Hepatic infarction

Summary

Hepatic infarction (MONDO:0001787) is a disease. A subtype of hepatic vascular disorder — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	hepatic infarction
Mondo ID	MONDO:0001787
MeSH	D000081011
DOID	DOID:13738
ICD-11	1003389114
SNOMED CT	17890003
UMLS	C0151731
MedGen	508812
Is cancer (heuristic)	no

Disease family

This is a subtype of hepatic vascular disorder. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › digestive system disorder › hepatobiliary disorder › liver disorder › hepatic vascular disorder › hepatic infarction

Related subtypes (10): portal vein thrombosis, nutmeg liver, liver angiosarcoma, peliosis hepatis, portal hypertension, hepatic vein thrombosis, Budd-Chiari syndrome, hepatic veno-occlusive disease, portosinusoidal vascular disease, Fontan-associated liver disease

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.