Breast capillary hemangioma

Summary

Breast capillary hemangioma (MONDO:0003896) is a disease. A subtype of capillary hemangioma — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	breast capillary hemangioma
Mondo ID	MONDO:0003896
DOID	DOID:6491
NCIT	C5210
UMLS	C1332619
MedGen	231963
Anatomy (UBERON)	UBERON:0000310
Is cancer (heuristic)	no

Also known as: breast capillary angioma · breast capillary hemangioma · capillary angioma of breast · capillary angioma of the breast · capillary hemangioma of breast · capillary hemangioma of the breast

Disease family

This is a subtype of capillary hemangioma. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › benign neoplasm › cardiovascular organ benign neoplasm › benign blood vessel neoplasm › hemangioma › capillary hemangioma › breast capillary hemangioma

Related subtypes (5): cherry hemangioma, capillary infantile hemangioma, hemangioblastoma, eyelid capillary hemangioma, pyogenic granuloma

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.