Epsilon-trimethyllysine hydroxylase deficiency

disease
On this page

Also known as autism, susceptibility to, X-linked 6, X-linked recessiveAUTSX6TMLHED

Summary

Epsilon-trimethyllysine hydroxylase deficiency (MONDO:0010469) is a disease with 2 cohort genes.

At a glance

  • Cohort genes: 2
  • ClinVar variants: 17

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameepsilon-trimethyllysine hydroxylase deficiency
Mondo IDMONDO:0010469
OMIM300872
UMLSC3550875
MedGen763789
Is cancer (heuristic)no

Also known as: autism, susceptibility to, X-linked 6, X-linked recessive · AUTSX6 · epsilon-trimethyllysine hydroxylase deficiency · TMLHED

Data availability: 17 ClinVar variants · 2 GenCC gene-disease records.

Disease family

Classification path: disease susceptibility › inherited disease susceptibilityautism, susceptiblity toepsilon-trimethyllysine hydroxylase deficiency

Related subtypes (25): autism, susceptibility to, X-linked 1, autism, susceptibility to, X-linked 2, autism, susceptibility to, X-linked 3, autism, susceptibility to, X-linked 4, autism, susceptibility to, X-linked 5, intellectual developmental disorder with autism and speech delay, autism, susceptibility to, 8, autism, susceptibility to, 3, autism, susceptibility to, 6, autism, susceptibility to, 7, autism, susceptibility to, 11, autism, susceptibility to, 12, autism, susceptibility to, 13, autism, susceptibility to, 9, autism, susceptibility to, 10, autism, susceptibility to, 15, autism, susceptibility to, 16, autism, susceptibility to, 17, intellectual developmental disorder with autism and macrocephaly, autism, susceptibility to, 19, autism, susceptibility to, 20, autism, susceptibility to, 14a, autism, susceptibility to, 14b, autism, susceptibility to, 1, autism, susceptibility to, 4

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

17 retrieved; paginated sample, class counts are floors:

11 uncertain significance, 2 likely pathogenic, 2 risk factor, 1 pathogenic, 1 conflicting classifications of pathogenicity

ClinVarVariant (HGVS)GeneClassificationReview
225231NM_018196.4(TMLHE):c.961_962del (p.Ile321fs)TMLHEPathogeniccriteria provided, multiple submitters, no conflicts
1299348NM_018196.4(TMLHE):c.638+2delTMLHELikely pathogenicno assertion criteria provided
4845897NM_018196.4(TMLHE):c.224G>A (p.Trp75Ter)TMLHELikely pathogeniccriteria provided, single submitter
31938NC_000023.11:g.(155524633_155545096)_(155545276_155612791)delSPRY3risk factorno assertion criteria provided
225229NM_018196.4(TMLHE):c.730G>C (p.Asp244His)TMLHErisk factorno assertion criteria provided
372796NM_018196.4(TMLHE):c.359-2A>GTMLHEConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1342626NM_018196.4(TMLHE):c.638+83_995+2127delTMLHEUncertain significancecriteria provided, single submitter
1685178NM_018196.4(TMLHE):c.158G>A (p.Trp53Ter)TMLHEUncertain significancecriteria provided, multiple submitters, no conflicts
1691494NM_018196.4(TMLHE):c.491C>T (p.Ser164Leu)TMLHEUncertain significancecriteria provided, single submitter
1709697NM_018196.4(TMLHE):c.523G>A (p.Glu175Lys)TMLHEUncertain significancecriteria provided, multiple submitters, no conflicts
225228NM_018196.4(TMLHE):c.229C>T (p.Arg77Ter)TMLHEUncertain significancecriteria provided, multiple submitters, no conflicts
225230NM_018196.4(TMLHE):c.1107G>T (p.Glu369Asp)TMLHEUncertain significancecriteria provided, multiple submitters, no conflicts
3382623NM_018196.4(TMLHE):c.995+2T>CTMLHEUncertain significancecriteria provided, single submitter
4081954NM_018196.4(TMLHE):c.639-10_758+10delTMLHEUncertain significanceno assertion criteria provided
450888NM_018196.4(TMLHE):c.794C>T (p.Thr265Ile)TMLHEUncertain significancecriteria provided, multiple submitters, no conflicts
828134NM_018196.4(TMLHE):c.6G>T (p.Trp2Cys)TMLHEUncertain significanceno assertion criteria provided
982862NM_018196.4(TMLHE):c.278G>A (p.Arg93His)TMLHEUncertain significancecriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 2 · Orphanet: 0 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
TMLHELimitedUnknownepsilon-trimethyllysine hydroxylase deficiency2

Cohort genes → proteins

2 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
TMLHEHGNC:18308ENSG00000185973Q9NVH6Trimethyllysine dioxygenase, mitochondrialgencc,clinvar
SPRY3HGNC:11271ENSG00000168939O43610Protein sprouty homolog 3clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
TMLHETrimethyllysine dioxygenase, mitochondrialConverts trimethyllysine (TML) into hydroxytrimethyllysine (HTML).
SPRY3Protein sprouty homolog 3Inhibits neurite branching, arbor length and neurite complexity.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.5

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)16.0×0.320
Other/Unknown10.9×0.805

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
TMLHEEnzyme (other)yes1.14.11.8TauD/TfdA-like, GBBH-like_N, Trimethyllysine_dOase
SPRY3Other/UnknownnoSprouty, Sprouty_domain

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
hindlimb stylopod muscle1
muscle of leg1
skeletal muscle tissue1
cortical plate1
male germ line stem cell (sensu Vertebrata) in testis1
pancreatic ductal cell1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
TMLHE136ubiquitousmarkerskeletal muscle tissue, hindlimb stylopod muscle, muscle of leg
SPRY3162broadyespancreatic ductal cell, male germ line stem cell (sensu Vertebrata) in testis, cortical plate

Protein interactions among cohort

Intra-cohort edges: 1.

Hub genes (top 10 by interactor count)

SymbolInteractor count
TMLHE1,629
SPRY3734

Intra-cohort edges

ABSources
SPRY3TMLHEstring_interaction

Structural data

PDB: 0 · AlphaFold-only: 2 · No structure: 0

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
TMLHEQ9NVH689.10
SPRY3O4361063.71

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 2 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Carnitine synthesis12855.0×4e-04TMLHE

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
negative regulation of neuron projection arborization18426.0×9e-04SPRY3
carnitine biosynthetic process11685.2×0.002TMLHE
negative regulation of fibroblast growth factor receptor signaling pathway1526.6×0.005SPRY3
animal organ development1366.4×0.005SPRY3
negative regulation of Ras protein signal transduction1337.0×0.005SPRY3
negative regulation of MAPK cascade1150.5×0.009SPRY3
negative regulation of ERK1 and ERK2 cascade1108.0×0.011SPRY3
nervous system development123.0×0.043SPRY3

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 2

Druggability breadth: 0 of 2 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
TMLHE00
SPRY300

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
TMLHE1.14.11.8trimethyllysine dioxygenase

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug1TMLHE
EDifficult family or no structure, no drug1SPRY3

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
TMLHE0
SPRY30

Clinical trials & evidence

Clinical trials

Clinical trials: 0.