Maternal 14q32.2 microdeletion syndrome

disease
On this page

Also known as maternal del(14)(q32.2)maternal monosomy 14q32.2

Summary

Maternal 14q32.2 microdeletion syndrome (MONDO:0016781) is a disease. A subtype of multiple congenital anomalies due to 14q32.2 maternally expressed gene defect — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Prevalence: <1 / 1 000 000 (Worldwide) [Orphanet-validated]
  • Phenotypes (HPO): 38

Clinical features

Epidemiology

Prevalence records

2 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Cases/families8WorldwideValidated
Point prevalence<1 / 1 000 000WorldwideValidated

Signs & symptoms

Clinical features (HPO)

38 HPO clinical features (Orphanet curated; top 38 by frequency):

HPO IDTermFrequency
HP:0001263Global developmental delayVery frequent (80-99%)
HP:0001561PolyhydramniosVery frequent (80-99%)
HP:0005257Thoracic hypoplasiaVery frequent (80-99%)
HP:0006665Coat hanger sign of ribsVery frequent (80-99%)
HP:0011968Feeding difficultiesVery frequent (80-99%)
HP:0000023Inguinal herniaFrequent (30-79%)
HP:0001371Flexion contractureFrequent (30-79%)
HP:0001382Joint hypermobilityFrequent (30-79%)
HP:0001537Umbilical herniaFrequent (30-79%)
HP:0001540Diastasis rectiFrequent (30-79%)
HP:0002878Respiratory failureFrequent (30-79%)
HP:0006267Large placentaFrequent (30-79%)
HP:0008897Postnatal growth retardationFrequent (30-79%)
HP:0000126HydronephrosisOccasional (5-29%)
HP:0000158MacroglossiaOccasional (5-29%)
HP:0000194Open mouthOccasional (5-29%)
HP:0000278RetrognathiaOccasional (5-29%)
HP:0000286EpicanthusOccasional (5-29%)
HP:0000337Broad foreheadOccasional (5-29%)
HP:0000341Narrow foreheadOccasional (5-29%)
HP:0000463Anteverted naresOccasional (5-29%)
HP:0000565EsotropiaOccasional (5-29%)
HP:0000767Pectus excavatumOccasional (5-29%)
HP:0000884Prominent sternumOccasional (5-29%)
HP:0000954Single transverse palmar creaseOccasional (5-29%)
HP:0001239Wrist flexion contractureOccasional (5-29%)
HP:0001252HypotoniaOccasional (5-29%)
HP:0001539OmphaloceleOccasional (5-29%)
HP:0001601LaryngomalaciaOccasional (5-29%)
HP:0001845Overlapping toeOccasional (5-29%)
HP:0002263Exaggerated cupid’s bowOccasional (5-29%)
HP:0005280Depressed nasal bridgeOccasional (5-29%)
HP:0005989Redundant neck skinOccasional (5-29%)
HP:0010511Long toeOccasional (5-29%)
HP:0012385CamptodactylyOccasional (5-29%)
HP:0012785Flexion contracture of fingerOccasional (5-29%)
HP:0045025Narrow palpebral fissureOccasional (5-29%)
HP:0001511Intrauterine growth retardationVery rare (<1-4%)

Identifiers

Disease identifiers

FieldValue
Canonical namematernal 14q32.2 microdeletion syndrome
Mondo IDMONDO:0016781
Orphanet254528
UMLSC5679640
MedGen1842712
GARD0017221
Is cancer (heuristic)no

Also known as: maternal del(14)(q32.2) · maternal monosomy 14q32.2

Disease family

This is a subtype of multiple congenital anomalies due to 14q32.2 maternally expressed gene defect. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by developmental or physiological process › disorder of development or morphogenesisdevelopmental defect during embryogenesismultiple congenital anomalies/dysmorphic syndromemultiple congenital anomalies/dysmorphic syndrome-intellectual disabilitymultiple congenital anomalies due to 14q32.2 maternally expressed gene defectmaternal 14q32.2 microdeletion syndrome

Related subtypes (2): paternal uniparental disomy of chromosome 14, maternal 14q32.2 hypermethylation syndrome

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.