blastema predominant kidney Wilms tumor

Summary

blastema predominant kidney Wilms tumor (MONDO:0003320) is a cancer. A subtype of kidney Wilms tumor — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Classification: Cancer

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	blastema predominant kidney Wilms tumor
Mondo ID	MONDO:0003320
DOID	DOID:5182
NCIT	C9147
UMLS	C0279609
MedGen	83528
GARD	0023446
Is cancer (heuristic)	yes

Also known as: blastema predominant kidney adenosarcoma · blastema predominant kidney Wilms tumor · blastema predominant kidney Wilms’ tumour · blastema predominant nephroblastoma · blastema predominant renal adenosarcoma · blastema predominant renal Wilm’s tumor · blastema predominant renal Wilm’s tumour · blastema predominant renal Wilms tumor · blastema predominant renal Wilms’ tumor · blastema predominant renal Wilms’ tumour · blastema predominant Wilms tumor · blastema predominant Wilms tumour

Disease family

This is a subtype of kidney Wilms tumor. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › cancer › malignant urinary system neoplasm › kidney cancer › kidney Wilms tumor › blastema predominant kidney Wilms tumor

Related subtypes (7): nonanaplastic kidney Wilms tumor, metachronous kidney Wilms’ tumor, mixed cell type kidney Wilms’ tumor, epithelial predominant Wilms’ tumor, stromal predominant kidney Wilms tumor, adult kidney Wilms tumor, childhood kidney Wilms tumor

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.