Childhood carcinoid tumor

Summary

Childhood carcinoid tumor (MONDO:0022642) is a cancer. A subtype of carcinoid tumor — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Classification: Cancer

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	childhood carcinoid tumor
Mondo ID	MONDO:0022642
NCIT	C118810
UMLS	C3899673
MedGen	859267
GARD	0009315
Is cancer (heuristic)	yes

Also known as: carcinoid tumor (disease) of childhood · carcinoid tumor childhood · carcinoid tumour (disease) of childhood · carcinoid tumour childhood · childhood carcinoid tumor · childhood carcinoid tumor (disease) · childhood carcinoid tumour (disease) · paediatric carcinoid tumour (disease) · pediatric carcinoid tumor (disease)

Disease family

This is a subtype of carcinoid tumor. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › endocrine gland neoplasm › neuroendocrine neoplasm › carcinoid tumor › childhood carcinoid tumor

Related subtypes (6): lung carcinoid tumor, atypical carcinoid tumor, gastric neuroendocrine tumor G1, somatostatinoma, intestinal neuroendocrine tumor G1, pancreatic neuroendocrine tumor G1

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.