Familial juvenile hyperuricemic nephropathy type 2

disease
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Also known as ADTKD-RENautosomal dominant tubulointerstitial kidney disease due to mutations in RENfamilial juvenile hyperuricemic nephropathy caused by mutation in RENFJHN type 2HNFJ2hyperuricemic nephropathy, familial juvenile, 2hyperuricemic nephropathy, familial juvenile, type 2REN familial juvenile hyperuricemic nephropathyREN-associated familial juvenile hyperuricemic nephropathyREN-associated FJHNREN-associated kidney diseasetubulointerstitial kidney disease, autosomal dominant, 4

Summary

Familial juvenile hyperuricemic nephropathy type 2 (MONDO:0013128) is a disease caused by REN (GenCC Definitive), with 2 cohort genes.

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Causal gene: REN (GenCC Definitive)
  • Cohort genes: 2
  • ClinVar variants: 98

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families35WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Identifiers

Disease identifiers

FieldValue
Canonical namefamilial juvenile hyperuricemic nephropathy type 2
Mondo IDMONDO:0013128
MeSHC567760
OMIM613092
Orphanet217330
DOIDDOID:0061119
SNOMED CT721840000
UMLSC2751310
MedGen414347
GARD0013461
Is cancer (heuristic)no

Also known as: ADTKD-REN · autosomal dominant tubulointerstitial kidney disease due to mutations in REN · familial juvenile hyperuricemic nephropathy caused by mutation in REN · familial juvenile hyperuricemic nephropathy type 2 · FJHN type 2 · HNFJ2 · hyperuricemic nephropathy, familial juvenile, 2 · hyperuricemic nephropathy, familial juvenile, type 2 · REN familial juvenile hyperuricemic nephropathy · REN-associated familial juvenile hyperuricemic nephropathy · REN-associated FJHN · REN-associated kidney disease · tubulointerstitial kidney disease, autosomal dominant, 4

Data availability: 98 ClinVar variants · 4 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › disease of genetic or genomic mechanism › hereditary disease › inherited kidney disorder › familial juvenile hyperuricemic nephropathyfamilial juvenile hyperuricemic nephropathy type 2

Related subtypes (5): familial juvenile hyperuricemic nephropathy type 1, hyperuricemic nephropathy, familial juvenile type 3, hyperuricemic nephropathy, familial juvenile type 4, tubulointerstitial kidney disease, autosomal dominant, 2, tubulointerstitial kidney disease, autosomal dominant 6

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

98 retrieved; paginated sample, class counts are floors:

58 uncertain significance, 18 conflicting classifications of pathogenicity, 5 likely pathogenic, 4 pathogenic/likely pathogenic, 4 benign, 4 benign/likely benign, 3 pathogenic, 2 likely benign

ClinVarVariant (HGVS)GeneClassificationReview
13125NM_000537.4(REN):c.36GCT[3] (p.Leu16del)LOC107548112Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
13126NM_000537.4(REN):c.47T>G (p.Leu16Arg)LOC107548112Pathogenicno assertion criteria provided
4531376NM_000537.4(REN):c.49T>C (p.Trp17Arg)LOC107548112Pathogeniccriteria provided, single submitter
974505NM_000537.4(REN):c.58T>C (p.Cys20Arg)LOC107548112Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1067090NM_000537.4(REN):c.249+1G>ARENPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
13123NM_000537.4(REN):c.145C>T (p.Arg49Ter)RENPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
50210NM_000537.4(REN):c.127C>T (p.Arg43Ter)RENPathogeniccriteria provided, single submitter
4074750NM_000537.4(REN):c.30G>A (p.Trp10Ter)LOC107548112Likely pathogeniccriteria provided, single submitter
438681NM_000537.4(REN):c.47T>A (p.Leu16His)LOC107548112Likely pathogenicno assertion criteria provided
3384161NM_000537.4(REN):c.1079dup (p.Leu361fs)RENLikely pathogenicno assertion criteria provided
3577981NM_000537.4(REN):c.1172_1173del (p.Thr391fs)RENLikely pathogeniccriteria provided, single submitter
3578102NM_000537.4(REN):c.250-1delRENLikely pathogeniccriteria provided, single submitter
1963968NM_000537.4(REN):c.95A>C (p.Lys32Thr)LOC107548112Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
873820NM_000537.4(REN):c.97C>T (p.Arg33Trp)LOC107548112Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
874781NM_000537.4(REN):c.45G>A (p.Leu15=)LOC107548112Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2358379NM_000537.4(REN):c.322T>G (p.Ser108Ala)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294950NM_000537.4(REN):c.1076A>T (p.Lys359Ile)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294951NM_000537.4(REN):c.961-12C>ARENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294952NM_000537.4(REN):c.855C>T (p.Asp285=)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294954NM_000537.4(REN):c.744C>A (p.Asp248Glu)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294955NM_000537.4(REN):c.663G>A (p.Glu221=)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294957NM_000537.4(REN):c.630C>T (p.Phe210=)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294958NM_000537.4(REN):c.492+12C>TRENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294960NM_000537.4(REN):c.398C>T (p.Ser133Leu)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294961NM_000537.4(REN):c.390C>T (p.Phe130=)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
294962NM_000537.4(REN):c.374-13G>ARENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
3578017NM_000537.4(REN):c.710C>T (p.Ser237Leu)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
753683NM_000537.4(REN):c.961-5C>ARENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
876615NM_000537.4(REN):c.1118C>T (p.Pro373Leu)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications
876616NM_000537.4(REN):c.1032G>A (p.Thr344=)RENConflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 16 · Orphanet: 2 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
KCTD11DefinitiveAutosomal dominantfamilial juvenile hyperuricemic nephropathy type 28
RENDefinitiveAutosomal dominantfamilial juvenile hyperuricemic nephropathy type 28

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
RENOrphanet:217330REN-related autosomal dominant tubulointerstitial kidney disease
RENOrphanet:97369Renal tubular dysgenesis of genetic origin

Cohort genes → proteins

2 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
KCTD11HGNC:21302ENSG00000213859Q693B1BTB/POZ domain-containing protein KCTD11gencc,clinvar
RENHGNC:9958ENSG00000143839P00797Reningencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
KCTD11BTB/POZ domain-containing protein KCTD11Plays a role as a marker and a regulator of neuronal differentiation; Up-regulated by a variety of neurogenic signals, such as retinoic acid, epidermal growth factor/EGF and NGFB/nerve growth factor.
RENReninRenin is a highly specific endopeptidase, whose only known function is to generate angiotensin I from angiotensinogen in the plasma, initiating a cascade of reactions that produce an elevation of blood pressure and increased sodium retenti…

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.5

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Protease118.3×0.108
Other/Unknown10.9×0.805

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
KCTD11Other/UnknownnoT1-type_BTB, SKP1/BTB/POZ_sf, KCTD11/21_C
RENProteaseyes3.4.23.15Aspartic_peptidase_A1, Aspartic_peptidase_AS, Aspartic_peptidase_N

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
esophagus mucosa1
lower esophagus mucosa1
tibial nerve1
adult mammalian kidney1
decidua1
stromal cell of endometrium1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
KCTD11132ubiquitousmarkerlower esophagus mucosa, tibial nerve, esophagus mucosa
REN126tissue_specificmarkerdecidua, adult mammalian kidney, stromal cell of endometrium

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
REN3,244
KCTD11420

Structural data

PDB: 1 · AlphaFold-only: 1 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
RENP0079791

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
KCTD11Q693B185.00

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 2 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Metabolism of Angiotensinogen to Angiotensins1634.4×0.002REN

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
response to cGMP14213.0×0.003REN
renin-angiotensin regulation of aldosterone production12808.7×0.003REN
juxtaglomerular apparatus development12808.7×0.003REN
neuroblast differentiation11053.2×0.006KCTD11
mesonephros development1766.0×0.006REN
amyloid-beta metabolic process1766.0×0.006REN
angiotensin maturation1648.1×0.006REN
negative regulation of neuroblast proliferation1601.9×0.006KCTD11
drinking behavior1495.6×0.006REN
response to immobilization stress1366.4×0.007REN
response to cAMP1255.3×0.009REN
regulation of MAPK cascade1227.7×0.009REN
negative regulation of smoothened signaling pathway1227.7×0.009KCTD11
cell maturation1221.7×0.009REN
hormone-mediated signaling pathway1200.6×0.009REN
neuroblast proliferation1183.2×0.009KCTD11
cellular response to xenobiotic stimulus1120.4×0.013REN
regulation of blood pressure1110.9×0.013REN
positive regulation of neuron differentiation199.1×0.014KCTD11
smoothened signaling pathway190.6×0.015KCTD11
male gonad development178.0×0.016REN
kidney development170.2×0.017REN
response to lipopolysaccharide162.4×0.018REN
protein homooligomerization161.1×0.018KCTD11
neuron differentiation150.1×0.021KCTD11
protein ubiquitination120.7×0.050KCTD11
proteolysis117.1×0.058REN

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 1 · Undrugged: 1

Druggability breadth: 2 of 2 evidence-associated genes (100%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
RENCAPTOPRIL

Top cohort targets by molecule count

SymbolMoleculesMax phase
REN134
KCTD1100

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
CAPTOPRIL4REN
ALISKIREN4REN
ALISKIREN FUMARATE4REN
SITOKIREN3REN
ZANKIREN2REN
DITEKIREN2REN
PEPSTATIN2REN
ENALKIREN2REN
REMIKIREN2REN
IMARIKIREN HYDROCHLORIDE2REN
IMARIKIREN2REN
TERLAKIREN2REN
VTP-279991REN

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
REN541Binding:472, Functional:68, ADMET:1
KCTD111Binding:1

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
REN3.4.23.15renin

Cohort genes with high screening signal

≥100 ChEMBL assays — a studied-ness signal; see Therapeutics for approved-drug status.

SymbolChEMBL assays
REN541

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

13 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
CAPTOPRIL4REN
ALISKIREN4REN
ALISKIREN FUMARATE4REN
SITOKIREN3REN
ZANKIREN2REN
DITEKIREN2REN
PEPSTATIN2REN
ENALKIREN2REN
REMIKIREN2REN
IMARIKIREN HYDROCHLORIDE2REN
IMARIKIREN2REN
TERLAKIREN2REN
VTP-279991REN

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1REN
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1KCTD11

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
KCTD111

Clinical trials & evidence

Clinical trials

Clinical trials: 0.