Balo concentric sclerosis

disease
On this page

Also known as Balo DiseaseBalo's concentric sclerosisBalo's diseaseBaló concentric sclerosisconcentric demyelinationdiffuse cerebral sclerosis of Schilderencephalitis periaxialis concentricaMarburg variantTumefactive multiple sclerosis

Summary

Balo concentric sclerosis (MONDO:0016430) is a disease and 1 clinical trial. A subtype of demyelinating disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide)
  • Clinical trials: 1

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameBalo concentric sclerosis
Mondo IDMONDO:0016430
Orphanet228165
DOIDDOID:0060215
SNOMED CT230380005
UMLSC0004712
MedGen498
GARD0005885
MedDRA10010252
NORD835
Is cancer (heuristic)no

Also known as: Balo concentric sclerosis · Balo Disease · Balo disease · Balo’s concentric sclerosis · Balo’s disease · Baló concentric sclerosis · concentric demyelination · diffuse cerebral sclerosis of Schilder · encephalitis periaxialis concentrica · Marburg variant · Tumefactive multiple sclerosis

Disease family

This is a subtype of demyelinating disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disordercentral nervous system disorderneurodegenerative diseasedemyelinating diseaseBalo concentric sclerosis

Related subtypes (7): demyelinating polyneuropathy, central pontine myelinolysis, polyradiculoneuropathy, Schilder disease, acute disseminated encephalomyelitis, demyelinating disease of central nervous system, boylan dew greco syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 1.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE31

Top trials by phase / activity

NCTPhaseStatusTitle
NCT00004645PHASE3UNKNOWNPhase III Randomized, Double-Blind, Sham-Controlled Study of Plasma Exchange for Acute Severe Attacks of Inflammatory Demyelinating Disease Refractory to Intravenous Methylprednisolone

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.