Ideomotor apraxia
diseaseOn this page
Also known as classic apraxia (finding) [ambiguous]limb-kinetic apraxia (finding)transcortical apraxia (finding)
Summary
Ideomotor apraxia (MONDO:0006800) is a disease and 3 clinical trials. A subtype of apraxia — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
At a glance
- Clinical trials: 3
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | ideomotor apraxia |
| Mondo ID | MONDO:0006800 |
| EFO | EFO:1000980 |
| MeSH | D020240 |
| DOID | DOID:4627 |
| SNOMED CT | 229706001 |
| UMLS | C0234523 |
| MedGen | 115961 |
| MedDRA | 10021216 |
| Is cancer (heuristic) | no |
Also known as: classic apraxia (finding) [ambiguous] · limb-kinetic apraxia (finding) · transcortical apraxia (finding)
Disease family
This is a subtype of apraxia. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.
Classification path: disease › human disease › disease by body system or component › nervous system disorder › perceptual disorders › apraxia › ideomotor apraxia
Related subtypes (1): gait apraxia
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 3.
Phase distribution (across all retrieved trials)
| Phase | Trials |
|---|---|
| Not specified | 3 |
Top trials by phase / activity
| NCT | Phase | Status | Title |
|---|---|---|---|
| NCT00024999 | Not specified | COMPLETED | EEG and EMG Analysis of Ideomotor Apraxia |
| NCT00063115 | Not specified | COMPLETED | Using fMRI to Understand the Roles of Brain Areas for Fine Hand Movements |
| NCT03153371 | Not specified | COMPLETED | Early-onset Alzheimer’s Disease Phenotypes: Neuropsychology and Neural Networks |
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.