Megacystis-microcolon-intestinal hypoperistalsis syndrome 2

disease
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Also known as MMIHS2

Summary

Megacystis-microcolon-intestinal hypoperistalsis syndrome 2 (MONDO:0025708) is a disease caused by MYH11 (GenCC Strong), with 3 cohort genes.

At a glance

  • Causal gene: MYH11 (GenCC Strong)
  • Cohort genes: 3
  • ClinVar variants: 172

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namemegacystis-microcolon-intestinal hypoperistalsis syndrome 2
Mondo IDMONDO:0025708
OMIM619351
UMLSC5543476
MedGen1788773
GARD0016447
Is cancer (heuristic)no

Also known as: MMIHS2

Data availability: 172 ClinVar variants · 2 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › syndromic diseasemegacystis-microcolon-intestinal hypoperistalsis syndromemegacystis-microcolon-intestinal hypoperistalsis syndrome 2

Related subtypes (4): megacystis-microcolon-intestinal hypoperistalsis syndrome 3, megacystis-microcolon-intestinal hypoperistalsis syndrome 4, megacystis-microcolon-intestinal hypoperistalsis syndrome 5, megacystis-microcolon-intestinal hypoperistalsis syndrome 1

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

172 retrieved; paginated sample, class counts are floors:

112 uncertain significance, 30 conflicting classifications of pathogenicity, 10 benign, 6 benign/likely benign, 5 pathogenic, 4 likely benign, 3 likely pathogenic, 2 pathogenic/likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
1341990GRCh37/hg19 16p13.11(chr16:15551302-16194578)x1ABCC1Pathogeniccriteria provided, single submitter
1120184NM_002474.3(MYH11):c.3519_3520delinsTT (p.Glu1173_Gln1174delinsAspTer)MYH11Pathogenicno assertion criteria provided
1252066NM_002474.3(MYH11):c.1033+1G>AMYH11Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1341984NM_002474.3(MYH11):c.3122-2_3124delMYH11Pathogeniccriteria provided, single submitter
156401NM_002474.3(MYH11):c.3598A>T (p.Lys1200Ter)MYH11Pathogenic/Likely pathogenicno assertion criteria provided
211548NM_002474.3(MYH11):c.3422_3470del (p.Lys1141fs)MYH11Pathogeniccriteria provided, single submitter
3776079NM_002474.3(MYH11):c.4435dup (p.Arg1479fs)MYH11Pathogeniccriteria provided, single submitter
2627065NM_002474.3(MYH11):c.3424C>T (p.Arg1142Ter)MYH11Likely pathogeniccriteria provided, single submitter
3366318NC_000016.9:g.(15797981_15802659)_(15892545_15917111)delMYH11Likely pathogeniccriteria provided, single submitter
3382738NM_002474.3(MYH11):c.582C>G (p.Tyr194Ter)MYH11Likely pathogeniccriteria provided, single submitter
1171454NM_002474.3(MYH11):c.2426G>A (p.Arg809Lys)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
1329442NM_002474.3(MYH11):c.2082G>A (p.Leu694=)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
14135NM_002474.3(MYH11):c.2135G>A (p.Arg712Gln)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
161317NM_002474.3(MYH11):c.739C>T (p.Arg247Cys)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
201039NM_002474.3(MYH11):c.5528C>T (p.Ser1843Leu)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
201057NM_002474.3(MYH11):c.2741C>T (p.Ala914Val)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
201068NM_002474.3(MYH11):c.3928G>A (p.Val1310Met)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
201086NM_002474.3(MYH11):c.5499G>C (p.Glu1833Asp)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
201092NM_002474.3(MYH11):c.5767G>A (p.Ala1923Thr)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
201100NM_001040113.2(MYH11):c.654+1G>AMYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
211547NM_002474.3(MYH11):c.2809_2810del (p.Arg937fs)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
252514NM_002474.3(MYH11):c.1913C>T (p.Ser638Leu)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
257258NM_002474.3(MYH11):c.3897C>T (p.Ala1299=)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
263518NM_002474.3(MYH11):c.3766A>C (p.Lys1256Gln)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
263862NM_002474.3(MYH11):c.472G>A (p.Ala158Thr)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
318108NM_002474.3(MYH11):c.4791+13G>AMYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
465745NM_002474.3(MYH11):c.5092G>A (p.Ala1698Thr)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
629821NM_002474.3(MYH11):c.4117-6T>GMYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
807997NM_002474.3(MYH11):c.5869G>C (p.Glu1957Gln)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
888295NM_002474.3(MYH11):c.3669C>A (p.Asp1223Glu)MYH11Conflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 10 · Orphanet: 8 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
MYH11StrongAutosomal recessivemegacystis-microcolon-intestinal hypoperistalsis syndrome 210

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
MYH11Orphanet:2241Megacystis-microcolon-intestinal hypoperistalsis syndrome
MYH11Orphanet:229Familial aortic dissection
MYH11Orphanet:91387Familial thoracic aortic aneurysm and aortic dissection
MYH11Orphanet:98829Acute myeloid leukemia with abnormal bone marrow eosinophils inv(16)(p13q22) or t(16;16)(p13;q22)
NDE1Orphanet:2177Hydranencephaly
NDE1Orphanet:443162NDE1-related microhydranencephaly
NDE1Orphanet:89844Lissencephaly syndrome, Norman-Roberts type
ABCC1Orphanet:90635Rare autosomal dominant non-syndromic sensorineural deafness type DFNA

Cohort genes → proteins

3 cohort genes, 3 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence3

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
MYH11HGNC:7569ENSG00000133392P35749Myosin-11gencc,clinvar
NDE1HGNC:17619ENSG00000072864Q9NXR1Nuclear distribution protein nudE homolog 1clinvar
ABCC1HGNC:51ENSG00000103222P33527Multidrug resistance-associated protein 1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
MYH11Myosin-11Muscle contraction.
NDE1Nuclear distribution protein nudE homolog 1Required for centrosome duplication and formation and function of the mitotic spindle.
ABCC1Multidrug resistance-associated protein 1Mediates export of organic anions and drugs from the cytoplasm.

Protein-family classification

Druggable: 1 · Difficult: 1 · Unknown: 1 · Druggable fraction: 0.33

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Transporter125.9×0.114
Scaffold/PPI15.8×0.246
Other/Unknown10.6×0.914

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
MYH11Scaffold/PPInoIQ_motif_EF-hand-BS, Myosin_head_motor_dom-like, Myosin_tail
NDE1Other/UnknownnoNUDE_dom, NUDE
ABCC1Transporteryes7.6.2.2ABC_transporter-like_ATP-bd, AAA+_ATPase, MRP

Expression context

Cohort genes with no expression data: 0.

3 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)3
unknown0

Top tissues across cohort

TissueCohort genes
lower esophagus2
lower esophagus muscularis layer2
right coronary artery1
colonic epithelium1
corpus callosum1
ventricular zone1
lower esophagus mucosa1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
MYH11143broadmarkerright coronary artery, lower esophagus, lower esophagus muscularis layer
NDE1134ubiquitousmarkercolonic epithelium, ventricular zone, corpus callosum
ABCC1134ubiquitousmarkerlower esophagus mucosa, lower esophagus, lower esophagus muscularis layer

Protein interactions among cohort

Intra-cohort edges: 0.

Hub genes (top 10 by interactor count)

SymbolInteractor count
MYH113,818
ABCC13,018
NDE11,761

Structural data

PDB: 3 · AlphaFold-only: 0 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
ABCC1P335275
MYH11P357491
NDE1Q9NXR11

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 69. Enrichment computed across 3 evidence-associated genes (3 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 3 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Metabolism of porphyrins1475.8×0.024ABCC1
Transport of RCbl within the body1475.8×0.024ABCC1
NFE2L2 regulating MDR associated enzymes1475.8×0.024ABCC1
Heme degradation1271.9×0.024ABCC1
Sema4D in semaphorin signaling1223.9×0.024MYH11
Cobalamin (Cbl, vitamin B12) transport and metabolism1211.5×0.024ABCC1
RHO GTPases activate CIT1200.3×0.024MYH11
RHO GTPases Activate ROCKs1200.3×0.024MYH11
Synthesis of Leukotrienes (LT) and Eoxins (EX)1190.3×0.024ABCC1
Arachidonate metabolism1190.3×0.024ABCC1
Sema4D induced cell migration and growth-cone collapse1190.3×0.024MYH11
RHO GTPases activate PAKs1181.3×0.024MYH11
Developmental Lineage of Mammary Gland Myoepithelial Cells1181.3×0.024MYH11
RHO GTPase Effectors245.3×0.024MYH11, NDE1
Signaling by Rho GTPases222.8×0.024MYH11, NDE1
Signaling by Rho GTPases, Miro GTPases and RHOBTB3222.3×0.024MYH11, NDE1
Paracetamol ADME1141.0×0.029ABCC1
Semaphorin interactions1131.3×0.029MYH11
EPHA-mediated growth cone collapse1126.9×0.029MYH11
Nuclear events mediated by NFE2L21112.0×0.031ABCC1
RHO GTPases activate PKNs1105.7×0.031MYH11
Sphingolipid de novo biosynthesis195.2×0.033ABCC1
Smooth Muscle Contraction188.5×0.034MYH11
Centrosome maturation184.6×0.034NDE1
Drug ADME176.1×0.036ABCC1
Amplification of signal from the kinetochores165.6×0.039NDE1
Cytoprotection by HMOX1161.4×0.039ABCC1
Metabolism of water-soluble vitamins and cofactors160.4×0.039ABCC1
Sphingolipid metabolism156.0×0.039ABCC1
EPH-Ephrin signaling155.2×0.039MYH11

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 3 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
antigen processing and presentation of lipid antigen via MHC class Ib15617.3×0.003ABCC1
cyclic nucleotide transport15617.3×0.003ABCC1
sphingolipid translocation15617.3×0.003ABCC1
intracellular nitrogen homeostasis15617.3×0.003ABCC1
pigment accumulation12808.7×0.005ABCC1
chromosome localization12808.7×0.005NDE1
granuloma formation11872.4×0.005ABCC1
skeletal muscle myosin thick filament assembly11872.4×0.005MYH11
glutathione transmembrane transport11404.3×0.005ABCC1
neural tissue regeneration11404.3×0.005ABCC1
mitotic centrosome separation1936.2×0.005NDE1
glucocorticoid metabolic process1936.2×0.005ABCC1
amygdala development1936.2×0.005ABCC1
NK T cell activation1936.2×0.005ABCC1
carboxylic acid transmembrane transport1936.2×0.005ABCC1
xenobiotic transport across blood-brain barrier1936.2×0.005ABCC1
leukotriene transport1802.5×0.005ABCC1
lymphocyte migration1802.5×0.005ABCC1
cobalamin transport1624.1×0.006ABCC1
response to fluid shear stress1624.1×0.006ABCC1
export across plasma membrane1561.7×0.006ABCC1
heme catabolic process1510.7×0.006ABCC1
elastic fiber assembly1510.7×0.006MYH11
amyloid-beta metabolic process1510.7×0.006ABCC1
hydrogen peroxide biosynthetic process1468.1×0.006ABCC1
endothelium development1432.1×0.006ABCC1
leukotriene metabolic process1432.1×0.006ABCC1
leukotriene biosynthetic process1432.1×0.006ABCC1
endothelial cell apoptotic process1432.1×0.006ABCC1
transepithelial transport1401.2×0.006ABCC1

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 1 · Undrugged: 2

Druggability breadth: 1 of 3 evidence-associated genes (33%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
ABCC1RIMONABANT

Top cohort targets by molecule count

SymbolMoleculesMax phase
ABCC1234
MYH1100
NDE100

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
RIMONABANT4ABCC1
VINBLASTINE4ABCC1
CYCLOSPORINE4ABCC1
DAUNORUBICIN4ABCC1
ETRAVIRINE4ABCC1
BENZBROMARONE4ABCC1
ESTRONE SULFURIC ACID4ABCC1
DOXORUBICIN4ABCC1
MITOXANTRONE4ABCC1
INDOMETHACIN4ABCC1
IVERMECTIN4ABCC1
VERAPAMIL4ABCC1
VINCRISTINE4ABCC1
VALSPODAR3ABCC1
TARIQUIDAR3ABCC1
QUERCETIN3ABCC1
DEXVERAPAMIL2ABCC1
VEDROPREVIR2ABCC1
VERLUKAST2ABCC1
BIRICODAR2ABCC1
CLESACOSTAT2ABCC1
BMS-7548072ABCC1
KAEMPFEROL1ABCC1

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
ABCC1459Binding:270, Functional:166, ADMET:23

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
ABCC17.6.2.2, 7.6.2.3ABC-type xenobiotic transporter, ABC-type glutathione-S-conjugate transporter

Cohort genes with high screening signal

≥100 ChEMBL assays — a studied-ness signal; see Therapeutics for approved-drug status.

SymbolChEMBL assays
ABCC1459

Pharmacogenomics

Cohort genes with a PharmGKB record: 3; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

23 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
RIMONABANT4ABCC1
VINBLASTINE4ABCC1
CYCLOSPORINE4ABCC1
DAUNORUBICIN4ABCC1
ETRAVIRINE4ABCC1
BENZBROMARONE4ABCC1
ESTRONE SULFURIC ACID4ABCC1
DOXORUBICIN4ABCC1
MITOXANTRONE4ABCC1
INDOMETHACIN4ABCC1
IVERMECTIN4ABCC1
VERAPAMIL4ABCC1
VINCRISTINE4ABCC1
VALSPODAR3ABCC1
TARIQUIDAR3ABCC1
QUERCETIN3ABCC1
DEXVERAPAMIL2ABCC1
VEDROPREVIR2ABCC1
VERLUKAST2ABCC1
BIRICODAR2ABCC1
CLESACOSTAT2ABCC1
BMS-7548072ABCC1
KAEMPFEROL1ABCC1

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1ABCC1
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug2MYH11, NDE1

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
MYH110
NDE10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.