Branch retinal artery occlusion

disease
On this page

Also known as retinal arterial branch occlusion

Summary

Branch retinal artery occlusion (MONDO:0001630) is a disease and 3 clinical trials. Top therapeutic interventions include oxygen. A subtype of retinal artery occlusion — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 3

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namebranch retinal artery occlusion
Mondo IDMONDO:0001630
DOIDDOID:13094
NCITC34436
SNOMED CT50821009
UMLSC0006123
MedGen14217
Is cancer (heuristic)no

Also known as: retinal arterial branch occlusion

Disease family

This is a subtype of retinal artery occlusion. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disorderretinal disorderretinal vascular disorderretinal vascular occlusionretinal artery occlusionbranch retinal artery occlusion

Related subtypes (3): central retinal artery occlusion, partial arterial retinal occlusion, transient retinal arterial occlusion

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 3.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified2
PHASE41

Top trials by phase / activity

NCTPhaseStatusTitle
NCT07240649PHASE4NOT_YET_RECRUITINGOutcomes From Hyperbaric Oxygen (HBO2) Treatment for Emerging Indications
NCT03475173Not specifiedRECRUITINGNew Non-invasive Modalities for Assessing Retinal Structure and Function
NCT01348633Not specifiedUNKNOWNRetinal Oxygen Saturation, Blood Flow, Vascular Function and High Resolution Morphometric Imaging in the Living Human Eye

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
OXYGEN41