Holoprosencephaly 11

disease
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Also known as CDON holoprosencephalyholoprosencephaly caused by mutation in CDONholoprosencephaly type 11HPE11

Summary

Holoprosencephaly 11 (MONDO:0013642) is a disease caused by CDON (GenCC Definitive), with 2 cohort genes.

At a glance

  • Causal gene: CDON (GenCC Definitive)
  • Cohort genes: 2
  • ClinVar variants: 476

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameholoprosencephaly 11
Mondo IDMONDO:0013642
OMIM614226
DOIDDOID:0110877
UMLSC3280215
MedGen481845
GARD0024938
Is cancer (heuristic)no

Also known as: CDON holoprosencephaly · holoprosencephaly 11 · holoprosencephaly caused by mutation in CDON · holoprosencephaly type 11 · HPE11

Data availability: 476 ClinVar variants · 4 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › syndromic diseaseholoprosencephalyholoprosencephaly 11

Related subtypes (16): holoprosencephaly 3, holoprosencephaly 4, holoprosencephaly 2, holoprosencephaly 1, holoprosencephaly 6, holoprosencephaly 8, holoprosencephaly 7, chromosome 1q41-q42 deletion syndrome, microform holoprosencephaly, lobar holoprosencephaly, alobar holoprosencephaly, holoprosencephaly 13, X-linked, holoprosencephaly 14, holoprosencephaly 12 with or without pancreatic agenesis, semilobar holoprosencephaly, holoprosencephaly 10

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

476 retrieved; paginated sample, class counts are floors:

242 uncertain significance, 99 likely benign, 81 benign, 33 conflicting classifications of pathogenicity, 18 benign/likely benign, 2 pathogenic, 1 likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
30748NM_001378964.1(CDON):c.2339T>A (p.Val780Glu)CDONPathogenicno assertion criteria provided
30749NM_001378964.1(CDON):c.2368A>G (p.Thr790Ala)CDONPathogenicno assertion criteria provided
30747NM_001378964.1(CDON):c.2065C>G (p.Pro689Ala)CDONLikely pathogeniccriteria provided, single submitter
1358028NM_001378964.1(CDON):c.2624G>A (p.Ser875Asn)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1398759NM_001378964.1(CDON):c.3100G>A (p.Gly1034Arg)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1491551NM_001378964.1(CDON):c.2770A>G (p.Lys924Glu)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
1496647NM_001378964.1(CDON):c.1025A>G (p.Asn342Ser)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
193724NM_001378964.1(CDON):c.1855G>C (p.Asp619His)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2072075NM_001378964.1(CDON):c.173G>A (p.Arg58His)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2108114NM_001378964.1(CDON):c.3761C>T (p.Pro1254Leu)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2144121NM_001378964.1(CDON):c.3560G>A (p.Arg1187His)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2147303NM_001378964.1(CDON):c.101A>C (p.Glu34Ala)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2153581NM_001378964.1(CDON):c.3258T>A (p.His1086Gln)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2386666NM_001378964.1(CDON):c.2383G>A (p.Val795Ile)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2439889NM_001378964.1(CDON):c.644C>G (p.Pro215Arg)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2730050NM_001378964.1(CDON):c.1619G>T (p.Arg540Ile)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2737484NM_001378964.1(CDON):c.3197_3198insATTGAATGGGAGCCTAAA (p.Leu1065_Asn1066insLysLeuAsnGlySerLeu)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2850732NM_001378964.1(CDON):c.2492A>G (p.His831Arg)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
303503NM_001378964.1(CDON):c.2159-14G>ACDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
303510NM_001378964.1(CDON):c.1847G>A (p.Arg616Gln)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
303511NM_001378964.1(CDON):c.1826A>G (p.Asn609Ser)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
3141684NM_001378964.1(CDON):c.2854G>A (p.Val952Met)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
3652383NM_001378964.1(CDON):c.2496T>G (p.Ile832Met)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
422386NM_001378964.1(CDON):c.791C>A (p.Pro264Gln)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
539750NM_001378964.1(CDON):c.1310G>A (p.Arg437His)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
702448NM_001378964.1(CDON):c.1069C>A (p.Arg357=)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
707303NM_001378964.1(CDON):c.2567A>G (p.Asn856Ser)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
842607NM_001378964.1(CDON):c.1387T>C (p.Ser463Pro)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
877739NM_001378964.1(CDON):c.3754G>C (p.Asp1252His)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications
877741NM_001378964.1(CDON):c.3602G>A (p.Gly1201Asp)CDONConflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 5 · Orphanet: 7 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
CDONDefinitiveAutosomal dominantholoprosencephaly 115

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
CDONOrphanet:220386Semilobar holoprosencephaly
CDONOrphanet:280195Septopreoptic holoprosencephaly
CDONOrphanet:280200Microform holoprosencephaly
CDONOrphanet:93924Lobar holoprosencephaly
CDONOrphanet:93925Alobar holoprosencephaly
CDONOrphanet:93926Midline interhemispheric variant of holoprosencephaly
CDONOrphanet:95496Pituitary stalk interruption syndrome

Cohort genes → proteins

2 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
CDONHGNC:17104ENSG00000064309Q4KMG0Cell adhesion molecule-related/down-regulated by oncogenesgencc,clinvar
ACRV1HGNC:127ENSG00000134940P26436Acrosomal protein SP-10clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
CDONCell adhesion molecule-related/down-regulated by oncogenesComponent of a cell-surface receptor complex that mediates cell-cell interactions between muscle precursor cells.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.5

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Antibody/Immunoglobulin114.6×0.135
Other/Unknown10.9×0.805

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
CDONAntibody/ImmunoglobulinyesIg_sub2, Ig_sub, FN3_dom
ACRV1Other/UnknownnoLY6_UPA_recep-like, Acrosomal_SP-10-like

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
calcaneal tendon1
ganglionic eminence1
ventricular zone1
left testis1
right testis1
testis1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
CDON222ubiquitousmarkerventricular zone, ganglionic eminence, calcaneal tendon
ACRV1145tissue_specificmarkerleft testis, right testis, testis

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
ACRV11,433
CDON1,065

Structural data

PDB: 1 · AlphaFold-only: 1 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
CDONQ4KMG03

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
ACRV1P2643662.66

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 7. Enrichment computed across 2 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Ligand-receptor interactions11427.5×0.005CDON
Activation of SMO1634.4×0.006CDON
Myogenesis1380.7×0.006CDON
Signaling by Hedgehog1184.2×0.009CDON
Hedgehog ‘on’ state1158.6×0.009CDON
Developmental Biology114.5×0.081CDON
Signal Transduction110.2×0.098CDON

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
positive regulation of skeletal muscle tissue development11404.3×0.004CDON
embryonic retina morphogenesis in camera-type eye11203.7×0.004CDON
embryonic body morphogenesis11053.2×0.004CDON
skeletal muscle satellite cell differentiation11053.2×0.004CDON
positive regulation of small GTPase mediated signal transduction11053.2×0.004CDON
negative regulation of biomineral tissue development1766.0×0.004CDON
cellular response to vitamin D1766.0×0.004CDON
myoblast fusion1300.9×0.008CDON
central nervous system neuron differentiation1300.9×0.008CDON
positive regulation of neuroblast proliferation1290.6×0.008CDON
cell fate specification1263.3×0.008CDON
lens development in camera-type eye1187.2×0.010CDON
neuroblast proliferation1183.2×0.010CDON
cerebral cortex development1102.8×0.016CDON
positive regulation of neuron differentiation199.1×0.016CDON
smoothened signaling pathway190.6×0.016CDON
anterior/posterior pattern specification190.6×0.016CDON
negative regulation of canonical Wnt signaling pathway158.9×0.023CDON
cell-cell adhesion150.8×0.025CDON
positive regulation of MAPK cascade140.3×0.030CDON
nervous system development123.0×0.049CDON
cell adhesion118.7×0.057CDON
spermatogenesis117.6×0.058ACRV1
positive regulation of transcription by RNA polymerase II17.4×0.130CDON

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 2

Druggability breadth: 0 of 2 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
CDON00
ACRV100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1CDON
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1ACRV1

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
CDON0
ACRV10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.