Conventional angiosarcoma

Summary

Conventional angiosarcoma (MONDO:0003025) is a disease. A subtype of angiosarcoma — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	conventional angiosarcoma
Mondo ID	MONDO:0003025
DOID	DOID:4512
NCIT	C9426
UMLS	C1333155
MedGen	234174
GARD	0023332
Is cancer (heuristic)	no

Also known as: conventional angiosarcoma

Disease family

This is a subtype of angiosarcoma. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › cancer › cardiovascular cancer › vascular cancer › angiosarcoma › conventional angiosarcoma

Related subtypes (18): spleen angiosarcoma, liver angiosarcoma, bone angiosarcoma, central nervous system angiosarcoma, pediatric angiosarcoma, aorta angiosarcoma, breast angiosarcoma, gallbladder angiosarcoma, thyroid gland angiosarcoma, skin angiosarcoma, superior vena cava angiosarcoma, prostate angiosarcoma, mediastinum angiosarcoma, ovarian angiosarcoma, pulmonary vein leiomyosarcoma, pulmonary artery leiomyosarcoma, Kaposi’s sarcoma, angiosarcoma of the scalp

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.