Macular degeneration, early-onset

disease
On this page

Also known as EOMD

Summary

Macular degeneration, early-onset (MONDO:0014501) is a disease with 1 cohort gene.

At a glance

  • Cohort genes: 1
  • ClinVar variants: 90

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namemacular degeneration, early-onset
Mondo IDMONDO:0014501
OMIM616118
UMLSC4015286
MedGen863723
GARD0024995
Is cancer (heuristic)no

Also known as: EOMD · macular degeneration, early-onset

Data availability: 90 ClinVar variants · 3 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disorderretinal disorderretinal degenerationmacular degenerationmacular degeneration, early-onset

Related subtypes (8): vitelliform macular dystrophy, degeneration of macula and posterior pole, macular retinal edema, autosomal recessive bestrophinopathy, occult macular dystrophy, Stargardt disease, patterned macular dystrophy, isolated macular dystrophy

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

90 retrieved; paginated sample, class counts are floors:

38 conflicting classifications of pathogenicity, 32 uncertain significance, 6 benign, 6 benign/likely benign, 4 likely benign, 3 likely pathogenic, 1 pathogenic/likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
1675083NM_001999.4(FBN2):c.3424T>G (p.Cys1142Gly)FBN2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
4531300NM_001999.4(FBN2):c.3724+3A>CFBN2Likely pathogeniccriteria provided, single submitter
930425NM_001999.4(FBN2):c.4055G>C (p.Cys1352Ser)FBN2Likely pathogeniccriteria provided, single submitter
930869NM_001999.4(FBN2):c.3592T>C (p.Cys1198Arg)FBN2Likely pathogeniccriteria provided, single submitter
1042687NM_001999.4(FBN2):c.704C>T (p.Thr235Met)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1302265NM_001999.4(FBN2):c.5983G>A (p.Gly1995Ser)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
137344NM_001999.4(FBN2):c.6511+5G>AFBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
155797NM_001999.4(FBN2):c.7808T>C (p.Phe2603Ser)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
161445NM_001999.4(FBN2):c.3430G>A (p.Glu1144Lys)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1646797NM_001999.4(FBN2):c.7385A>G (p.Asn2462Ser)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213228NM_001999.4(FBN2):c.3394G>A (p.Val1132Ile)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213255NM_001999.4(FBN2):c.8239C>G (p.Leu2747Val)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213318NM_001999.4(FBN2):c.3709C>T (p.Arg1237Cys)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213364NM_001999.4(FBN2):c.7690C>G (p.Gln2564Glu)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213366NM_001999.4(FBN2):c.7942C>A (p.Gln2648Lys)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213377NM_001999.4(FBN2):c.8674G>T (p.Asp2892Tyr)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213392NM_001999.4(FBN2):c.2536G>A (p.Glu846Lys)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
213404NM_001999.4(FBN2):c.5800+5G>AFBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
234773NM_001999.4(FBN2):c.3304A>G (p.Ser1102Gly)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
234845NM_001999.4(FBN2):c.8058C>A (p.Phe2686Leu)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
263401NM_001999.4(FBN2):c.6077C>T (p.Ser2026Phe)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
263857NM_001999.4(FBN2):c.1720A>G (p.Ile574Val)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
263894NM_001999.4(FBN2):c.7418G>A (p.Arg2473Gln)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
264426NM_001999.4(FBN2):c.2557A>C (p.Ile853Leu)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
264520NM_001999.4(FBN2):c.1423G>A (p.Gly475Ser)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
289728NM_001999.4(FBN2):c.6982G>T (p.Ala2328Ser)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
2903097NM_001999.4(FBN2):c.3725-3C>TFBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
350759NM_001999.4(FBN2):c.8376C>G (p.Ile2792Met)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
3591682NM_001999.4(FBN2):c.1232-17A>GFBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
380412NM_001999.4(FBN2):c.2252A>C (p.Glu751Ala)FBN2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 10 · Orphanet: 1 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
FBN2LimitedAutosomal dominantmacular degeneration, early-onset10

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
FBN2Orphanet:115Congenital contractural arachnodactyly

Cohort genes → proteins

1 cohort genes, 1 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence1

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
FBN2HGNC:3604ENSG00000138829P35556Fibrillin-2gencc,clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
FBN2Fibrillin-2Fibrillins are structural components of 10-12 nm extracellular calcium-binding microfibrils, which occur either in association with elastin or in elastin-free bundles.

Protein-family classification

Druggable: 0 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Other/Unknown11.8×0.558

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
FBN2Other/UnknownnoEGF-type_Asp/Asn_hydroxyl_site, EGF, EGF-like_Ca-bd_dom

Expression context

Cohort genes with no expression data: 0.

1 cohort gene are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)1
unknown0

Top tissues across cohort

TissueCohort genes
adrenal tissue1
cartilage tissue1
placenta1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
FBN2194ubiquitousmarkercartilage tissue, placenta, adrenal tissue

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
FBN22,570

Structural data

PDB: 0 · AlphaFold-only: 1 · No structure: 0

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
FBN2P35556

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 3. Enrichment computed across 1 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Elastic fibre formation1335.9×0.005FBN2
Molecules associated with elastic fibres1308.6×0.005FBN2
Degradation of the extracellular matrix1117.7×0.008FBN2

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
obsolete sequestering of TGFbeta in extracellular matrix14213.0×0.002FBN2
bone trabecula formation12106.5×0.002FBN2
embryonic eye morphogenesis11532.0×0.002FBN2
embryonic limb morphogenesis1401.2×0.004FBN2
positive regulation of bone mineralization1391.9×0.004FBN2
camera-type eye development1358.6×0.004FBN2
glucose metabolic process1255.3×0.005FBN2
positive regulation of osteoblast differentiation1224.7×0.005FBN2
glucose homeostasis1130.6×0.008FBN2

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 1

Druggability breadth: 0 of 1 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
FBN200

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 1; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug1FBN2

Undrugged target profiles

1 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
FBN20

Clinical trials & evidence

Clinical trials

Clinical trials: 0.