Fetal parvovirus syndrome

disease
On this page

Also known as fifth diseaseHuman parvovirus B19 caused infectious embryofetopathyHuman parvovirus B19 infectious embryofetopathyMaternofetal infection by parvovirusmother-to-child transmission of parvovirus syndromeParvovirus antenatal infectionParvovirus B19 antenatal infection

Summary

Fetal parvovirus syndrome (MONDO:0017453) is a disease. A subtype of infectious embryofetopathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide)
  • Phenotypes (HPO): 9

Clinical features

Signs & symptoms

Clinical features (HPO)

9 HPO clinical features (Orphanet curated; top 9 by frequency):

HPO IDTermFrequency
HP:0001541AscitesVery frequent (80-99%)
HP:0001789Hydrops fetalisVery frequent (80-99%)
HP:0001873ThrombocytopeniaVery frequent (80-99%)
HP:0001903AnemiaVery frequent (80-99%)
HP:0000478Abnormality of the eyeOccasional (5-29%)
HP:0000504Abnormality of visionOccasional (5-29%)
HP:0001511Intrauterine growth retardationOccasional (5-29%)
HP:0001639Hypertrophic cardiomyopathyOccasional (5-29%)
HP:0010880Increased nuchal translucencyOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical namefetal parvovirus syndrome
Mondo IDMONDO:0017453
MeSHC536301
Orphanet295
ICD-11648536096
SNOMED CT715197005
UMLSC2931167
MedGen443992
GARD0004236
Is cancer (heuristic)no

Also known as: fifth disease · Human parvovirus B19 caused infectious embryofetopathy · Human parvovirus B19 infectious embryofetopathy · Maternofetal infection by parvovirus · mother-to-child transmission of parvovirus syndrome · Parvovirus antenatal infection · Parvovirus B19 antenatal infection

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › infectious embryofetopathy › fetal parvovirus syndrome

Related subtypes (9): congenital syphilis, congenital toxoplasmosis, congenital rubella syndrome, congenital varicella syndrome, congenital enterovirus infection, congenital Epstein-Barr virus infection, fetal enterovirus syndrome, fetal parainfluenza virus type 3 syndrome, congenital herpes virus infection

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.