muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7

disease
On this page

Also known as ISPD muscular dystrophy-dystroglycanopathy, type AMDDGA7muscular dystrophy-dystroglycanopathy, type A caused by mutation in ISPDWalker-Warburg syndrome or muscle-eye-brain disease, ISPD-related

Summary

muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7 (MONDO:0013835) is a disease caused by CRPPA (GenCC Definitive), with 3 cohort genes.

At a glance

  • Causal gene: CRPPA (GenCC Definitive)
  • Cohort genes: 3
  • ClinVar variants: 456

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7
Mondo IDMONDO:0013835
OMIM614643
DOIDDOID:0111234
UMLSC3553330
MedGen766244
GARD0015829
Is cancer (heuristic)no

Also known as: ISPD muscular dystrophy-dystroglycanopathy, type A · MDDGA7 · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7 · muscular dystrophy-dystroglycanopathy, type A caused by mutation in ISPD · Walker-Warburg syndrome or muscle-eye-brain disease, ISPD-related

Data availability: 456 ClinVar variants · 3 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disordercongenital muscular dystrophymuscular dystrophy-dystroglycanopathymuscular dystrophy-dystroglycanopathy, type Amuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7

Related subtypes (13): muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A3, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A2, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A5, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A6, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 10, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 11, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A13, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A14, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A9

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

456 retrieved; paginated sample, class counts are floors:

173 uncertain significance, 136 likely benign, 58 pathogenic, 51 conflicting classifications of pathogenicity, 12 likely pathogenic, 11 benign, 9 benign/likely benign, 6 pathogenic/likely pathogenic

ClinVarVariant (HGVS)GeneClassificationReview
1072533NC_000007.13:g.(?16445666)(16445982_?)delCRPPAPathogeniccriteria provided, single submitter
1072534NC_000007.13:g.(?16131310)(16255832_?)delCRPPAPathogeniccriteria provided, single submitter
1076250NM_001101426.4(CRPPA):c.550C>T (p.Arg184Ter)CRPPAPathogeniccriteria provided, multiple submitters, no conflicts
1252045NM_001101426.4(CRPPA):c.790-1G>CCRPPAPathogeniccriteria provided, single submitter
156455NM_001101426.4(CRPPA):c.1105GTT[3] (p.Val372del)CRPPAPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1680626NC_000007.13:g.(?16341026)(16341111_?)delCRPPAPathogeniccriteria provided, single submitter
1680627NC_000007.13:g.(?16341026)(16415886_?)delCRPPAPathogeniccriteria provided, single submitter
1680628NC_000007.13:g.(?16348128)(16445982_?)delCRPPAPathogeniccriteria provided, single submitter
1680630NC_000007.13:g.(?16460671)(16460947_?)delCRPPAPathogeniccriteria provided, single submitter
1805380NM_001101426.4(CRPPA):c.258-2A>GCRPPAPathogeniccriteria provided, single submitter
1983342NM_001101426.4(CRPPA):c.79_80del (p.Thr27fs)CRPPAPathogeniccriteria provided, single submitter
2070304NM_001101426.4(CRPPA):c.17_24dup (p.Ala9fs)CRPPAPathogeniccriteria provided, single submitter
2078955NM_001101426.4(CRPPA):c.534+1delCRPPAPathogeniccriteria provided, single submitter
2122850NM_001101426.4(CRPPA):c.1092C>A (p.Cys364Ter)CRPPAPathogeniccriteria provided, single submitter
2142758NM_001101426.4(CRPPA):c.1137dup (p.Lys380Ter)CRPPAPathogeniccriteria provided, single submitter
2423709NC_000007.13:g.(?16415697)(16460947_?)delCRPPAPathogeniccriteria provided, single submitter
2423710NC_000007.13:g.(?16297995)(16317871_?)delCRPPAPathogeniccriteria provided, single submitter
2423714NC_000007.13:g.(?16131320)(16460947_?)delCRPPAPathogeniccriteria provided, single submitter
2423715NC_000007.13:g.(?16131320)(16255842_?)delCRPPAPathogeniccriteria provided, single submitter
279985NM_001101426.4(CRPPA):c.53dup (p.Ser19Glufs)CRPPAPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
282846NM_001101426.4(CRPPA):c.643C>T (p.Gln215Ter)CRPPAPathogeniccriteria provided, multiple submitters, no conflicts
287941NM_001101426.4(CRPPA):c.258-1G>CCRPPAPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
2924564NM_001101426.4(CRPPA):c.896del (p.Gly299fs)CRPPAPathogeniccriteria provided, single submitter
2930168NM_001101426.4(CRPPA):c.337C>T (p.Gln113Ter)CRPPAPathogeniccriteria provided, single submitter
2941204NM_001101426.4(CRPPA):c.6_13dup (p.Pro5fs)CRPPAPathogeniccriteria provided, single submitter
31561NM_001101426.4(CRPPA):c.1120-1G>TCRPPAPathogeniccriteria provided, multiple submitters, no conflicts
31562NM_001101426.3(CRPPA):c.(535_684)+6399_(535_684)+14526delCRPPAPathogenicno assertion criteria provided
31563NM_001101426.4(CRPPA):c.789+2T>GCRPPAPathogenicno assertion criteria provided
31565NM_001101426.4(CRPPA):c.647C>A (p.Ala216Asp)CRPPAPathogenicno assertion criteria provided
31566NM_001101426.4(CRPPA):c.832A>T (p.Lys278Ter)CRPPAPathogenicno assertion criteria provided

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 7 · Orphanet: 4 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
CRPPADefinitiveAutosomal recessivemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 77

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
CRPPAOrphanet:352479ISPD-related limb-girdle muscular dystrophy R20
CRPPAOrphanet:370980Congenital muscular dystrophy without intellectual disability
CRPPAOrphanet:588Muscle-eye-brain disease
CRPPAOrphanet:899Walker-Warburg syndrome

Cohort genes → proteins

3 cohort genes, 2 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence3

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
CRPPAHGNC:37276ENSG00000214960A4D126D-ribitol-5-phosphate cytidylyltransferasegencc,clinvar
LRRC72HGNC:42972ENSG00000205858A6NJI9Leucine-rich repeat-containing protein 72clinvar
CRPPA-AS1HGNC:48962ENSG00000229688CRPPA antisense RNA 1clinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
CRPPAD-ribitol-5-phosphate cytidylyltransferaseCytidylyltransferase required for protein O-linked mannosylation.

Protein-family classification

Druggable: 1 · Difficult: 0 · Unknown: 2 · Druggable fraction: 0.33

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Enzyme (other)14.0×0.460
Other/Unknown21.2×0.587

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
CRPPAEnzyme (other)yes2.7.7.40ISPD_synthase_CS, Nucleotide-diphossugar_trans, IspD/TarI
LRRC72Other/UnknownnoLeu-rich_rpt, LRR_dom_sf, LRC72
CRPPA-AS1Other/Unknownno

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)3
unknown0

Top tissues across cohort

TissueCohort genes
male germ line stem cell (sensu Vertebrata) in testis3
left testis2
calcaneal tendon1
corpus callosum1
right testis1
sperm1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
CRPPA134ubiquitousyescorpus callosum, male germ line stem cell (sensu Vertebrata) in testis, calcaneal tendon
LRRC7221tissue_specificmarkermale germ line stem cell (sensu Vertebrata) in testis, left testis, right testis
CRPPA-AS1147tissue_specificmarkersperm, male germ line stem cell (sensu Vertebrata) in testis, left testis

Protein interactions among cohort

Intra-cohort edges: 1.

Hub genes (top 10 by interactor count)

SymbolInteractor count
CRPPA1,629
LRRC72370
CRPPA-AS10

Intra-cohort edges

ABSources
CRPPALRRC72string_interaction

Structural data

PDB: 1 · AlphaFold-only: 1 · No structure: 1

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
CRPPAA4D1261

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
LRRC72A6NJI979.40

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 1. Enrichment computed across 3 evidence-associated genes (1 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
Matriglycan biosynthesis on DAG11815.7×0.001CRPPA

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 1 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
isoprenoid biosynthetic process11685.2×0.002CRPPA
protein O-linked glycosylation via mannose1936.2×0.002CRPPA
axon guidance190.6×0.011CRPPA

Therapeutics

Drug target analysis

Approved (phase 4): 0 · Phase ≥3: 0 · Phased (≥1): 0 · Undrugged: 3

Druggability breadth: 0 of 3 evidence-associated genes (0%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Top cohort targets by molecule count

SymbolMoleculesMax phase
CRPPA00
LRRC7200
CRPPA-AS100

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 1.

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
CRPPA2.7.7.40D-ribitol-5-phosphate cytidylyltransferase

Pharmacogenomics

Cohort genes with a PharmGKB record: 2; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

0 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)0
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug1CRPPA
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug2LRRC72, CRPPA-AS1

Undrugged target profiles

3 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
CRPPA0
LRRC720
CRPPA-AS10

Clinical trials & evidence

Clinical trials

Clinical trials: 0.