Congenital herpes virus infection

Summary

Congenital herpes virus infection (MONDO:0042971) is a disease. A subtype of Herpesviridae infectious disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	congenital herpes virus infection
Mondo ID	MONDO:0042971
SNOMED CT	715337002
UMLS	C4275250
MedGen	908142
GARD	0002669
Is cancer (heuristic)	no

Also known as: congenital herpes virus infection · congenital infection caused by herpes virus · Herpesviridae caused infectious embryofetopathy · Herpesviridae infectious embryofetopathy

Disease family

This is a subtype of Herpesviridae infectious disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › viral infectious disease › primary viral infectious disease › Herpesviridae infectious disease › congenital herpes virus infection

Related subtypes (7): herpetic gastritis, herpes simplex infectious disease, Epstein-Barr virus infection, cytomegalovirus infection, human herpesvirus 8 infection, varicella zoster infection, roseolovirus infectious disease

Subtypes (2): congenital herpes simplex virus infection, fetal cytomegalovirus syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.