Moderate multiminicore disease with hand involvement

disease
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Summary

Moderate multiminicore disease with hand involvement (MONDO:0015793) is a disease. A subtype of multiminicore myopathy — broader associated-gene and molecular evidence is on the parent page (see Disease family below).

At a glance

  • Phenotypes (HPO): 14

Clinical features

Signs & symptoms

Clinical features (HPO)

14 HPO clinical features (Orphanet curated; top 14 by frequency):

HPO IDTermFrequency
HP:0001319Neonatal hypotoniaFrequent (30-79%)
HP:0001382Joint hypermobilityFrequent (30-79%)
HP:0001762Talipes equinovarusFrequent (30-79%)
HP:0002194Delayed gross motor developmentFrequent (30-79%)
HP:0003324Generalized muscle weaknessFrequent (30-79%)
HP:0003327Axial muscle weaknessFrequent (30-79%)
HP:0008954Intrinsic hand muscle atrophyFrequent (30-79%)
HP:0008959Distal upper limb muscle weaknessFrequent (30-79%)
HP:0009046Difficulty runningFrequent (30-79%)
HP:0010628Facial palsyFrequent (30-79%)
HP:0012391Hyporeflexia of upper limbsFrequent (30-79%)
HP:0003803Type 1 muscle fiber predominanceOccasional (5-29%)
HP:0004976Knee dislocationOccasional (5-29%)
HP:0005001Recurrent patellar dislocationOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical namemoderate multiminicore disease with hand involvement
Mondo IDMONDO:0015793
Orphanet178145
UMLSC1861753
MedGen396213
GARD0017078
Is cancer (heuristic)no

Disease family

This is a subtype of multiminicore myopathy. Genetic, therapeutic, and trial evidence is largely curated at the broader-term level — see the parent page for the associated-gene cohort and molecular evidence.

Classification path: disease › human disease › disease by body system or component › nervous system disorderqualitative or quantitative protein defects in neuromuscular diseases › neuromuscular disease caused by qualitative or quantitative defects of selenoprotein N1 › multiminicore myopathymoderate multiminicore disease with hand involvement

Related subtypes (4): congenital multicore myopathy with external ophthalmoplegia, rigid spine muscular dystrophy 1, antenatal multiminicore disease with arthrogryposis multiplex congenita, classic multiminicore myopathy

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

No tiered GWAS variants or ClinVar records for this disease.

Genes & proteins

No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).

Function

No pathway enrichment — requires an associated-gene cohort.

Therapeutics

No druggable-target or therapeutic data for this disease’s cohort.

Clinical trials & evidence

Clinical trials

Clinical trials: 0.

No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.