Hyperuricemia-pulmonary hypertension-renal failure-alkalosis syndrome

disease
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Also known as HUPRA syndromeHUPRAShyperuricemia, pulmonary hypertension, renal failure, and alkalosishyperuricemia, pulmonary hypertension, renal failure, and alkalosis syndrome

Summary

Hyperuricemia-pulmonary hypertension-renal failure-alkalosis syndrome (MONDO:0013458) is a disease caused by SARS2 (GenCC Strong), with 2 cohort genes.

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Causal gene: SARS2 (GenCC Strong)
  • Cohort genes: 2
  • ClinVar variants: 168

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families4WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Identifiers

Disease identifiers

FieldValue
Canonical namehyperuricemia-pulmonary hypertension-renal failure-alkalosis syndrome
Mondo IDMONDO:0013458
OMIM613845
Orphanet363694
UMLSC3151209
MedGen462559
GARD0017569
Is cancer (heuristic)no

Also known as: HUPRA syndrome · HUPRAS · hyperuricemia, pulmonary hypertension, renal failure, and alkalosis · hyperuricemia, pulmonary hypertension, renal failure, and alkalosis syndrome

Data availability: 168 ClinVar variants · 5 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › cardiovascular disordervascular disorderarterial disorderhypertensive disorderpulmonary hypertensionhyperuricemia-pulmonary hypertension-renal failure-alkalosis syndrome

Related subtypes (5): Braddock syndrome, chronic thromboembolic pulmonary hypertension, pulmonary arterial hypertension, pulmonary hypertension owing to lung disease and/or hypoxia, pulmonary hypertension, neonatal

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

168 retrieved; paginated sample, class counts are floors:

116 uncertain significance, 26 conflicting classifications of pathogenicity, 10 benign, 8 benign/likely benign, 6 likely benign, 1 pathogenic, 1 likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
242874NM_017827.4(SARS2):c.1042T>C (p.Phe348Leu)SARS2Pathogenicno assertion criteria provided
30982NM_017827.4(SARS2):c.1169A>G (p.Asp390Gly)SARS2Likely pathogeniccriteria provided, single submitter
138961NM_017827.4(SARS2):c.248C>T (p.Ser83Leu)LOC130064387Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1420911NM_017827.4(SARS2):c.17C>T (p.Ala6Val)LOC130064387Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2054564NM_017827.4(SARS2):c.176A>C (p.Asp59Ala)LOC130064387Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
215114NM_017827.4(SARS2):c.30G>A (p.Trp10Ter)LOC130064387Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
138958NM_017827.4(SARS2):c.590-5T>ASARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
215102NM_017827.4(SARS2):c.310C>T (p.Arg104Trp)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
215104NM_017827.4(SARS2):c.91A>G (p.Arg31Gly)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
215106NM_017827.4(SARS2):c.364-7C>ASARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
215107NM_017827.4(SARS2):c.390G>A (p.Gln130=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
215109NM_017827.4(SARS2):c.501G>A (p.Ala167=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2574738NM_017827.4(SARS2):c.603A>G (p.Gln201=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
329210NM_017827.4(SARS2):c.1347+10G>ASARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
329214NM_017827.4(SARS2):c.917-4C>TSARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
329216NM_017827.4(SARS2):c.888G>A (p.Leu296=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
329218NM_017827.4(SARS2):c.654-11C>GSARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
329219NM_017827.4(SARS2):c.589+4G>ASARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
329221NM_017827.4(SARS2):c.534+10G>ASARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
3583866NM_017827.4(SARS2):c.755G>A (p.Arg252His)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
432690NM_017827.4(SARS2):c.694C>T (p.Arg232Cys)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
506542NM_017827.4(SARS2):c.1315A>T (p.Thr439Ser)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
559230NM_017827.4(SARS2):c.1317C>T (p.Thr439=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
638611NM_017827.4(SARS2):c.1347G>A (p.Thr449=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
669445NM_017827.4(SARS2):c.777G>A (p.Thr259=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
894086NM_017827.4(SARS2):c.1292G>A (p.Arg431His)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
894473NM_017827.4(SARS2):c.900G>A (p.Ala300=)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
978825NM_017827.4(SARS2):c.1205G>A (p.Arg402His)SARS2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2048371NM_017827.4(SARS2):c.146G>A (p.Arg49His)LOC130064387Uncertain significancecriteria provided, multiple submitters, no conflicts
329225NM_017827.4(SARS2):c.175G>C (p.Asp59His)LOC130064387Uncertain significancecriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 5 · Orphanet: 3 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
SARS2StrongAutosomal recessivehyperuricemia-pulmonary hypertension-renal failure-alkalosis syndrome5

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
SARS2Orphanet:363694Hyperuricemia-pulmonary hypertension-renal failure-alkalosis syndrome
SARS1Orphanet:2512Autosomal recessive primary microcephaly
SARS1Orphanet:88616Autosomal recessive non-syndromic intellectual disability

Cohort genes → proteins

2 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence2

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
SARS2HGNC:17697ENSG00000104835Q9NP81Serine–tRNA ligase, mitochondrialgencc,clinvar
SARS1HGNC:10537ENSG00000031698P49591Serine–tRNA ligase, cytoplasmicclinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
SARS2Serine–tRNA ligase, mitochondrialCatalyzes the attachment of serine to tRNA(Ser).
SARS1Serine–tRNA ligase, cytoplasmicCatalyzes the attachment of serine to tRNA(Ser) in a two-step reaction: serine is first activated by ATP to form Ser-AMP and then transferred to the acceptor end of tRNA(Ser).

Protein-family classification

Druggable: 2 · Difficult: 0 · Unknown: 0 · Druggable fraction: 1.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)212.0×0.007

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
SARS2Enzyme (other)yes6.1.1.11aa-tRNA-synt_IIb, Ser-tRNA-ligase_type_1, aa-tRNA-synth_II
SARS1Enzyme (other)yes6.1.1.11aa-tRNA-synt_IIb, Ser-tRNA-ligase_type_1, aa-tRNA-synth_II

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)2
unknown0

Top tissues across cohort

TissueCohort genes
apex of heart1
metanephros cortex1
right lobe of thyroid gland1
body of pancreas1
frontal pole1
islet of Langerhans1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
SARS2138ubiquitousmarkerapex of heart, right lobe of thyroid gland, metanephros cortex
SARS1291ubiquitousmarkerislet of Langerhans, body of pancreas, frontal pole

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
SARS12,306
SARS21,998

Structural data

PDB: 2 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
SARS1P495919
SARS2Q9NP816

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 9. Enrichment computed across 2 evidence-associated genes (2 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
tRNA Aminoacylation2285.5×1e-04SARS2, SARS1
Translation262.1×0.001SARS2, SARS1
Mitochondrial tRNA aminoacylation1259.6×0.010SARS2
Cytosolic tRNA aminoacylation1219.6×0.010SARS1
Metabolism of proteins212.4×0.012SARS2, SARS1
Selenoamino acid metabolism198.5×0.015SARS1
Selenocysteine synthesis160.1×0.021SARS1
Metabolism of amino acids and derivatives133.8×0.033SARS1
Metabolism15.8×0.165SARS1

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
seryl-tRNA aminoacylation28426.0×6e-08SARS2, SARS1
mitochondrial seryl-tRNA aminoacylation18426.0×4e-04SARS2
negative regulation of vascular endothelial growth factor production18426.0×4e-04SARS1
selenocysteine incorporation1936.2×0.002SARS1
tRNA modification1300.9×0.006SARS1
cytoplasmic translation192.6×0.015SARS1
negative regulation of angiogenesis184.3×0.015SARS1
translation151.4×0.022SARS1
negative regulation of transcription by RNA polymerase II18.9×0.110SARS1

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 2

Druggability breadth: 1 of 2 evidence-associated genes (50%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
SARS200
SARS100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 2.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
SARS12ADMET:1, Binding:1

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
SARS26.1.1.11serine-tRNA ligase
SARS16.1.1.11serine-tRNA ligase

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug2SARS2, SARS1
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug0

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
SARS20
SARS12

Clinical trials & evidence

Clinical trials

Clinical trials: 0.