Bilateral frontal polymicrogyria

disease
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Summary

Bilateral frontal polymicrogyria (MONDO:0016162) is a disease. A subtype of bilateral polymicrogyria — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Phenotypes (HPO): 11

Clinical features

Signs & symptoms

Clinical features (HPO)

11 HPO clinical features (Orphanet curated; top 11 by frequency):

HPO IDTermFrequency
HP:0000750Delayed speech and language developmentVery frequent (80-99%)
HP:0001249Intellectual disabilityVery frequent (80-99%)
HP:0001263Global developmental delayVery frequent (80-99%)
HP:0001256Intellectual disability, mildFrequent (30-79%)
HP:0001285Spastic tetraparesisFrequent (30-79%)
HP:0001250SeizureOccasional (5-29%)
HP:0001269HemiparesisOccasional (5-29%)
HP:0002353EEG abnormalityOccasional (5-29%)
HP:0004302Functional motor deficitOccasional (5-29%)
HP:0006801Hyperactive deep tendon reflexesOccasional (5-29%)
HP:0002200Pseudobulbar signsExcluded (0%)

Identifiers

Disease identifiers

FieldValue
Canonical namebilateral frontal polymicrogyria
Mondo IDMONDO:0016162
Orphanet208444
DOIDDOID:0080921
ICD-11688947844
UMLSC5437679
MedGen1754014
GARD0010783
Is cancer (heuristic)no

Disease family

This is a subtype of bilateral polymicrogyria. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disorderpolymicrogyriabilateral polymicrogyriabilateral frontal polymicrogyria

Related subtypes (4): bilateral frontoparietal polymicrogyria, bilateral parasagittal parieto-occipital polymicrogyria, bilateral generalized polymicrogyria, bilateral perisylvian polymicrogyria

Subtypes (1): complex cortical dysplasia with other brain malformations 7

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.