Ehlers-Danlos syndrome, musculocontractural type 2

disease
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Also known as DSE Ehlers-Danlos syndrome, musculocontractural typeEDSMC2Ehlers-Danlos syndrome, musculocontractural type caused by mutation in DSE

Summary

Ehlers-Danlos syndrome, musculocontractural type 2 (MONDO:0014236) is a disease caused by DSE (GenCC Definitive), with 1 cohort gene.

At a glance

  • Causal gene: DSE (GenCC Definitive)
  • Cohort genes: 1
  • ClinVar variants: 303

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameEhlers-Danlos syndrome, musculocontractural type 2
Mondo IDMONDO:0014236
OMIM615539
DOIDDOID:0080735, DOID:0080737
UMLSC3809845
MedGen816175
GARD0015982
Is cancer (heuristic)no

Also known as: DSE Ehlers-Danlos syndrome, musculocontractural type · EDSMC2 · Ehlers-Danlos syndrome, musculocontractural type 2 · Ehlers-Danlos syndrome, musculocontractural type caused by mutation in DSE

Data availability: 303 ClinVar variants · 6 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary diseaseinborn errors of metabolismcongenital disorder of glycosylationEhlers-Danlos syndrome, musculocontractural typeEhlers-Danlos syndrome, musculocontractural type 2

Related subtypes (1): Ehlers-Danlos syndrome, musculocontractural type 1

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

303 retrieved; paginated sample, class counts are floors:

143 likely benign, 124 uncertain significance, 13 conflicting classifications of pathogenicity, 10 benign/likely benign, 6 pathogenic, 5 benign, 2 likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
2119102NM_013352.4(DSE):c.480del (p.Ala161fs)DSEPathogeniccriteria provided, single submitter
3773661NM_013352.4(DSE):c.874C>T (p.Gln292Ter)DSEPathogeniccriteria provided, single submitter
446171NM_013352.4(DSE):c.799A>G (p.Arg267Gly)DSEPathogenicno assertion criteria provided
4807721NM_013352.4(DSE):c.406C>T (p.Gln136Ter)DSEPathogeniccriteria provided, single submitter
4809221NM_013352.4(DSE):c.811del (p.Gln271fs)DSEPathogeniccriteria provided, single submitter
88848NM_013352.4(DSE):c.803C>T (p.Ser268Leu)DSEPathogenicno assertion criteria provided
1708660NM_013352.4(DSE):c.416+1G>ADSELikely pathogeniccriteria provided, multiple submitters, no conflicts
3592992NM_013352.4(DSE):c.2097G>A (p.Trp699Ter)DSELikely pathogeniccriteria provided, single submitter
1053091NM_013352.4(DSE):c.2186G>A (p.Ser729Asn)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1200367NM_013352.4(DSE):c.2635C>T (p.Arg879Trp)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1702235NM_013352.4(DSE):c.189G>A (p.Ser63=)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1702236NM_013352.4(DSE):c.2184G>A (p.Lys728=)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
391864NM_013352.4(DSE):c.359T>C (p.Ile120Thr)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
392192NM_013352.4(DSE):c.1004G>A (p.Arg335His)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
450138NM_013352.4(DSE):c.1897G>A (p.Val633Met)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
452829NM_013352.4(DSE):c.2168G>A (p.Arg723Gln)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
508454NM_013352.4(DSE):c.2601C>T (p.Tyr867=)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
511744NM_013352.4(DSE):c.450G>A (p.Pro150=)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
541546NM_013352.4(DSE):c.2005A>G (p.Ile669Val)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
739017NM_013352.4(DSE):c.1961G>A (p.Arg654Gln)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
752550NM_013352.4(DSE):c.1381G>A (p.Ala461Thr)DSEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1000265NM_013352.4(DSE):c.121G>A (p.Asp41Asn)DSEUncertain significancecriteria provided, multiple submitters, no conflicts
1002117NM_013352.4(DSE):c.110A>G (p.Asn37Ser)DSEUncertain significancecriteria provided, multiple submitters, no conflicts
1019817NM_013352.4(DSE):c.325T>G (p.Leu109Val)DSEUncertain significancecriteria provided, single submitter
1030280NM_013352.4(DSE):c.2215G>T (p.Ala739Ser)DSEUncertain significancecriteria provided, single submitter
1030281NM_013352.4(DSE):c.2761A>G (p.Thr921Ala)DSEUncertain significancecriteria provided, multiple submitters, no conflicts
1030282NM_013352.4(DSE):c.364G>A (p.Ala122Thr)DSEUncertain significancecriteria provided, multiple submitters, no conflicts
1030283NM_013352.4(DSE):c.95T>A (p.Met32Lys)DSEUncertain significancecriteria provided, single submitter
1054354NM_013352.4(DSE):c.53G>A (p.Cys18Tyr)DSEUncertain significancecriteria provided, single submitter
1055608NM_013352.4(DSE):c.277G>A (p.Asp93Asn)DSEUncertain significancecriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 7 · Orphanet: 1 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
DSEDefinitiveAutosomal recessiveEhlers-Danlos syndrome, musculocontractural type 27

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
DSEOrphanet:2953Musculocontractural Ehlers-Danlos syndrome

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
DSEHGNC:21144ENSG00000111817Q9UL01Dermatan-sulfate epimerasegencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
DSEDermatan-sulfate epimeraseConverts D-glucuronic acid to L-iduronic acid (IdoUA) residues.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 0 · Druggable fraction: 1.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)112.0×0.083

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
DSEEnzyme (other)yes5.1.3.19Chondroitin_lyas, Dermatan-Sulfate_Isomerase

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
calcaneal tendon1
germinal epithelium of ovary1
parietal pleura1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
DSE275ubiquitousmarkerparietal pleura, calcaneal tendon, germinal epithelium of ovary

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
DSE467

Structural data

PDB: 1 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
DSEQ9UL011

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 1 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
DS-GAG biosynthesis1951.7×0.001DSE

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
dermatan sulfate proteoglycan metabolic process15617.3×5e-04DSE
chondroitin sulfate proteoglycan metabolic process14213.0×5e-04DSE
dermatan sulfate proteoglycan biosynthetic process11685.2×8e-04DSE
heparan sulfate proteoglycan biosynthetic process1561.7×0.002DSE

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 0 of 1 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
DSE00

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
DSE5.1.3.19chondroitin-glucuronate 5-epimerase

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1DSE
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug0

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
DSE0

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

  • Cohort genes: DSE