long QT syndrome 5

disease
On this page

Also known as KCNE1 long QT syndromelong QT syndrome caused by mutation in KCNE1long QT syndrome type 5LQT5

Summary

long QT syndrome 5 (MONDO:0013372) is a disease caused by KCNE1 (GenCC Definitive), with 1 cohort gene.

At a glance

  • Causal gene: KCNE1 (GenCC Definitive)
  • Cohort genes: 1
  • ClinVar variants: 127

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namelong QT syndrome 5
Mondo IDMONDO:0013372
MeSHC566766
OMIM613695
DOIDDOID:0110647
NCITC172094
UMLSC1867904
MedGen358092
GARD0010433
Is cancer (heuristic)no

Also known as: KCNE1 long QT syndrome · long QT syndrome 5 · long QT syndrome caused by mutation in KCNE1 · long QT syndrome type 5 · LQT5

Data availability: 127 ClinVar variants · 3 GenCC gene-disease records · 3 cell lines.

Disease family

An umbrella term covering 1 Mondo subtype.

Classification path: disease › human disease › disease by body system or component › syndromic diseaselong QT syndromefamilial long QT syndromelong QT syndrome 5

Related subtypes (18): Jervell and Lange-Nielsen syndrome, Andersen-Tawil syndrome, cardiac arrhythmia, ankyrin-B-related, Timothy syndrome, long QT syndrome 3, long QT syndrome 9, long QT syndrome 10, long QT syndrome 11, long QT syndrome 12, long QT syndrome 13, long QT syndrome 2, long QT syndrome 6, long QT syndrome 14, long QT syndrome 15, long QT syndrome 8, long QT syndrome 16, long QT syndrome 1, long QT syndrome 4

Subtypes (1): Jervell and Lange-Nielsen syndrome 2

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

127 retrieved; paginated sample, class counts are floors:

65 uncertain significance, 22 conflicting classifications of pathogenicity, 18 benign, 10 benign/likely benign, 9 likely benign, 1 pathogenic/likely pathogenic, 1 conflicting classifications of pathogenicity; other; risk factor, 1 pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
13475NG_009091.1(KCNE1):g.[66853A>C;66857T>C]KCNE1Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
430060NM_000219.6(KCNE1):c.12dup (p.Asn5Ter)KCNE1Pathogeniccriteria provided, multiple submitters, no conflicts
1202620NM_000219.6(KCNE1):c.31_118del (p.Pro11fs)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
132657NM_000219.6(KCNE1):c.163G>A (p.Gly55Ser)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
132660NM_000219.6(KCNE1):c.199C>T (p.Arg67Cys)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
132661NM_000219.6(KCNE1):c.200G>A (p.Arg67His)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
132676NM_000219.6(KCNE1):c.292C>T (p.Arg98Trp)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
132678NM_000219.6(KCNE1):c.325G>A (p.Val109Ile)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
132679NM_000219.6(KCNE1):c.374C>T (p.Thr125Met)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
13477NM_000219.6(KCNE1):c.226G>A (p.Asp76Asn)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
13478NM_000219.6(KCNE1):c.221C>T (p.Ser74Leu)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
13479NM_000219.6(KCNE1):c.253G>A (p.Asp85Asn)KCNE1Conflicting classifications of pathogenicity; other; risk factorcriteria provided, conflicting classifications
1705696NM_000219.6(KCNE1):c.106dup (p.Arg36fs)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
3230646NM_000219.6(KCNE1):c.61G>A (p.Val21Ile)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
339748NM_000219.6(KCNE1):c.*1338C>TKCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
339760NM_000219.6(KCNE1):c.*553A>GKCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
339767NM_000219.6(KCNE1):c.*196C>GKCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
339771NM_000219.6(KCNE1):c.*30C>GKCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
339772NM_000219.6(KCNE1):c.54G>A (p.Gln18=)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
339783NM_000219.6(KCNE1):c.-377+13G>AKCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
504745NM_000219.6(KCNE1):c.-5C>AKCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
506395NM_000219.6(KCNE1):c.111C>T (p.Ser37=)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
598781NM_000219.6(KCNE1):c.166_180del (p.Phe56_Gly60del)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
644369NM_000219.6(KCNE1):c.199C>G (p.Arg67Gly)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
963591NM_000219.6(KCNE1):c.120C>T (p.Gly40=)KCNE1Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1056763NM_000219.6(KCNE1):c.80T>C (p.Met27Thr)KCNE1Uncertain significancecriteria provided, multiple submitters, no conflicts
1060040NM_000219.6(KCNE1):c.142C>T (p.Leu48Phe)KCNE1Uncertain significancecriteria provided, multiple submitters, no conflicts
1193618NM_000219.6(KCNE1):c.199C>A (p.Arg67Ser)KCNE1Uncertain significancecriteria provided, multiple submitters, no conflicts
132670NM_000219.6(KCNE1):c.23C>T (p.Ala8Val)KCNE1Uncertain significancecriteria provided, multiple submitters, no conflicts
132673NM_000219.6(KCNE1):c.247G>A (p.Glu83Lys)KCNE1Uncertain significancecriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 7 · Orphanet: 3 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
KCNE1DefinitiveAutosomal dominantlong QT syndrome 57

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
KCNE1Orphanet:101016Romano-Ward syndrome
KCNE1Orphanet:334Hereditary atrial fibrillation
KCNE1Orphanet:90647Jervell and Lange-Nielsen syndrome

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
KCNE1HGNC:6240ENSG00000180509P15382Potassium voltage-gated channel subfamily E member 1gencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
KCNE1Potassium voltage-gated channel subfamily E member 1Ancillary protein that functions as a regulatory subunit of the voltage-gated potassium (Kv) channel complex composed of pore-forming and potassium-conducting alpha subunits and of regulatory beta subunits.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 0 · Druggable fraction: 1.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Ion channel1111.5×0.009

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
KCNE1Ion channelyesK_chnl_KCNE, KCNE1

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
blood1
leukocyte1
monocyte1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
KCNE1121broadmarkerblood, monocyte, leukocyte

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
KCNE11,005

Structural data

PDB: 1 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
KCNE1P153825

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 4. Enrichment computed across 1 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Phase 3 - rapid repolarisation11142.0×0.003KCNE1
Phase 2 - plateau phase1761.3×0.003KCNE1
Cardiac conduction1108.8×0.012KCNE1
Muscle contraction177.2×0.013KCNE1

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
vestibular nucleus development18426.0×0.001KCNE1
secretory granule organization13370.4×0.001KCNE1
negative regulation of protein targeting to membrane12808.7×0.001KCNE1
negative regulation of delayed rectifier potassium channel activity12808.7×0.001KCNE1
regulation of potassium ion transport11872.4×0.001KCNE1
cardiac muscle cell contraction11685.2×0.001KCNE1
membrane repolarization during action potential11685.2×0.001KCNE1
membrane repolarization during cardiac muscle cell action potential11685.2×0.001KCNE1
membrane repolarization during ventricular cardiac muscle cell action potential11685.2×0.001KCNE1
epithelial cell maturation11532.0×0.001KCNE1
membrane repolarization11296.3×0.002KCNE1
potassium ion export across plasma membrane11053.2×0.002KCNE1
ventricular cardiac muscle cell action potential1991.3×0.002KCNE1
positive regulation of potassium ion transmembrane transport1991.3×0.002KCNE1
regulation of ventricular cardiac muscle cell membrane repolarization1842.6×0.002KCNE1
cardiac muscle cell action potential involved in contraction1702.2×0.002KCNE1
regulation of potassium ion transmembrane transport1624.1×0.002KCNE1
regulation of heart rate by cardiac conduction1374.5×0.003KCNE1
cellular response to cAMP1290.6×0.004KCNE1
regulation of membrane potential1230.8×0.005KCNE1
potassium ion transmembrane transport1135.9×0.008KCNE1
sensory perception of sound1100.9×0.010KCNE1

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 1 · Undrugged: 0

Druggability breadth: 1 of 1 evidence-associated genes (100%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
KCNE1AMBRISENTAN

Top cohort targets by molecule count

SymbolMoleculesMax phase
KCNE1144

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
AMBRISENTAN4KCNE1
DULOXETINE4KCNE1
PALONOSETRON4KCNE1
DARUNAVIR4KCNE1
DARIFENACIN4KCNE1
TOLTERODINE4KCNE1
SOLIFENACIN4KCNE1
EVEROLIMUS4KCNE1
RALTEGRAVIR4KCNE1
MARAVIROC4KCNE1
ALVIMOPAN4KCNE1
NEBIVOLOL4KCNE1
SUNITINIB4KCNE1
NELFINAVIR4KCNE1

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
KCNE1117Functional:63, Binding:47, ADMET:6, Toxicity:1

Cohort genes with high screening signal

≥100 ChEMBL assays — a studied-ness signal; see Therapeutics for approved-drug status.

SymbolChEMBL assays
KCNE1117

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

14 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
AMBRISENTAN4KCNE1
DULOXETINE4KCNE1
PALONOSETRON4KCNE1
DARUNAVIR4KCNE1
DARIFENACIN4KCNE1
TOLTERODINE4KCNE1
SOLIFENACIN4KCNE1
EVEROLIMUS4KCNE1
RALTEGRAVIR4KCNE1
MARAVIROC4KCNE1
ALVIMOPAN4KCNE1
NEBIVOLOL4KCNE1
SUNITINIB4KCNE1
NELFINAVIR4KCNE1

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1KCNE1
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug0

Undrugged target profiles

0 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

Clinical trials & evidence

Clinical trials

Clinical trials: 0.