Cerebral malaria

disease
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Summary

Cerebral malaria (MONDO:0005625) is a disease and 7 clinical trials. Top therapeutic interventions include quinine, mannitol, and sorbitol. A subtype of malaria — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 7

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namecerebral malaria
Mondo IDMONDO:0005625
EFOEFO:0006857
MeSHD016779
DOIDDOID:14069
NCITC128373
SNOMED CT53622003
UMLSC0024534
MedGen44258
GARD0024213
Anatomy (UBERON)UBERON:0000955
Is cancer (heuristic)no

Disease family

This is a subtype of malaria. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious diseaseparasitic infectious diseaseprotozoa infectious diseasemalariacerebral malaria

Related subtypes (6): Plasmodium ovale malaria, Plasmodium malariae malaria, mixed malaria, blackwater fever, Plasmodium falciparum malaria, Plasmodium vivax malaria

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 7.

Phase distribution (across all retrieved trials)

PhaseTrials
PHASE33
PHASE2/PHASE31
PHASE1/PHASE21
PHASE21
Not specified1

Top trials by phase / activity

NCTPhaseStatusTitle
NCT00113854PHASE3UNKNOWNMannitol as Adjunct Therapy for Childhood Cerebral Malaria
NCT00124267PHASE3UNKNOWNEfficacy of Intrarectal Versus Intravenous Quinine for the Treatment of Childhood Cerebral Malaria
NCT00697164PHASE2/PHASE3UNKNOWNRandomized Trial of Erythropoietin During Cerebral Malaria
NCT01589289PHASE3COMPLETEDRapid Diagnostic Tests and Clinical/Laboratory Predictors of Tropical Diseases in Neurological Disorders in DRC
NCT01660672PHASE1/PHASE2COMPLETEDEnteral Levetiracetam For Seizure Control In Pediatric Cerebral Malaria
NCT01982812PHASE2COMPLETEDA Safety and Feasibility Study of Enteral LVT vs. Standard of Care for Seizure Control in Pediatric CM
NCT06915285Not specifiedCOMPLETEDAutomated Detection of Malarial Retinopathy in Patients Diagnosed With Cerebral Malaria

Drugs tested across these trials (top 30)

MoleculeMax phaseTrials referencing
QUININE42
MANNITOL41
SORBITOL41