Benign neoplasm of cecum

Summary

Benign neoplasm of cecum (MONDO:0021464) is a cancer. A subtype of benign colon neoplasm — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Classification: Cancer

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	benign neoplasm of cecum
Mondo ID	MONDO:0021464
ICD-10-CM	D12.0
ICD-11	1697040268
NCIT	C4772
SNOMED CT	92040001
UMLS	C0496859
MedGen	105420
Anatomy (UBERON)	UBERON:0001153
Is cancer (heuristic)	yes

Also known as: benign cecum neoplasm · benign cecum tumor · benign cecum tumour · benign neoplasm of the cecum · benign tumor of cecum · benign tumor of the cecum · benign tumour of cecum · benign tumour of the cecum · caecum benign neoplasm

Disease family

This is a subtype of benign colon neoplasm. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › digestive system disorder › intestinal disorder › large intestine disorder › colonic disorder › colonic neoplasm › benign colon neoplasm › benign neoplasm of cecum

Related subtypes (4): lipoma of colon, colon leiomyoma, colonic lymphangioma, cavernous hemangioma of colon

Subtypes (1): benign neoplasm of appendix

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.