Klippel-Feil syndrome 1, autosomal dominant

disease
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Also known as GDF6 isolated Klippel-Feil syndromeisolated Klippel-Feil syndrome caused by mutation in GDF6KFS1Klippel-Feil Syndrome

Summary

Klippel-Feil syndrome 1, autosomal dominant (MONDO:0007306) is a disease caused by GDF6 (GenCC Definitive), with 3 cohort genes and 2 clinical trials.

At a glance

  • Causal gene: GDF6 (GenCC Definitive)
  • Cohort genes: 3
  • ClinVar variants: 452
  • Clinical trials: 2

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical nameKlippel-Feil syndrome 1, autosomal dominant
Mondo IDMONDO:0007306
MeSHC536887
OMIM118100
DOIDDOID:0080589
UMLSC1861689
MedGen396196
GARD0015049
NORD1336
Is cancer (heuristic)no

Also known as: GDF6 isolated Klippel-Feil syndrome · isolated Klippel-Feil syndrome caused by mutation in GDF6 · KFS1 · Klippel-Feil Syndrome · Klippel-Feil syndrome 1, autosomal dominant

Data availability: 452 ClinVar variants · 2 GenCC gene-disease records · 2 cell lines.

Disease family

Classification path: disease › human disease › disease by body system or component › musculoskeletal system disorderKlippel-Feil syndromeKlippel-Feil syndrome 1, autosomal dominant

Related subtypes (5): Klippel-Feil syndrome 2, autosomal recessive, Wildervanck syndrome, Klippel-Feil syndrome 3, autosomal dominant, Klippel-Feil anomaly-myopathy-facial dysmorphism syndrome, Calabro syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

452 retrieved; paginated sample, class counts are floors:

238 uncertain significance, 148 likely benign, 35 benign, 23 conflicting classifications of pathogenicity, 7 benign/likely benign, 1 likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
1679350NM_001001557.4(GDF6):c.986_1005del (p.Pro329fs)GDF6Likely pathogeniccriteria provided, single submitter
1002016NM_001001557.4(GDF6):c.536C>A (p.Pro179Gln)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1023649NM_001001557.4(GDF6):c.902A>G (p.Glu301Gly)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1094356NM_001001557.4(GDF6):c.407-3C>TGDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1400072NM_001001557.4(GDF6):c.923_928dup (p.306GA[3])GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1404300NM_001001557.4(GDF6):c.959C>G (p.Pro320Arg)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1938713NM_001001557.4(GDF6):c.219C>A (p.Asp73Glu)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1959955NM_001001557.4(GDF6):c.454G>A (p.Val152Met)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
286405NM_001001557.4(GDF6):c.725C>G (p.Ala242Gly)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2927707NM_001001557.4(GDF6):c.625C>T (p.Pro209Ser)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
364042NM_001001557.4(GDF6):c.1304C>T (p.Ala435Val)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
364044NM_001001557.4(GDF6):c.957C>A (p.Ala319=)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
364047NM_001001557.4(GDF6):c.770C>T (p.Pro257Leu)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
60534NM_001001557.4(GDF6):c.169G>C (p.Asp57His)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
707560NM_001001557.4(GDF6):c.24C>G (p.Leu8=)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
707574NM_001001557.4(GDF6):c.112G>C (p.Gly38Arg)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
836995NM_001001557.4(GDF6):c.815C>T (p.Pro272Leu)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
8371NM_001001557.4(GDF6):c.746C>A (p.Ala249Glu)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
8372NM_001001557.4(GDF6):c.866T>C (p.Leu289Pro)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
8373NM_001001557.4(GDF6):c.1271A>G (p.Lys424Arg)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
863448NM_001001557.4(GDF6):c.322G>A (p.Ala108Thr)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
914077NM_001001557.4(GDF6):c.250G>A (p.Glu84Lys)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
914078NM_001001557.4(GDF6):c.18C>T (p.Val6=)GDF6Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
39198NM_198578.4(LRRK2):c.4883G>C (p.Arg1628Pro)LRRK2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1001816NM_001001557.4(GDF6):c.1310A>G (p.Asn437Ser)GDF6Uncertain significancecriteria provided, single submitter
1002101NM_001001557.4(GDF6):c.896C>T (p.Ser299Leu)GDF6Uncertain significancecriteria provided, single submitter
1007576NM_001001557.4(GDF6):c.611C>T (p.Pro204Leu)GDF6Uncertain significancecriteria provided, single submitter
1008100NM_001001557.4(GDF6):c.377C>T (p.Thr126Met)GDF6Uncertain significancecriteria provided, multiple submitters, no conflicts
1008463NM_001001557.4(GDF6):c.272G>C (p.Arg91Pro)GDF6Uncertain significancecriteria provided, multiple submitters, no conflicts
1015372NM_001001557.4(GDF6):c.323C>A (p.Ala108Asp)GDF6Uncertain significancecriteria provided, single submitter

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 8 · Orphanet: 6 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
GDF6DefinitiveAutosomal dominantKlippel-Feil syndrome 1, autosomal dominant8

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
GDF6Orphanet:2345Isolated Klippel-Feil syndrome
GDF6Orphanet:3237Multiple synostoses syndrome
GDF6Orphanet:65Leber congenital amaurosis
GDF6Orphanet:98938Colobomatous microphthalmia
LRRK2Orphanet:2828Young-onset Parkinson disease
LRRK2Orphanet:411602Hereditary late-onset Parkinson disease

Cohort genes → proteins

3 cohort genes, 3 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence3

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
GDF6HGNC:4221ENSG00000156466Q6KF10Growth/differentiation factor 6gencc,clinvar
LRRK2HGNC:18618ENSG00000188906Q5S007Leucine-rich repeat serine/threonine-protein kinase 2clinvar
PLEKHF2HGNC:20757ENSG00000175895Q9H8W4Pleckstrin homology domain-containing family F member 2clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
GDF6Growth/differentiation factor 6Growth factor that controls proliferation and cellular differentiation in the retina and bone formation.
LRRK2Leucine-rich repeat serine/threonine-protein kinase 2Serine/threonine-protein kinase which phosphorylates a broad range of proteins involved in multiple processes such as neuronal plasticity, innate immunity, autophagy, and vesicle trafficking.
PLEKHF2Pleckstrin homology domain-containing family F member 2May play a role in early endosome fusion upstream of RAB5, hence regulating receptor trafficking and fluid-phase transport.

Protein-family classification

Druggable: 1 · Difficult: 1 · Unknown: 1 · Druggable fraction: 0.33

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Kinase19.2×0.313
Transcription factor12.8×0.482
Other/Unknown10.6×0.914

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
GDF6Other/UnknownnoTGF-b_propeptide, TGF-b_C, TGF-beta-like
LRRK2KinaseyesProt_kinase_dom, Leu-rich_rpt, Leu-rich_rpt_typical-subtyp
PLEKHF2Transcription factornoZnf_FYVE, PH_domain, Znf_FYVE_PHD

Expression context

Cohort genes with no expression data: 0.

3 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)3
unknown0

Top tissues across cohort

TissueCohort genes
placenta1
primordial germ cell in gonad1
ventricular zone1
buccal mucosa cell1
leukocyte1
monocyte1
oocyte1
secondary oocyte1
upper leg skin1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
GDF6104broadmarkerplacenta, primordial germ cell in gonad, ventricular zone
LRRK2220broadmarkerbuccal mucosa cell, monocyte, leukocyte
PLEKHF2268ubiquitousmarkersecondary oocyte, oocyte, upper leg skin

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
LRRK27,628
PLEKHF21,324
GDF61,127

Structural data

PDB: 1 · AlphaFold-only: 2 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
LRRK2Q5S00744

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
PLEKHF2Q9H8W487.36
GDF6Q6KF1070.88

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 4. Enrichment computed across 3 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
PTK6 promotes HIF1A stabilization11631.4×0.002LRRK2
Signaling by PTK61543.8×0.002LRRK2
Signaling by Non-Receptor Tyrosine Kinases1543.8×0.002LRRK2
Signal Transduction110.2×0.098LRRK2

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 3 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
cell migration involved in metanephros development15617.3×0.004GDF6
cellular response to curcumin15617.3×0.004LRRK2
Wnt signalosome assembly15617.3×0.004LRRK2
regulation of kidney size12808.7×0.004LRRK2
negative regulation of late endosome to lysosome transport12808.7×0.004LRRK2
obsolete negative regulation of protein processing involved in protein targeting to mitochondrion12808.7×0.004LRRK2
retinal cell apoptotic process12808.7×0.004GDF6
regulation of neuron maturation11872.4×0.004LRRK2
regulation of cAMP/PKA signal transduction11872.4×0.004LRRK2
positive regulation of protein autoubiquitination11872.4×0.004LRRK2
regulation of synaptic vesicle transport11872.4×0.004LRRK2
negative regulation of motile cilium assembly11872.4×0.004LRRK2
regulation of branching morphogenesis of a nerve11872.4×0.004LRRK2
regulation of dopamine receptor signaling pathway11404.3×0.004LRRK2
positive regulation of dopamine receptor signaling pathway11404.3×0.004LRRK2
regulation of retrograde transport, endosome to Golgi11404.3×0.004LRRK2
regulation of CAMKK-AMPK signaling cascade11404.3×0.004LRRK2
tangential migration from the subventricular zone to the olfactory bulb11123.5×0.005LRRK2
regulation of cell projection organization11123.5×0.005LRRK2
regulation of neuroblast proliferation11123.5×0.005LRRK2
regulation of locomotion1936.2×0.005LRRK2
regulation of mitochondrial depolarization1936.2×0.005LRRK2
positive regulation of synaptic vesicle endocytosis1936.2×0.005LRRK2
obsolete negative regulation of protein targeting to mitochondrion1936.2×0.005LRRK2
intracellular distribution of mitochondria1802.5×0.005LRRK2
cellular response to manganese ion1802.5×0.005LRRK2
cellular response to dopamine1802.5×0.005LRRK2
obsolete regulation of lysosomal lumen pH1702.2×0.005LRRK2
regulation of ER to Golgi vesicle-mediated transport1702.2×0.005LRRK2
protein localization to endoplasmic reticulum exit site1702.2×0.005LRRK2

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 1 · Undrugged: 2

Druggability breadth: 1 of 3 evidence-associated genes (33%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
LRRK2PONATINIB

Top cohort targets by molecule count

SymbolMoleculesMax phase
LRRK2424
GDF600
PLEKHF200

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
PONATINIB4LRRK2
FEDRATINIB4LRRK2
AXITINIB4LRRK2
RUXOLITINIB4LRRK2
PALBOCICLIB4LRRK2
ENTRECTINIB4LRRK2
TOFACITINIB CITRATE4LRRK2
TOFACITINIB4LRRK2
VANDETANIB4LRRK2
BOSUTINIB4LRRK2
BRIGATINIB4LRRK2
NINTEDANIB4LRRK2
SUNITINIB4LRRK2
ERLOTINIB4LRRK2
MIDOSTAURIN4LRRK2
DACTOLISIB3LRRK2
ADENINE3LRRK2
OLVEREMBATINIB3LRRK2
CANERTINIB3LRRK2
FASUDIL3LRRK2
ALVOCIDIB3LRRK2
ABIVERTINIB3LRRK2
ALISERTIB3LRRK2
DOVITINIB3LRRK2
LESTAURTINIB3LRRK2
RUBOXISTAURIN3LRRK2
SU-0148132LRRK2
REBASTINIB2LRRK2
CENISERTIB2LRRK2
ADAVOSERTIB2LRRK2

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
LRRK2809Binding:799, ADMET:7, Functional:3

Cohort genes with high screening signal

≥100 ChEMBL assays — a studied-ness signal; see Therapeutics for approved-drug status.

SymbolChEMBL assays
LRRK2809

Pharmacogenomics

Cohort genes with a PharmGKB record: 3; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

30 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
PONATINIB4LRRK2
FEDRATINIB4LRRK2
AXITINIB4LRRK2
RUXOLITINIB4LRRK2
PALBOCICLIB4LRRK2
ENTRECTINIB4LRRK2
TOFACITINIB CITRATE4LRRK2
TOFACITINIB4LRRK2
VANDETANIB4LRRK2
BOSUTINIB4LRRK2
BRIGATINIB4LRRK2
NINTEDANIB4LRRK2
SUNITINIB4LRRK2
ERLOTINIB4LRRK2
MIDOSTAURIN4LRRK2
DACTOLISIB3LRRK2
ADENINE3LRRK2
OLVEREMBATINIB3LRRK2
CANERTINIB3LRRK2
FASUDIL3LRRK2
ALVOCIDIB3LRRK2
ABIVERTINIB3LRRK2
ALISERTIB3LRRK2
DOVITINIB3LRRK2
LESTAURTINIB3LRRK2
RUBOXISTAURIN3LRRK2
SU-0148132LRRK2
REBASTINIB2LRRK2
CENISERTIB2LRRK2
ADAVOSERTIB2LRRK2

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1LRRK2
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug2GDF6, PLEKHF2

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
GDF60
PLEKHF20

Clinical trials & evidence

Clinical trials

Clinical trials: 2.

Phase distribution (across all retrieved trials)

PhaseTrials
Not specified2

Top trials by phase / activity

NCTPhaseStatusTitle
NCT03741790Not specifiedACTIVE_NOT_RECRUITINGAirway Management of Pediatric Patients With Klippel-Feil Syndrome
NCT06489392Not specifiedCOMPLETEDMehri Turki Webbed Neck Classification