Murray valley encephalitis
diseaseOn this page
Also known as Australian encephalitisAustralian X diseaseMurray Valley encephalitis virus caused disease or disorderMurray Valley encephalitis virus disease or disorderMurray Valley encephalitis virus infectious disease
Summary
Murray valley encephalitis (MONDO:0001137) is a disease. A subtype of Flaviviridae infectious disease — broader associated-gene and molecular evidence is on the parent page (see Disease family below).
Clinical features
No curated clinical features (Orphanet) for this disease.
Identifiers
Disease identifiers
| Field | Value |
|---|---|
| Canonical name | Murray valley encephalitis |
| Mondo ID | MONDO:0001137 |
| DOID | DOID:10842 |
| ICD-10-CM | A83.4 |
| ICD-11 | 1040970454 |
| SNOMED CT | 66454007 |
| UMLS | C0153066 |
| MedGen | 509134 |
| Is cancer (heuristic) | no |
Also known as: Australian encephalitis · Australian X disease · Murray Valley encephalitis virus caused disease or disorder · Murray Valley encephalitis virus disease or disorder · Murray Valley encephalitis virus infectious disease
Disease family
Classification path: disease › human disease › disease by etiologic mechanism › disease of primarily extrinsic mechanism › infectious disease › viral infectious disease › primary viral infectious disease › Flaviviridae infectious disease › Murray valley encephalitis
Related subtypes (9): Powassan encephalitis, West Nile fever, dengue disease, pestivirus infectious disease, st. Louis encephalitis, Kyasanur forest disease, Omsk hemorrhagic fever, Zika virus infectious disease, yellow fever
Genetics & variants
GWAS landscape
No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.
Variant details and genetic-evidence tiers
No tiered GWAS variants or ClinVar records for this disease.
Genes & proteins
No associated-gene cohort resolved for this disease. Atlas builds the molecular and therapeutic sections — associated genes, protein families, druggability, pathways, interactions, and drug associations — by aggregating over a disease’s associated genes (resolved via GWAS / GenCC / ClinVar / CIViC), and none resolved here. This is expected for antibody-mediated, autoimmune, or otherwise non-gene-defined conditions; the curated evidence for this disease is its clinical features, GWAS susceptibility, and clinical trials (above).
Function
No pathway enrichment — requires an associated-gene cohort.
Therapeutics
No druggable-target or therapeutic data for this disease’s cohort.
Clinical trials & evidence
Clinical trials
Clinical trials: 0.
Related Atlas pages
No linked Atlas pages yet — the cross-entity mesh grows as the corpus expands.