIM	300870
DOID	DOID:0080968
UMLS	C1835857
MedGen	324377
GARD	0018321
Is cancer (heuristic)	no

Also known as: ANIB5

Disease family

This is a subtype of intracranial berry aneurysm. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: human disease › disease by body system or component › nervous system disorder › central nervous system disorder › brain disorder › cerebrovascular disorder › intracranial arterial disease › cerebral arterial disease › brain aneurysm › intracranial berry aneurysm › aneurysm, intracranial berry, 5

Related subtypes (11): aneurysm, intracranial berry type 1, aneurysm, intracranial berry, 2, aneurysm, intracranial berry, 3, aneurysm, intracranial berry, 4, aneurysm, intracranial berry, 6, aneurysm, intracranial berry, 7, aneurysm, intracranial berry, 8, aneurysm, intracranial berry, 9, aneurysm, intracranial berry, 10, aneurysm, intracranial berry, 11, aneurysm, intracranial berry, 12

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.