Dissecting cellulitis of the scalp

Summary

Dissecting cellulitis of the scalp (MONDO:0009848) is a disease. A subtype of subcutaneous tissue disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Prevalence: Unknown (Worldwide)
• Phenotypes (HPO): 7

Clinical features

Signs & symptoms

Clinical features (HPO)

7 HPO clinical features (Orphanet curated; top 7 by frequency):

HPO ID	Term	Frequency
HP:0000969	Edema	Very frequent (80-99%)
HP:0000989	Pruritus	Very frequent (80-99%)
HP:0001482	Subcutaneous nodule	Very frequent (80-99%)
HP:0001581	Recurrent skin infections	Very frequent (80-99%)
HP:0001595	Abnormality of the hair	Very frequent (80-99%)
HP:0100658	Cellulitis	Very frequent (80-99%)
HP:0100809	Scalp tenderness	Very frequent (80-99%)

Identifiers

Disease identifiers

Field	Value
Canonical name	dissecting cellulitis of the scalp
Mondo ID	MONDO:0009848
MeSH	C562486
OMIM	260910
Orphanet	345
ICD-11	872245808
SNOMED CT	77333008
UMLS	C0263506
MedGen	78097
GARD	0001883
MedDRA	10056961
Is cancer (heuristic)	no

Also known as: dissecting cellulitis of the scalp

Disease family

This is a subtype of subcutaneous tissue disorder. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › integumentary system disorder › subcutaneous tissue disorder › dissecting cellulitis of the scalp

Related subtypes (18): nodular fasciitis, hemangioma of subcutaneous tissue, adiposis dolorosa, isolated anhidrosis with normal sweat glands, fibrodysplasia ossificans progressiva, multiple symmetric lipomatosis, familial multiple lipomatosis, familial angiolipomatosis, cerebrotendinous xanthomatosis, Pierpont syndrome, CLOVES syndrome, encephalocraniocutaneous lipomatosis, Lipedema, FLOTCH syndrome, mycetoma, nodular non-suppurative panniculitis, Roch-Leri mesosomatous lipomatosis, cytophagic histiocytic panniculitis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.