Lumbar plexus neoplasm

Summary

Lumbar plexus neoplasm (MONDO:0004546) is a cancer. A subtype of lumbosacral plexus lesion — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

• Classification: Cancer

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

Field	Value
Canonical name	lumbar plexus neoplasm
Mondo ID	MONDO:0004546
DOID	DOID:8389
NCIT	C5824
UMLS	C1334437
MedGen	272802
GARD	0024060
Anatomy (UBERON)	UBERON:0034987
Is cancer (heuristic)	yes

Also known as: lumbar nerve plexus neoplasm · lumbar nerve plexus neoplasm (disease) · lumbar nerve plexus tumor · lumbar nerve plexus tumour · lumbar plexus neoplasms · lumbar plexus tumor · lumbar plexus tumour · neoplasm of lumbar nerve plexus · neoplasm of lumbar plexus · neoplasm of the lumbar plexus · tumor of lumbar nerve plexus · tumor of lumbar plexus · tumor of the lumbar plexus · tumour of lumbar nerve plexus · tumour of lumbar plexus · tumour of the lumbar plexus

Disease family

This is a subtype of lumbosacral plexus lesion. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disorder › peripheral nervous system disorder › peripheral neuropathy › nerve plexus disorder › lumbosacral plexus lesion › lumbar plexus neoplasm

Related subtypes (2): lesion of sciatic nerve, sacral nerve plexus disorder

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.