Cutaneous mucoepidermoid carcinoma
diseaseOn this page
Also known as zone of skin mucoepidermoid carcinoma
Summary
Cutaneous mucoepidermoid carcinoma (MONDO:0003091) is a cancer. A subtype of skin carcinoma — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
At a glance
- Classification: Cancer
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | cutaneous mucoepidermoid carcinoma |
| Mondo ID | MONDO:0003091 |
| DOID | DOID:4683 |
| NCIT | C4472 |
| SNOMED CT | 254713002 |
| UMLS | C0346019 |
| MedGen | 91072 |
| GARD | 0023364 |
| Anatomy (UBERON) | UBERON:0000014 |
| Is cancer (heuristic) | yes |
Also known as: zone of skin mucoepidermoid carcinoma
Disease family
This is a subtype of skin carcinoma. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › integumentary system disorder › integumentary system cancer › skin cancer › skin carcinoma › cutaneous mucoepidermoid carcinoma
Related subtypes (12): labia minora carcinoma, labia majora carcinoma, skin squamous cell carcinoma, cutaneous Paget disease, anal margin carcinoma, eyelid carcinoma, Borst-Jadassohn intraepidermal carcinoma, skin carcinoma in situ, skin basal cell carcinoma, vulvar seborrheic keratosis, skin appendage carcinoma, cutaneous neuroendocrine carcinoma
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.