Frontal sinus inverted papilloma

Summary

Frontal sinus inverted papilloma (MONDO:0004448) is a disease. A subtype of inverted papilloma — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	frontal sinus inverted papilloma
Mondo ID	MONDO:0004448
DOID	DOID:8060
NCIT	C6842
UMLS	C1333644
MedGen	272638
GARD	0024011
Anatomy (UBERON)	UBERON:0001760
Is cancer (heuristic)	no

Also known as: frontal sinus inverted papilloma · inverted papilloma of frontal sinus · inverted papilloma of the frontal sinus

Disease family

This is a subtype of inverted papilloma. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumor › neoplastic disease or syndrome › neoplasm › epithelial neoplasm › papillary epithelial neoplasm › papilloma › inverted papilloma › frontal sinus inverted papilloma

Related subtypes (4): inverted transitional cell papilloma, ethmoid sinus inverted papilloma, sphenoid sinus inverted papilloma, maxillary sinus inverted papilloma

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.