Blind loop syndrome
disease diseaseOn this page
Also known as stasis (blind loop) syndromestasis syndrome
Summary
Blind loop syndrome (MONDO:0005673) is a disease and 2 clinical trials. A subtype of malabsorption syndrome — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
At a glance
- Clinical trials: 2
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | blind loop syndrome |
| Mondo ID | MONDO:0005673 |
| EFO | EFO:0007175 |
| MeSH | D001765 |
| DOID | DOID:10606 |
| ICD-11 | 1719064637 |
| NCIT | C34431 |
| SNOMED CT | 66379009 |
| UMLS | C0005750 |
| MedGen | 600 |
| Is cancer (heuristic) | no |
Also known as: blind loop syndrome · stasis (blind loop) syndrome · stasis syndrome
Disease family
This is a subtype of malabsorption syndrome. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › digestive system disorder › intestinal disorder › malabsorption syndrome › blind loop syndrome
Related subtypes (8): tropical sprue, intestinal disaccharidase deficiency, celiac disease, hereditary folate malabsorption, lysine malabsorption syndrome, idiopathic malabsorption due to bile acid synthesis defects, autoimmune enteropathy, lactose intolerance
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 2.
Phase distribution (across all retrieved trials)
| Phase | Trials |
|---|---|
| Not specified | 2 |
Top trials by phase / activity
| NCT | Phase | Status | Title |
|---|---|---|---|
| NCT05938439 | Not specified | RECRUITING | Evaluation of the Performance of MAgnetic Gastrointestinal Universal Septotome for Treatment of Candy Cane Syndrome |
| NCT02819037 | Not specified | COMPLETED | Small Intestinal Bacterial Overgrowth Obese |
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.