Hydatidiform mole, recurrent, 3

disease
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Also known as HYDM3

Summary

Hydatidiform mole, recurrent, 3 (MONDO:0032746) is a disease caused by MEI1 (GenCC Strong), with 1 cohort gene.

At a glance

  • Causal gene: MEI1 (GenCC Strong)
  • Cohort genes: 1
  • ClinVar variants: 11

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namehydatidiform mole, recurrent, 3
Mondo IDMONDO:0032746
OMIM618431
UMLSC5193093
MedGen1677775
GARD0018367
Is cancer (heuristic)no

Also known as: HYDM3

Data availability: 11 ClinVar variants · 1 GenCC gene-disease record.

Disease family

Classification path: disease › human disease › disease by etiologic mechanism › cancer or benign tumorneoplastic disease or syndromeneoplasmtrophoblastic neoplasmhydatidiform molehydatidiform mole, recurrent, 3

Related subtypes (4): complete hydatidiform mole, partial hydatidiform mole, invasive hydatidiform mole, hydatidiform mole, recurrent, 4

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

11 retrieved; paginated sample, class counts are floors:

5 likely pathogenic, 3 uncertain significance, 2 pathogenic, 1 likely benign

ClinVarVariant (HGVS)GeneClassificationReview
627577NM_152513.4(MEI1):c.1196+1G>AMEI1Pathogenicno assertion criteria provided
627578NM_152513.4(MEI1):c.2206del (p.Val736fs)MEI1Pathogeniccriteria provided, single submitter
2137679NM_152513.4(MEI1):c.3170-1G>CMEI1Likely pathogeniccriteria provided, multiple submitters, no conflicts
2442306NM_152513.4(MEI1):c.3772G>T (p.Asp1258Tyr)MEI1Likely pathogeniccriteria provided, multiple submitters, no conflicts
4686563NM_152513.4(MEI1):c.2339del (p.Pro780fs)MEI1Likely pathogeniccriteria provided, single submitter
4849214NM_152513.4(MEI1):c.445C>T (p.Arg149Ter)MEI1Likely pathogeniccriteria provided, single submitter
627576NM_152513.4(MEI1):c.3452G>A (p.Trp1151Ter)MEI1Likely pathogeniccriteria provided, single submitter
3065761NM_152513.4(MEI1):c.567C>A (p.Tyr189Ter)MEI1Uncertain significancecriteria provided, single submitter
3383129NM_152513.4(MEI1):c.2647C>T (p.Arg883Ter)MEI1Uncertain significancecriteria provided, single submitter
3383130NM_152513.4(MEI1):c.186G>C (p.Lys62Asn)MEI1Uncertain significancecriteria provided, single submitter
2442305NM_152513.4(MEI1):c.868CTC[1] (p.Leu291del)MEI1Likely benigncriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 2 · Orphanet: 1 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
MEI1StrongAutosomal recessivehydatidiform mole, recurrent, 32

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
MEI1Orphanet:254688Complete hydatidiform mole

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
MEI1HGNC:28613ENSG00000167077Q5TIA1Meiosis inhibitor protein 1gencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
MEI1Meiosis inhibitor protein 1Required for normal meiotic chromosome synapsis.

Protein-family classification

Druggable: 0 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Other/Unknown11.8×0.558

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
MEI1Other/UnknownnoARM-like, ARM-type_fold, Immune_Signaling-Apoptosis_Reg

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
bone marrow cell1
granulocyte1
right testis1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
MEI1178broadmarkerbone marrow cell, right testis, granulocyte

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
MEI1759

Structural data

PDB: 0 · AlphaFold-only: 1 · No structure: 0

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
MEI1Q5TIA184.09

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 0. Enrichment computed across 1 evidence-associated genes (0 with Reactome annotation).

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
meiosis I12407.4×4e-04MEI1

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 0 of 1 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
MEI100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1MEI1

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
MEI10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.