IgG4-related aortitis

disease
On this page

Also known as IgG4-related periaortitis

Summary

IgG4-related aortitis (MONDO:0018672) is a disease. A subtype of immunoglobulin G4-related sclerosing disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: Unknown (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 25

Clinical features

Signs & symptoms

Clinical features (HPO)

25 HPO clinical features (Orphanet curated; top 25 by frequency):

HPO IDTermFrequency
HP:0010702Increased circulating antibody levelVery frequent (80-99%)
HP:0032300Increased circulating IgG4 levelVery frequent (80-99%)
HP:0001824Weight lossFrequent (30-79%)
HP:0001945FeverFrequent (30-79%)
HP:0002027Abdominal painFrequent (30-79%)
HP:0002099AsthmaFrequent (30-79%)
HP:0002960AutoimmunityFrequent (30-79%)
HP:0003212Increased circulating IgE levelFrequent (30-79%)
HP:0003419Low back painFrequent (30-79%)
HP:0003493Antinuclear antibody positivityFrequent (30-79%)
HP:0003565Elevated erythrocyte sedimentation rateFrequent (30-79%)
HP:0011227Elevated circulating C-reactive protein concentrationFrequent (30-79%)
HP:0012393AllergyFrequent (30-79%)
HP:0012649Increased inflammatory responseFrequent (30-79%)
HP:0012727Thoracic aortic aneurysmFrequent (30-79%)
HP:0032061HypereosinophiliaFrequent (30-79%)
HP:0000126HydronephrosisOccasional (5-29%)
HP:0002647Aortic dissectionOccasional (5-29%)
HP:0004431Complement deficiencyOccasional (5-29%)
HP:0004970Ascending tubular aorta aneurysmOccasional (5-29%)
HP:0005214Intestinal obstructionOccasional (5-29%)
HP:0012303Abnormal aortic arch morphologyOccasional (5-29%)
HP:0031252Dilated left subclavian arteryOccasional (5-29%)
HP:0032230Cytoplasmic antineutrophil antibody positivityOccasional (5-29%)
HP:0430021Abnormal common carotid artery morphologyOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical nameIgG4-related aortitis
Mondo IDMONDO:0018672
Orphanet449400
ICD-11593151236
UMLSC5569008
MedGen1800431
GARD0021883
Is cancer (heuristic)no

Also known as: IgG4-related periaortitis

Disease family

This is a subtype of immunoglobulin G4-related sclerosing disease. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › immune system disorderautoimmune diseaseimmunoglobulin G4-related sclerosing diseaseIgG4-related aortitis

Related subtypes (13): autoimmune pancreatitis, IgG4-related mesenteritis, IgG4-related sclerosing cholangitis, IgG4-related kidney disease, IgG4-related pachymeningitis, IgG4-related submandibular gland disease, IgG4-related ophthalmic disorder, eosinophilic angiocentric fibrosis, primary cutaneous plasmacytosis, cutaneous pseudolymphoma, IgG4-related retroperitoneal fibrosis, IgG4-related mediastinitis, IgG4-related thyroid disease

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.