glaucoma type 1C
diseaseOn this page
Also known as glaucoma 1, primary open angle, Cglaucoma 1C, primary open angleGLC1C
Summary
glaucoma type 1C (MONDO:0011129) is a disease. A subtype of open-angle glaucoma — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | glaucoma type 1C |
| Mondo ID | MONDO:0011129 |
| OMIM | 601682 |
| UMLS | C1866483 |
| MedGen | 356544 |
| GARD | 0002484 |
| Is cancer (heuristic) | no |
Also known as: glaucoma 1, primary open angle, C · glaucoma 1C, primary open angle · GLC1C
Disease family
This is a subtype of open-angle glaucoma. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › disorder of orbital region › eye disorder › glaucoma › open-angle glaucoma › glaucoma type 1C
Related subtypes (6): residual stage of open angle glaucoma, low tension glaucoma, glaucoma 1, open angle, P, glaucoma 1, open angle, O, juvenile open angle glaucoma, OPTN-related open angle glaucoma
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.