Isolated optic neuritis

disease
On this page

Also known as ION

Summary

Isolated optic neuritis (MONDO:0044688) is a disease. A subtype of autoimmune/inflammatory optic neuropathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Phenotypes (HPO): 12

Clinical features

Signs & symptoms

Clinical features (HPO)

12 HPO clinical features (Orphanet curated; top 12 by frequency):

HPO IDTermFrequency
HP:0001123Visual field defectVery frequent (80-99%)
HP:0007641DyschromatopsiaVery frequent (80-99%)
HP:0030857Eye movement-induced painVery frequent (80-99%)
HP:0032036Reduced contrast sensitivityVery frequent (80-99%)
HP:0200026Ocular painVery frequent (80-99%)
HP:0000572Visual lossFrequent (30-79%)
HP:0000603Central scotomaFrequent (30-79%)
HP:0001085PapilledemaFrequent (30-79%)
HP:0002315HeadacheFrequent (30-79%)
HP:0200057Marcus Gunn pupilFrequent (30-79%)
HP:0030786PhotopsiaOccasional (5-29%)
HP:0033342Anti-aquaporin 4 antibody positivityExcluded (0%)

Identifiers

Disease identifiers

FieldValue
Canonical nameisolated optic neuritis
Mondo IDMONDO:0044688
Orphanet499096
UMLSC5848152
MedGen1864429
GARD0022039
Is cancer (heuristic)no

Also known as: ION · ion

Disease family

An umbrella term covering 4 Mondo subtypes.

Classification path: disease › human disease › disease by body system or component › disorder of orbital regioneye disorder › autoimmune/inflammatory optic neuropathy › isolated optic neuritis

Related subtypes (4): neuromyelitis optica, chronic relapsing inflammatory optic neuropathy, recurrent idiopathic neuroretinitis, optic perineuritis

Subtypes (4): isolated optic neuritis without anti-MOG antibodies, isolated optic neuritis with anti-MOG antibodies, single isolated optic neuritis, relapsing isolated optic neuritis

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.