muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4

disease
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Also known as FCMDFukuyama congenital muscular dystrophyFukuyama Type Congenital Muscular DystrophyMDDGA4muscle-eye-brain-FKTN relatedmuscular dystrophy-dystroglycanopathy (congenital with Brain and eye anomalies) type A, 4Walker-Warburg syndrome or muscle-eye-brain disease, FKTN-related

Summary

muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4 (MONDO:0009678) is a disease caused by FKTN (GenCC Definitive), with 3 cohort genes.

At a glance

  • Prevalence: Unknown (Worldwide) [Orphanet-validated]
  • Causal gene: FKTN (GenCC Definitive)
  • Cohort genes: 3
  • ClinVar variants: 236
  • Phenotypes (HPO): 31

Clinical features

Epidemiology

Prevalence records

1 prevalence record(s), Orphanet:

TypeClassValueGeographyValidation
Prevalence at birth1-9 / 100 0002.8JapanValidated

Signs & symptoms

Clinical features (HPO)

31 HPO clinical features (Orphanet curated; top 31 by frequency):

HPO IDTermFrequency
HP:0000298Mask-like faciesVery frequent (80-99%)
HP:0000750Delayed speech and language developmentVery frequent (80-99%)
HP:0001252HypotoniaVery frequent (80-99%)
HP:0001263Global developmental delayVery frequent (80-99%)
HP:0001288Gait disturbanceVery frequent (80-99%)
HP:0001357PlagiocephalyVery frequent (80-99%)
HP:0001371Flexion contractureVery frequent (80-99%)
HP:0003198MyopathyVery frequent (80-99%)
HP:0003457EMG abnormalityVery frequent (80-99%)
HP:0003560Muscular dystrophyVery frequent (80-99%)
HP:0007260Type II lissencephalyVery frequent (80-99%)
HP:0010864Intellectual disability, severeVery frequent (80-99%)
HP:0030046Hypoglycosylation of alpha-dystroglycanVery frequent (80-99%)
HP:0000238HydrocephalusFrequent (30-79%)
HP:0000248BrachycephalyFrequent (30-79%)
HP:0000545MyopiaFrequent (30-79%)
HP:0000767Pectus excavatumFrequent (30-79%)
HP:0001250SeizureFrequent (30-79%)
HP:0001612Weak cryFrequent (30-79%)
HP:0002119VentriculomegalyFrequent (30-79%)
HP:0002353EEG abnormalityFrequent (30-79%)
HP:0100490Camptodactyly of fingerFrequent (30-79%)
HP:0000268DolichocephalyOccasional (5-29%)
HP:0000501GlaucomaOccasional (5-29%)
HP:0000505Visual impairmentOccasional (5-29%)
HP:0000518CataractOccasional (5-29%)
HP:0000648Optic atrophyOccasional (5-29%)
HP:0001511Intrauterine growth retardationOccasional (5-29%)
HP:0001644Dilated cardiomyopathyOccasional (5-29%)
HP:0007370Aplasia/Hypoplasia of the corpus callosumOccasional (5-29%)
HP:0007973Retinal dysplasiaOccasional (5-29%)

Identifiers

Disease identifiers

FieldValue
Canonical namemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4
Mondo IDMONDO:0009678
OMIM253800
Orphanet272
DOIDDOID:0050559
NCITC126741
SNOMED CT111502003
UMLSC0410174
MedGen140820
GARD0006475
NORD1169
Is cancer (heuristic)no

Also known as: FCMD · Fukuyama congenital muscular dystrophy · Fukuyama Type Congenital Muscular Dystrophy · MDDGA4 · muscle-eye-brain-FKTN related · muscular dystrophy-dystroglycanopathy (congenital with Brain and eye anomalies) type A, 4 · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4 · Walker-Warburg syndrome or muscle-eye-brain disease, FKTN-related

Data availability: 236 ClinVar variants · 4 GenCC gene-disease records · 3 cell lines.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disordercongenital muscular dystrophymuscular dystrophy-dystroglycanopathymuscular dystrophy-dystroglycanopathy, type Amuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4

Related subtypes (13): muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A3, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A2, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A5, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A6, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 10, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 11, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A13, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A14, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A9

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

236 retrieved; paginated sample, class counts are floors:

100 uncertain significance, 56 conflicting classifications of pathogenicity, 32 likely pathogenic, 17 pathogenic/likely pathogenic, 15 pathogenic, 13 benign/likely benign, 3 likely benign

ClinVarVariant (HGVS)GeneClassificationReview
1075765NM_001079802.2(FKTN):c.567_568delinsTT (p.Leu189_Arg190delinsPheTer)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1252030NM_001079802.2(FKTN):c.78C>G (p.Tyr26Ter)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
167069NM_001079802.2(FKTN):c.411C>A (p.Cys137Ter)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1970586NM_001079802.2(FKTN):c.164G>A (p.Trp55Ter)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
225359NM_001079802.2(FKTN):c.607C>T (p.Arg203Ter)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
281839NM_001079802.2(FKTN):c.330dup (p.Thr111fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
283622NM_001079802.2(FKTN):c.456_457del (p.Ser154fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
3199NM_006731.2(FKTN):c.*4392_*4393ins[AB185332.1]FKTNPathogenicno assertion criteria provided
3200NM_001079802.2(FKTN):c.139C>T (p.Arg47Ter)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
3201NM_001079802.2(FKTN):c.187_188del (p.Met63fs)FKTNPathogeniccriteria provided, single submitter
3202NM_006731.2:c.911-24_911-23insN[1200]FKTNPathogenicno assertion criteria provided
3203NM_001079802.2(FKTN):c.1167dup (p.Phe390fs)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
3205NM_001079802.2(FKTN):c.454dup (p.Ser152fs)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
3206NM_001079802.2(FKTN):c.346C>T (p.Gln116Ter)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
3208NM_001079802.2(FKTN):c.920G>A (p.Arg307Gln)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
3212NM_001079802.2(FKTN):c.*5374_*5846delFKTNPathogenicno assertion criteria provided
3216NM_001079802.2(FKTN):c.919C>T (p.Arg307Ter)FKTNPathogeniccriteria provided, multiple submitters, no conflicts
3596214NM_001079802.2(FKTN):c.1045-6C>GFKTNPathogeniccriteria provided, multiple submitters, no conflicts
370133NM_001079802.2(FKTN):c.429del (p.Asp144fs)FKTNPathogeniccriteria provided, single submitter
370768NM_001079802.2(FKTN):c.109G>T (p.Gly37Ter)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
371091NM_001079802.2(FKTN):c.1106del (p.Phe369fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
496331NM_001079802.2(FKTN):c.648-1243G>TFKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
555661NM_001079802.2(FKTN):c.42del (p.Thr14_Leu15insTer)FKTNPathogeniccriteria provided, single submitter
556831NM_001079802.2(FKTN):c.1099del (p.Val367fs)FKTNPathogeniccriteria provided, single submitter
557908NM_001079802.2(FKTN):c.1129_1130del (p.Met377fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
558161NM_001079802.2(FKTN):c.1172+1G>AFKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
558217NM_001079802.2(FKTN):c.1264_1286del (p.Asn422fs)FKTNPathogeniccriteria provided, single submitter
596647NM_001079802.2(FKTN):c.369+1G>CFKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
654926NM_001079802.2(FKTN):c.1261_1286delinsACC (p.Ala421fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
666969NM_001079802.2(FKTN):c.528dup (p.His177fs)FKTNPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 13 · Orphanet: 12 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
FKTNDefinitiveAutosomal recessivemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 413

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
FKTNOrphanet:154Familial isolated dilated cardiomyopathy
FKTNOrphanet:206554Fukutin-related limb-girdle muscular dystrophy R13
FKTNOrphanet:272Congenital muscular dystrophy, Fukuyama type
FKTNOrphanet:370980Congenital muscular dystrophy without intellectual disability
FKTNOrphanet:588Muscle-eye-brain disease
FKTNOrphanet:899Walker-Warburg syndrome
FKRPOrphanet:34515FKRP-related limb-girdle muscular dystrophy R9
FKRPOrphanet:370959Congenital muscular dystrophy with cerebellar involvement
FKRPOrphanet:370968Congenital muscular dystrophy with intellectual disability
FKRPOrphanet:370980Congenital muscular dystrophy without intellectual disability
FKRPOrphanet:588Muscle-eye-brain disease
FKRPOrphanet:899Walker-Warburg syndrome

Cohort genes → proteins

3 cohort genes, 3 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence3

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
FKTNHGNC:3622ENSG00000106692O75072Ribitol-5-phosphate transferase FKTNgencc,clinvar
FKRPHGNC:17997ENSG00000181027Q9H9S5Ribitol 5-phosphate transferase FKRPclinvar
MYH1HGNC:7567ENSG00000109061P12882Myosin-1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
FKTNRibitol-5-phosphate transferase FKTNCatalyzes the transfer of a ribitol-phosphate from CDP-ribitol to the distal N-acetylgalactosamine of the phosphorylated O-mannosyl trisaccharide (N-acetylgalactosamine-beta-3-N-acetylglucosamine-beta-4-(phosphate-6-)mannose), a carbohydra…
FKRPRibitol 5-phosphate transferase FKRPCatalyzes the transfer of a ribitol 5-phosphate from CDP-L-ribitol to the ribitol 5-phosphate previously attached by FKTN/fukutin to the phosphorylated O-mannosyl trisaccharide (N-acetylgalactosamine-beta-3-N-acetylglucosamine-beta-4-(phos…
MYH1Myosin-1Required for normal hearing.

Protein-family classification

Druggable: 0 · Difficult: 1 · Unknown: 2 · Druggable fraction: 0.0

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Scaffold/PPI15.8×0.327
Other/Unknown21.2×0.587

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
FKTNOther/UnknownnoLicD/FKTN/FKRP_NTP_transf, FKTN/MNN-like, FKTN_N
FKRPOther/UnknownnoLicD/FKTN/FKRP_NTP_transf, LicD_transferase, FKRP_N
MYH1Scaffold/PPInoIQ_motif_EF-hand-BS, Myosin_head_motor_dom-like, Myosin_tail

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)3
unknown0

Top tissues across cohort

TissueCohort genes
adrenal tissue1
calcaneal tendon1
germinal epithelium of ovary1
cardiac muscle of right atrium1
hindlimb stylopod muscle1
left ventricle myocardium1
biceps brachii1
skeletal muscle tissue of rectus abdominis1
vastus lateralis1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
FKTN277ubiquitousyescalcaneal tendon, adrenal tissue, germinal epithelium of ovary
FKRP230ubiquitousmarkerleft ventricle myocardium, cardiac muscle of right atrium, hindlimb stylopod muscle
MYH1151tissue_specificmarkerskeletal muscle tissue of rectus abdominis, biceps brachii, vastus lateralis

Protein interactions among cohort

Intra-cohort edges: 1.

Hub genes (top 10 by interactor count)

SymbolInteractor count
MYH12,004
FKRP1,436
FKTN1,226

Intra-cohort edges

ABSources
FKRPFKTNintact, string_interaction

Structural data

PDB: 1 · AlphaFold-only: 2 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
FKRPQ9H9S58

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
FKTNO7507292.48
MYH1P1288273.85

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 3 evidence-associated genes (2 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 2 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Matriglycan biosynthesis on DAG12815.7×1e-06FKTN, FKRP

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 3 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
skeletal muscle fiber differentiation21123.5×4e-05FKTN, FKRP
protein O-linked glycosylation via mannose2624.1×7e-05FKTN, FKRP
basement membrane organization2340.4×2e-04FKTN, FKRP
muscle contraction2138.7×8e-04FKRP, MYH1
pentitol metabolic process15617.3×0.001FKRP
filtration diaphragm assembly15617.3×0.001FKRP
pentose metabolic process12808.7×0.002FKRP
creatine metabolic process11404.3×0.003FKRP
connective tissue development11404.3×0.003FKRP
oxygen metabolic process11404.3×0.003FKRP
maintenance of protein localization in endoplasmic reticulum11123.5×0.004FKRP
localization of cell1936.2×0.004FKRP
connective tissue replacement1802.5×0.004FKRP
cerebellar cortex development1702.2×0.005FKTN
diaphragm development1624.1×0.005FKRP
protein import1561.7×0.005FKRP
response to alcohol1510.7×0.005FKRP
reelin-mediated signaling pathway1401.2×0.006FKRP
respiratory system process1312.1×0.007FKRP
glial cell differentiation1295.6×0.007FKRP
skeletal muscle tissue regeneration1295.6×0.007FKRP
protein tetramerization1208.1×0.010FKRP
negative regulation of JNK cascade1187.2×0.010FKTN
neuromuscular process1175.5×0.011FKRP
adult walking behavior1165.2×0.011FKRP
glycolytic process1127.7×0.013FKRP
heart morphogenesis1124.8×0.013FKRP
camera-type eye development1119.5×0.013FKRP
response to activity1108.0×0.014FKRP
response to glucocorticoid1108.0×0.014FKRP

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 3

Druggability breadth: 0 of 3 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
FKTN00
FKRP00
MYH100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 0.

Pharmacogenomics

Cohort genes with a PharmGKB record: 3; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug0
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug3FKTN, FKRP, MYH1

Undrugged target profiles

3 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
FKTN0
FKRP0
MYH10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.