Bullous impetigo

disease
On this page

Summary

Bullous impetigo (MONDO:0018182) is a disease. A subtype of staphylococcal toxemia — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide)
  • Phenotypes (HPO): 8

Clinical features

Signs & symptoms

Clinical features (HPO)

8 HPO clinical features (Orphanet curated; top 8 by frequency):

HPO IDTermFrequency
HP:0005406Recurrent bacterial skin infectionsVery frequent (80-99%)
HP:0008066Abnormal blistering of the skinVery frequent (80-99%)
HP:0010783ErythemaVery frequent (80-99%)
HP:0200039PustuleVery frequent (80-99%)
HP:0003095Septic arthritisOccasional (5-29%)
HP:0100763Abnormality of the lymphatic systemOccasional (5-29%)
HP:0100806SepsisOccasional (5-29%)
HP:0100820GlomerulopathyOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical namebullous impetigo
Mondo IDMONDO:0018182
Orphanet36237
ICD-10-CML01.03
ICD-111398484288
SNOMED CT399183005
UMLSC0021100
MedGen507838
GARD0018820
MedDRA10006563
Is cancer (heuristic)no

Disease family

This is a subtype of staphylococcal toxemia. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious diseasebacterial infectious diseasestaphylococcal infectionstaphylococcal toxemiabullous impetigo

Related subtypes (4): staphylococcal scarlet fever, staphylococcal scalded skin syndrome, staphylococcal necrotizing pneumonia, staphylococcal toxic-shock syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.