Muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12

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Also known as MDDGA12muscular dystrophy-dystroglycanopathy, type A caused by mutation in POMKPOMK muscular dystrophy-dystroglycanopathy, type A

Summary

Muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12 (MONDO:0014101) is a disease caused by POMK (GenCC Definitive), with 4 cohort genes.

At a glance

  • Causal gene: POMK (GenCC Definitive)
  • Cohort genes: 4
  • ClinVar variants: 247

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12
Mondo IDMONDO:0014101
OMIM615249
DOIDDOID:0111235
UMLSC3808964
MedGen815294
GARD0015927
Is cancer (heuristic)no

Also known as: MDDGA12 · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12 · muscular dystrophy-dystroglycanopathy, type A caused by mutation in POMK · POMK muscular dystrophy-dystroglycanopathy, type A

Data availability: 247 ClinVar variants · 5 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disordercongenital muscular dystrophymuscular dystrophy-dystroglycanopathymuscular dystrophy-dystroglycanopathy, type Amuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12

Related subtypes (13): muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A3, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A2, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A5, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A6, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 10, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 11, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A13, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A14, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A9

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

247 retrieved; paginated sample, class counts are floors:

118 uncertain significance, 80 likely benign, 21 pathogenic, 14 conflicting classifications of pathogenicity, 6 benign/likely benign, 5 pathogenic/likely pathogenic, 3 benign

ClinVarVariant (HGVS)GeneClassificationReview
4073416NM_000723.5(CACNB1):c.124_133del (p.Asp42fs)CACNB1Pathogeniccriteria provided, single submitter
1071646NM_032237.5(POMK):c.152del (p.Asp51fs)POMKPathogeniccriteria provided, single submitter
1076046NM_032237.5(POMK):c.43C>T (p.Arg15Ter)POMKPathogeniccriteria provided, multiple submitters, no conflicts
1323591NM_032237.5(POMK):c.907C>T (p.Arg303Ter)POMKPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
133346NM_032237.5(POMK):c.325C>T (p.Gln109Ter)POMKPathogeniccriteria provided, single submitter
1339764NM_032237.5(POMK):c.386_387del (p.Leu129fs)POMKPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1451698NM_032237.5(POMK):c.452_455dup (p.His152fs)POMKPathogeniccriteria provided, single submitter
160348NM_032237.5(POMK):c.288del (p.Leu97fs)POMKPathogenicno assertion criteria provided
1911792NM_032237.5(POMK):c.754dup (p.Asp252fs)POMKPathogeniccriteria provided, single submitter
2002655NM_032237.5(POMK):c.280A>T (p.Arg94Ter)POMKPathogeniccriteria provided, single submitter
2158045NM_032237.5(POMK):c.94_98del (p.Thr32fs)POMKPathogeniccriteria provided, single submitter
2165161NM_032237.5(POMK):c.247C>T (p.Gln83Ter)POMKPathogeniccriteria provided, single submitter
2634888NM_032237.5(POMK):c.398_399del (p.His133fs)POMKPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
2926947NM_032237.5(POMK):c.459del (p.Pro153_Leu154insTer)POMKPathogeniccriteria provided, single submitter
2932937NM_032237.5(POMK):c.724dup (p.Val242fs)POMKPathogeniccriteria provided, single submitter
2945834NM_032237.5(POMK):c.847del (p.Trp283fs)POMKPathogeniccriteria provided, single submitter
2951325NM_032237.5(POMK):c.645T>G (p.Tyr215Ter)POMKPathogeniccriteria provided, single submitter
3752434NM_032237.5(POMK):c.61del (p.Val21fs)POMKPathogeniccriteria provided, single submitter
3760619NM_032237.5(POMK):c.91_98dup (p.Leu34fs)POMKPathogeniccriteria provided, single submitter
4791932NM_032237.5(POMK):c.43del (p.Arg15fs)POMKPathogeniccriteria provided, single submitter
50610NM_032237.5(POMK):c.410T>G (p.Leu137Arg)POMKPathogenicno assertion criteria provided
582702NM_032237.5(POMK):c.238_239del (p.Glu80fs)POMKPathogeniccriteria provided, single submitter
651249NC_000008.11:g.(?43103539)(43103840_?)delPOMKPathogeniccriteria provided, single submitter
817678NM_032237.5(POMK):c.43dup (p.Arg15fs)POMKPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
849427NM_032237.5(POMK):c.10C>T (p.Gln4Ter)POMKPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
949777NM_032237.5(POMK):c.282+1G>CPOMKPathogeniccriteria provided, single submitter
1312144NM_032237.5(POMK):c.256C>T (p.Arg86Cys)POMKConflicting classifications of pathogenicitycriteria provided, conflicting classifications
160349NM_032237.5(POMK):c.905T>A (p.Val302Asp)POMKConflicting classifications of pathogenicitycriteria provided, conflicting classifications
2683017NM_032237.5(POMK):c.803del (p.Phe268fs)POMKConflicting classifications of pathogenicitycriteria provided, conflicting classifications
432418NM_032237.5(POMK):c.20A>G (p.Asn7Ser)POMKConflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 9 · Orphanet: 5 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
POMKDefinitiveAutosomal recessivemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 129

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
POMKOrphanet:370959Congenital muscular dystrophy with cerebellar involvement
POMKOrphanet:445110Limb-girdle muscular dystrophy due to POMK deficiency
POMKOrphanet:899Walker-Warburg syndrome
HGSNATOrphanet:791Retinitis pigmentosa
HGSNATOrphanet:79271Sanfilippo syndrome type C

Cohort genes → proteins

4 cohort genes, 4 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence4

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
POMKHGNC:26267ENSG00000185900Q9H5K3Protein O-mannose kinasegencc,clinvar
CACNB1HGNC:1401ENSG00000067191Q02641Voltage-dependent L-type calcium channel subunit beta-1clinvar
HGSNATHGNC:26527ENSG00000165102Q68CP4Heparan-alpha-glucosaminide N-acetyltransferaseclinvar
FNTAHGNC:3782ENSG00000168522P49354Protein farnesyltransferase/geranylgeranyltransferase type-1 subunit alphaclinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
POMKProtein O-mannose kinaseProtein O-mannose kinase that specifically mediates phosphorylation at the 6-position of an O-mannose of the trisaccharide (N-acetylgalactosamine (GalNAc)-beta-1,3-N-acetylglucosamine (GlcNAc)-beta-1,4-mannose) to generate phosphorylated O…
CACNB1Voltage-dependent L-type calcium channel subunit beta-1Regulatory subunit of L-type calcium channels.
HGSNATHeparan-alpha-glucosaminide N-acetyltransferaseLysosomal acetyltransferase that acetylates the non-reducing terminal alpha-glucosamine residue of intralysosomal heparin or heparan sulfate, converting it into a substrate for luminal alpha-N-acetyl glucosaminidase.
FNTAProtein farnesyltransferase/geranylgeranyltransferase type-1 subunit alphaEssential subunit of both the farnesyltransferase and the geranylgeranyltransferase complex.

Protein-family classification

Druggable: 3 · Difficult: 1 · Unknown: 0 · Druggable fraction: 0.75

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)26.0×0.112
Kinase16.9×0.205
Scaffold/PPI14.3×0.212

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
POMKKinaseyes2.7.1.183Prot_kinase_dom, Ser-Thr/Tyr_kinase_cat_dom, Kinase-like_dom_sf
CACNB1Scaffold/PPInoVDCC_L_bsu, SH3_domain, VDCC_L_b1su
HGSNATEnzyme (other)yes2.3.1.78HGSNAT_cat
FNTAEnzyme (other)yes2.5.1.58Prenyl_trans_a

Expression context

Cohort genes with no expression data: 0.

3 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)4
unknown0

Top tissues across cohort

TissueCohort genes
Brodmann (1909) area 101
middle temporal gyrus1
paraflocculus1
gastrocnemius1
hindlimb stylopod muscle1
triceps brachii1
cervix squamous epithelium1
mucosa of stomach1
right uterine tube1
esophagus squamous epithelium1
gingival epithelium1
olfactory bulb1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
POMK289ubiquitousyesparaflocculus, middle temporal gyrus, Brodmann (1909) area 10
CACNB1224ubiquitousmarkergastrocnemius, hindlimb stylopod muscle, triceps brachii
HGSNAT287ubiquitousmarkermucosa of stomach, right uterine tube, cervix squamous epithelium
FNTA298ubiquitousmarkeresophagus squamous epithelium, gingival epithelium, olfactory bulb

Protein interactions among cohort

Intra-cohort edges: 3.

Hub genes (top 10 by interactor count)

SymbolInteractor count
POMK1,251
CACNB11,128
FNTA1,080
HGSNAT863

Intra-cohort edges

ABSources
FNTAHGSNATstring_interaction
FNTAPOMKstring_interaction
HGSNATPOMKstring_interaction

Structural data

PDB: 3 · AlphaFold-only: 1 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
FNTAP4935414
HGSNATQ68CP412
CACNB1Q026416

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
POMKQ9H5K391.62

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 27. Enrichment computed across 4 evidence-associated genes (4 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 4 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
MPS IIIC - Sanfilippo syndrome C12855.0×0.009HGSNAT
DAG1 core M3 glycosylations1475.8×0.025POMK
GBP-mediated host defense1259.6×0.025FNTA
Presynaptic depolarization and calcium channel opening1237.9×0.025CACNB1
Phase 2 - plateau phase1190.3×0.025CACNB1
Mechanical load activates signaling by PIEZO1 and integrins in osteocytes1167.9×0.025CACNB1
HS-GAG degradation1124.1×0.025HGSNAT
Apoptotic cleavage of cellular proteins1119.0×0.025FNTA
RAS processing1119.0×0.025FNTA
Phase 0 - rapid depolarisation186.5×0.031CACNB1
Inactivation, recovery and regulation of the phototransduction cascade179.3×0.031FNTA
NCAM signaling for neurite out-growth168.0×0.031CACNB1
Cellular responses to mechanical stimuli164.9×0.031CACNB1
NCAM1 interactions162.1×0.031CACNB1
O-linked glycosylation136.1×0.049POMK
Potential therapeutics for SARS128.6×0.058FNTA
Cardiac conduction127.2×0.058CACNB1
Muscle contraction119.3×0.073CACNB1
Transmission across Chemical Synapses119.0×0.073CACNB1
Axon guidance111.3×0.110CACNB1
Neuronal System111.1×0.110CACNB1
Nervous system development110.7×0.110CACNB1
Cellular responses to stimuli17.9×0.142CACNB1
Neutrophil degranulation15.8×0.183HGSNAT
Post-translational protein modification14.8×0.208POMK
Developmental Biology13.6×0.259CACNB1
Metabolism of proteins13.1×0.286POMK

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 4 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
peptide pheromone maturation14213.0×0.006FNTA
protein farnesylation11404.3×0.006FNTA
protein geranylgeranylation1702.2×0.006FNTA
regulation of microtubule-based movement1702.2×0.006FNTA
positive regulation of muscle contraction1601.9×0.006CACNB1
carbohydrate phosphorylation1526.6×0.006POMK
heparan sulfate proteoglycan catabolic process1468.1×0.006HGSNAT
skeletal muscle acetylcholine-gated channel clustering1468.1×0.006FNTA
positive regulation of skeletal muscle acetylcholine-gated channel clustering1468.1×0.006FNTA
regulation of calcium ion transmembrane transport via high voltage-gated calcium channel1421.3×0.006CACNB1
lysosomal transport1175.5×0.011HGSNAT
protein complex oligomerization1168.5×0.011HGSNAT
positive regulation of Rac protein signal transduction1162.0×0.011FNTA
Rac protein signal transduction1140.4×0.012FNTA
neuromuscular process1131.7×0.012POMK
cellular response to amyloid-beta198.0×0.015CACNB1
sensory perception of pain193.6×0.015POMK
learning or memory160.2×0.022POMK
protein O-linked glycosylation156.2×0.022POMK
calcium ion transmembrane transport152.7×0.023CACNB1
calcium ion transport145.3×0.025CACNB1
transforming growth factor beta receptor signaling pathway139.8×0.027FNTA
brain development119.9×0.051POMK
chemical synaptic transmission119.3×0.051CACNB1

Therapeutics

Drug target analysis

Approved (phase 4): 2 · Phase ≥3: 2 · Phased (≥1): 2 · Undrugged: 2

Druggability breadth: 2 of 4 evidence-associated genes (50%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
CACNB1NIMODIPINE
FNTACORTISONE ACETATE

Top cohort targets by molecule count

SymbolMoleculesMax phase
FNTA64
CACNB134
POMK00
HGSNAT00

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
NIMODIPINE4CACNB1
TACRINE4CACNB1
CORTISONE ACETATE4FNTA
LONAFARNIB4FNTA
TIPIFARNIB3FNTA
Z1602CACNB1
L-778123 FREE BASE1FNTA
GGTI-24181FNTA
BMS-2146621FNTA

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 3.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
FNTA513Binding:438, Functional:75
CACNB119Binding:19

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
POMK2.7.1.183glycoprotein-mannosyl O6-kinase
HGSNAT2.3.1.78heparan-alpha-glucosaminide N-acetyltransferase
FNTA2.5.1.58, 2.5.1.59protein farnesyltransferase, protein geranylgeranyltransferase type I

Cohort genes with high screening signal

≥100 ChEMBL assays — a studied-ness signal; see Therapeutics for approved-drug status.

SymbolChEMBL assays
FNTA513

Pharmacogenomics

Cohort genes with a PharmGKB record: 4; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

9 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
NIMODIPINE4CACNB1
TACRINE4CACNB1
CORTISONE ACETATE4FNTA
LONAFARNIB4FNTA
TIPIFARNIB3FNTA
Z1602CACNB1
L-778123 FREE BASE1FNTA
GGTI-24181FNTA
BMS-2146621FNTA

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)2CACNB1, FNTA
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1HGSNAT
DDruggable family + AlphaFold only, no drug1POMK
EDifficult family or no structure, no drug0

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
POMK0
HGSNAT0

Clinical trials & evidence

Clinical trials

Clinical trials: 0.