Dermatophytosis of scalp or beard

Summary

Dermatophytosis of scalp or beard (MONDO:0002967) is a disease. A subtype of dermatophytosis — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	dermatophytosis of scalp or beard
Mondo ID	MONDO:0002967
DOID	DOID:4337
ICD-10-CM	B35.0
NCIT	C34536
SNOMED CT	266148000
UMLS	C0011640
MedGen	4237
Anatomy (UBERON)	UBERON:0000403
Is cancer (heuristic)	no

Also known as: dermatophytosis of scalp and beard · scalp dermatophytosis · scalp ringworm

Disease family

This is a subtype of dermatophytosis. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › fungal infectious disease › cutaneous mycosis › tinea infection › dermatophytosis › dermatophytosis of scalp or beard

Related subtypes (10): tinea barbae, ectothrix infectious disease, endothrix infectious disease, dermatophytosis of groin and perianal area, tinea corporis, tinea unguium, tinea profunda, tinea manuum, tinea pedis, Majocchi granuloma

Subtypes (1): Kerion celsi

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.