Aplasia cutis-myopia syndrome

disease
On this page

Also known as aplasia cutis myopiaGershoni-Baruch-Leibo syndrome

Summary

Aplasia cutis-myopia syndrome (MONDO:0010988) is a disease. A subtype of aplasia cutis congenita — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 11

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families4WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Signs & symptoms

Clinical features (HPO)

11 HPO clinical features (Orphanet curated; top 11 by frequency):

HPO IDTermFrequency
HP:0001057Aplasia cutis congenitaVery frequent (80-99%)
HP:0001362Skull defectVery frequent (80-99%)
HP:0006934Congenital nystagmusVery frequent (80-99%)
HP:0007703Abnormality of retinal pigmentationVery frequent (80-99%)
HP:0011003High myopiaVery frequent (80-99%)
HP:0000924Abnormality of the skeletal systemOccasional (5-29%)
HP:0001287MeningitisOccasional (5-29%)
HP:0001892Abnormal bleedingOccasional (5-29%)
HP:0001928Abnormality of coagulationOccasional (5-29%)
HP:0012639Abnormal nervous system morphologyOccasional (5-29%)
HP:0200042Skin ulcerOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical nameaplasia cutis-myopia syndrome
Mondo IDMONDO:0010988
MeSHC563394
OMIM601075
Orphanet1117
SNOMED CT720499004
UMLSC1832826
MedGen331375
GARD0000756
Is cancer (heuristic)no

Also known as: aplasia cutis myopia · Gershoni-Baruch-Leibo syndrome

Disease family

This is a subtype of aplasia cutis congenita. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › integumentary system disorder › skin disorder › dermis disorder › mixed dermis disorder › aplasia cutis congenitaaplasia cutis-myopia syndrome

Related subtypes (2): aplasia cutis autosomal recessive, aplasia cutis congenita dominant

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.