Ideomotor apraxia

disease
On this page

Also known as classic apraxia (finding) [ambiguous]limb-kinetic apraxia (finding)transcortical apraxia (finding)

Summary

Ideomotor apraxia (MONDO:0006800) is a disease and 3 clinical trials. A subtype of apraxia — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Clinical trials: 3

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameideomotor apraxia
Mondo IDMONDO:0006800
EFOEFO:1000980
MeSHD020240
DOIDDOID:4627
SNOMED CT229706001
UMLSC0234523
MedGen115961
MedDRA10021216
Is cancer (heuristic)no

Also known as: classic apraxia (finding) [ambiguous] · limb-kinetic apraxia (finding) · transcortical apraxia (finding)

Disease family

This is a subtype of apraxia. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disorderperceptual disordersapraxiaideomotor apraxia

Related subtypes (1): gait apraxia

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 3.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified3

Top trials by phase / activity

NCTPhaseStatusTitle
NCT00024999Not specifiedCOMPLETEDEEG and EMG Analysis of Ideomotor Apraxia
NCT00063115Not specifiedCOMPLETEDUsing fMRI to Understand the Roles of Brain Areas for Fine Hand Movements
NCT03153371Not specifiedCOMPLETEDEarly-onset Alzheimer’s Disease Phenotypes: Neuropsychology and Neural Networks

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.