epithelial predominant Wilms' tumor

Summary

epithelial predominant Wilms’ tumor (MONDO:0003322) is a cancer. A subtype of kidney Wilms tumor — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Classification: Cancer

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	epithelial predominant Wilms’ tumor
Mondo ID	MONDO:0003322
DOID	DOID:5189
NCIT	C9146
UMLS	C0279608
MedGen	76000
GARD	0023448
Is cancer (heuristic)	yes

Also known as: epithelial predominant kidney adenosarcoma · epithelial predominant kidney Wilms tumor · epithelial predominant kidney Wilms tumour · epithelial predominant nephroblastoma · epithelial predominant renal adenosarcoma · epithelial predominant renal Wilm’s tumor · epithelial predominant renal Wilm’s tumour · epithelial predominant renal Wilms tumor · epithelial predominant renal Wilms’ tumor · epithelial predominant renal Wilms’ tumour · epithelial predominant Wilms tumor · epithelial predominant Wilms tumour

Disease family

This is a subtype of kidney Wilms tumor. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › cancer › malignant urinary system neoplasm › kidney cancer › kidney Wilms tumor › epithelial predominant Wilms’ tumor

Related subtypes (7): nonanaplastic kidney Wilms tumor, metachronous kidney Wilms’ tumor, mixed cell type kidney Wilms’ tumor, blastema predominant kidney Wilms tumor, stromal predominant kidney Wilms tumor, adult kidney Wilms tumor, childhood kidney Wilms tumor

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.