Muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8

disease
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Also known as MDDGA8muscle-eye-brain-POMGNT2 relatedmuscular dystrophy-dystroglycanopathy, type A caused by mutation in POMGNT2POMGNT2 muscular dystrophy-dystroglycanopathy, type A

Summary

Muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8 (MONDO:0013904) is a disease caused by POMGNT2 (GenCC Definitive), with 3 cohort genes.

At a glance

  • Causal gene: POMGNT2 (GenCC Definitive)
  • Cohort genes: 3
  • ClinVar variants: 434

Clinical features

No curated clinical features (Orphanet) for this disease.

Identifiers

Disease identifiers

FieldValue
Canonical namemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8
Mondo IDMONDO:0013904
OMIM614830
DOIDDOID:0111231
UMLSC3553813
MedGen766727
GARD0015846
Is cancer (heuristic)no

Also known as: MDDGA8 · muscle-eye-brain-POMGNT2 related · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8 · muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies, type A, 8 · muscular dystrophy-dystroglycanopathy, type A caused by mutation in POMGNT2 · POMGNT2 muscular dystrophy-dystroglycanopathy, type A

Data availability: 434 ClinVar variants · 4 GenCC gene-disease records.

Disease family

Classification path: disease › human disease › disease by body system or component › nervous system disordercongenital nervous system disordercongenital muscular dystrophymuscular dystrophy-dystroglycanopathymuscular dystrophy-dystroglycanopathy, type Amuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 8

Related subtypes (13): muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A1, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A3, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A2, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A5, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A6, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 7, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 10, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 11, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 12, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A13, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A14, muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A9

Genetics & variants

GWAS landscape

No GWAS associations recorded — common-variant (GWAS) studies don’t cover this disease (typical for Mendelian / rare diseases). See the curated gene cohort and Mendelian overlap below.

Variant details and genetic-evidence tiers

ClinVar germline variants

434 retrieved; paginated sample, class counts are floors:

223 uncertain significance, 156 likely benign, 20 pathogenic, 16 conflicting classifications of pathogenicity, 9 benign/likely benign, 4 pathogenic/likely pathogenic, 3 likely pathogenic, 3 benign

ClinVarVariant (HGVS)GeneClassificationReview
156455NM_001101426.4(CRPPA):c.1105GTT[3] (p.Val372del)CRPPAPathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1055376NM_032806.6(POMGNT2):c.1326G>A (p.Trp442Ter)POMGNT2Pathogeniccriteria provided, single submitter
1077124NM_032806.6(POMGNT2):c.1232_1233del (p.Gln411fs)POMGNT2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
1252076NM_032806.6(POMGNT2):c.503T>C (p.Phe168Ser)POMGNT2Pathogenicno assertion criteria provided
1376803NM_032806.6(POMGNT2):c.509del (p.Asp170fs)POMGNT2Pathogeniccriteria provided, single submitter
1400524NM_032806.6(POMGNT2):c.118C>T (p.Arg40Ter)POMGNT2Pathogeniccriteria provided, single submitter
1452812NM_032806.6(POMGNT2):c.820_821del (p.Lys274fs)POMGNT2Pathogeniccriteria provided, single submitter
1453141NM_032806.6(POMGNT2):c.410_411delinsG (p.Ala137fs)POMGNT2Pathogeniccriteria provided, single submitter
1459409NC_000003.11:g.(?43121181)(43122923_?)delPOMGNT2Pathogeniccriteria provided, single submitter
1997096NM_032806.6(POMGNT2):c.40del (p.Val14fs)POMGNT2Pathogeniccriteria provided, single submitter
2055124NM_032806.6(POMGNT2):c.1510del (p.Val504fs)POMGNT2Pathogeniccriteria provided, single submitter
2058172NM_032806.6(POMGNT2):c.381_382del (p.Gln128fs)POMGNT2Pathogeniccriteria provided, single submitter
2142831NM_032806.6(POMGNT2):c.817_818del (p.Glu273fs)POMGNT2Pathogeniccriteria provided, single submitter
3620611NM_032806.6(POMGNT2):c.248G>A (p.Trp83Ter)POMGNT2Pathogeniccriteria provided, single submitter
37207NM_032806.6(POMGNT2):c.1333C>T (p.Arg445Ter)POMGNT2Pathogeniccriteria provided, single submitter
436373NM_032806.6(POMGNT2):c.745C>T (p.Gln249Ter)POMGNT2Pathogeniccriteria provided, single submitter
4739533NM_032806.6(POMGNT2):c.851del (p.Leu284fs)POMGNT2Pathogeniccriteria provided, single submitter
4812128NM_032806.6(POMGNT2):c.740_741del (p.Phe247fs)POMGNT2Pathogeniccriteria provided, single submitter
545449NM_032806.6(POMGNT2):c.758C>T (p.Pro253Leu)POMGNT2Pathogeniccriteria provided, multiple submitters, no conflicts
570608NM_032806.6(POMGNT2):c.1232del (p.Gln411fs)POMGNT2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
660661NM_032806.6(POMGNT2):c.1042C>T (p.Gln348Ter)POMGNT2Pathogeniccriteria provided, single submitter
694624NM_032806.6(POMGNT2):c.590G>A (p.Trp197Ter)POMGNT2Pathogenicno assertion criteria provided
817989NM_032806.6(POMGNT2):c.1258del (p.Ala420fs)POMGNT2Pathogenic/Likely pathogeniccriteria provided, multiple submitters, no conflicts
859488NM_032806.6(POMGNT2):c.1000_1003del (p.Leu334fs)POMGNT2Pathogeniccriteria provided, single submitter
1058894NM_032806.6(POMGNT2):c.1555G>T (p.Glu519Ter)POMGNT2Likely pathogeniccriteria provided, single submitter
3008348NM_032806.6(POMGNT2):c.1170T>G (p.Tyr390Ter)POMGNT2Likely pathogeniccriteria provided, single submitter
3780481NM_032806.6(POMGNT2):c.1264C>T (p.Gln422Ter)POMGNT2Likely pathogeniccriteria provided, single submitter
262107NM_032806.6(POMGNT2):c.561C>T (p.His187=)POMGNT2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
37208NM_032806.6(POMGNT2):c.473G>A (p.Arg158His)POMGNT2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications
382924NM_032806.6(POMGNT2):c.450A>G (p.Pro150=)POMGNT2Conflicting classifications of pathogenicitycriteria provided, conflicting classifications

Genes & proteins

Mendelian disease overlap and somatic drivers

GenCC: 6 · Orphanet: 6 · OMIM-shared: 0 · Dual-evidence (GWAS+Mendelian): 0

GenCC gene–disease validity (cohort genes)

the Disease column is the GenCC-asserted condition — a cohort gene’s strongest validity may be for a related predisposition syndrome.

GeneClassificationInheritanceDiseaseRecords
POMGNT2DefinitiveAutosomal recessivemuscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type a, 86

Orphanet rare-disease linkage (cohort genes)

GeneOrphanet IDRare disease
POMGNT2Orphanet:899Walker-Warburg syndrome
ABHD5Orphanet:98907Neutral lipid storage disease with ichthyosis
CRPPAOrphanet:352479ISPD-related limb-girdle muscular dystrophy R20
CRPPAOrphanet:370980Congenital muscular dystrophy without intellectual disability
CRPPAOrphanet:588Muscle-eye-brain disease
CRPPAOrphanet:899Walker-Warburg syndrome

Cohort genes → proteins

3 cohort genes, 3 distinct canonical proteins.

Evidence partition

SubsetGenes
multi_evidence3

Cohort genes (full)

SymbolHGNCEnsemblUniProtNameEvidence
POMGNT2HGNC:25902ENSG00000144647Q8NAT1Protein O-linked-mannose beta-1,4-N-acetylglucosaminyltransferase 2gencc,clinvar
ABHD5HGNC:21396ENSG00000011198Q8WTS11-acylglycerol-3-phosphate O-acyltransferase ABHD5clinvar
CRPPAHGNC:37276ENSG00000214960A4D126D-ribitol-5-phosphate cytidylyltransferaseclinvar

Cohort function summary

Lead sentence per gene, UniProt-curated.

SymbolProtein nameFunction (lead sentence)
POMGNT2Protein O-linked-mannose beta-1,4-N-acetylglucosaminyltransferase 2O-linked mannose beta-1,4-N-acetylglucosaminyltransferase that transfers UDP-N-acetyl-D-glucosamine to the 4-position of the mannose to generate N-acetyl-D-glucosamine-beta-1,4-O-D-mannosylprotein.
ABHD51-acylglycerol-3-phosphate O-acyltransferase ABHD5Coenzyme A-dependent lysophosphatidic acid acyltransferase that catalyzes the transfer of an acyl group on a lysophosphatidic acid.
CRPPAD-ribitol-5-phosphate cytidylyltransferaseCytidylyltransferase required for protein O-linked mannosylation.

Protein-family classification

Druggable: 2 · Difficult: 0 · Unknown: 1 · Druggable fraction: 0.67

Family distribution

Cohort families vs a genome-wide background (hypergeometric, BH-FDR; fold = observed/expected). Counts kept; sorted by enrichment, so the catch-all Other/Unknown bucket no longer leads.

FamilyGenesFoldFDR
Antibody/Immunoglobulin19.7×0.298
Enzyme (other)14.0×0.345
Other/Unknown10.6×0.914

Per-gene assignment

SymbolFamilyDruggable?ECInterPro (top 3)
POMGNT2Antibody/Immunoglobulinyes2.4.1.312FN3_dom, Glycosyltransferase_61, Ig-like_fold
ABHD5Other/UnknownnoAB_hydrolase_1, AB_hydrolase_fold
CRPPAEnzyme (other)yes2.7.7.40ISPD_synthase_CS, Nucleotide-diphossugar_trans, IspD/TarI

Expression context

Cohort genes with no expression data: 0.

2 cohort genes are a single-cell marker in ≥1 SCXA experiment.

Breadth distribution (Bgee present_calls)

BucketGenes
narrow (1-5 tissues)0
moderate (6-20)0
broad (>20)3
unknown0

Top tissues across cohort

TissueCohort genes
cardiac muscle of right atrium1
lateral nuclear group of thalamus1
right hemisphere of cerebellum1
amniotic fluid1
lower esophagus mucosa1
upper leg skin1
calcaneal tendon1
corpus callosum1
male germ line stem cell (sensu Vertebrata) in testis1

Per-gene tissue summary (top 30)

SymbolBgee breadthFANTOM5 breadthSCXATop tissues
POMGNT2245ubiquitousmarkerlateral nuclear group of thalamus, right hemisphere of cerebellum, cardiac muscle of right atrium
ABHD5276ubiquitousmarkeramniotic fluid, lower esophagus mucosa, upper leg skin
CRPPA134ubiquitousyescorpus callosum, male germ line stem cell (sensu Vertebrata) in testis, calcaneal tendon

Protein interactions among cohort

Intra-cohort edges: 1.

Hub genes (top 10 by interactor count)

SymbolInteractor count
ABHD52,084
CRPPA1,629
POMGNT2968

Intra-cohort edges

ABSources
CRPPAPOMGNT2string_interaction

Structural data

PDB: 2 · AlphaFold-only: 1 · No structure: 0

Cohort genes with PDB structures (top 30)

SymbolUniProtPDB entries
POMGNT2Q8NAT13
CRPPAA4D1261

AlphaFold-only cohort genes (top 30 by pLDDT)

SymbolUniProtpLDDT
ABHD5Q8WTS187.62

Function

Pathway analysis

Distinct Reactome pathways touched by cohort: 6. Enrichment computed across 3 evidence-associated genes (3 with Reactome annotation).

Pathways by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 3 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

PathwayCohort genesFoldFDRSample cohort genes
DAG1 core M3 glycosylations1634.4×0.009POMGNT2
Matriglycan biosynthesis on DAG11271.9×0.009CRPPA
Triglyceride metabolism1223.9×0.009ABHD5
Triglyceride catabolism1158.6×0.009ABHD5
Metabolism of lipids110.5×0.111ABHD5
Metabolism13.9×0.237ABHD5

GO biological processes by enrichment

Over-representation of cohort genes vs the genome-wide background (hypergeometric test, Benjamini-Hochberg FDR; fold = observed/expected over 3 annotated cohort genes). Counts and members are kept as ground-truth; sorted by enrichment.

GO termCohort genesFoldFDRSample cohort genes
protein O-linked glycosylation via mannose2624.1×4e-05POMGNT2, CRPPA
negative regulation of triglyceride storage11872.4×0.003ABHD5
positive regulation of triglyceride catabolic process1702.2×0.005ABHD5
isoprenoid biosynthetic process1561.7×0.005CRPPA
phosphatidic acid biosynthetic process1170.2×0.013ABHD5
lipid homeostasis1112.3×0.016ABHD5
protein O-linked glycosylation174.9×0.021POMGNT2
fatty acid metabolic process164.6×0.021ABHD5
neuron migration144.6×0.027POMGNT2
axon guidance130.2×0.036CRPPA
cell differentiation19.7×0.100ABHD5

Therapeutics

Drug target analysis

Approved (phase 4): 1 · Phase ≥3: 1 · Phased (≥1): 1 · Undrugged: 2

Druggability breadth: 1 of 3 evidence-associated genes (33%) have a ChEMBL target (buckets above are over the deeply-mined display cohort).

Genes with an approved drug

The molecule shown is one approved compound that hits the gene — not necessarily a drug of choice or one indicated for this disease.

SymbolExample approved molecule
ABHD5NIFEDIPINE

Top cohort targets by molecule count

SymbolMoleculesMax phase
ABHD584
POMGNT200
CRPPA00

Drugs targeting cohort genes (top 30)

MoleculeMax phaseTargets in cohort
NIFEDIPINE4ABHD5
BENZBROMARONE4ABHD5
ETHACRYNIC ACID4ABHD5
MENADIONE4ABHD5
DISULFIRAM4ABHD5
INAMRINONE4ABHD5
CURCUMIN3ABHD5
EBSELEN3ABHD5

Bioactivity and enzyme data

Enzyme cohort genes (≥1 EC): 2.

Cohort genes with ChEMBL bioactivity (full, sorted by assay count)

SymbolAssaysType breakdown
ABHD52Binding:2

Cohort enzymes (BRENDA EC)

SymbolEC numbersNames
POMGNT22.4.1.312protein O-mannose beta-1,4-N-acetylglucosaminyltransferase
CRPPA2.7.7.40D-ribitol-5-phosphate cytidylyltransferase

Pharmacogenomics

Cohort genes with a PharmGKB record: 3; with CPIC/DPWG dosing guidelines: 0.

No cohort gene has a CPIC/DPWG genotype-guided dosing guideline (PharmGKB).

Chemical tractability of cohort targets

8 approved/phased compounds have measured bioactivity against a cohort gene (and aren’t yet in disease-level trials). This is a research / tractability signal, NOT a therapeutic recommendation — a bioactivity row often reflects off-target or screening binding (e.g. promiscuous kinase inhibitors against a cohort kinase), implying no disease mechanism.

CompoundMax phaseCohort target (bioactivity)
NIFEDIPINE4ABHD5
BENZBROMARONE4ABHD5
ETHACRYNIC ACID4ABHD5
MENADIONE4ABHD5
DISULFIRAM4ABHD5
INAMRINONE4ABHD5
CURCUMIN3ABHD5
EBSELEN3ABHD5

Druggability pyramid

Cohort genes binned by druggability tier (high → low):

TierDefinitionGenesSymbols
AApproved (phase 4 drug)1ABHD5
BPhased (≥1) drug, not yet approved0
CDruggable family + PDB, no drug2POMGNT2, CRPPA
DDruggable family + AlphaFold only, no drug0
EDifficult family or no structure, no drug0

Undrugged target profiles

2 cohort genes are undrugged. Ranked by ‘starting-point quality’ (assay depth + drugged-partner adjacency).

SymbolChEMBL assaysDrugged partners (top 3)
POMGNT20
CRPPA0

Clinical trials & evidence

Clinical trials

Clinical trials: 0.